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methemoglobinemia/ataque epiléptico
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Indoxacarb Poisoning Presenting as Methemoglobinemia and Seizure.
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We are reporting a case of poisoning with a non-organophosphate pesticide (indoxacarb) resulting in methemoglobinemia and seizures, and successfully treated with ventilator care and intravenous ascorbic acid. Since there are limited data concerning the human toxicity of indoxacarb, physicians in
Seizures and Methemoglobinemia After Topical Application of Eutectic Mixture of Lidocaine and Prilocaine on a 3.5-Year-Old Child with Molluscum Contagiosum and Atopic Dermatitis.
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A eutectic mixture of lidocaine and prilocaine (EMLA) is used topically to provide local anesthesia for a variety of painful superficial procedures. Although the side effects of EMLA are usually mild and transient local reactions, potentially life-threatening complications can occur. We report a
Seizures and methemoglobinemia in an infant after excessive EMLA application.
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Topical anesthetics are commonly used in many health care settings and for many clinical conditions. However, there are a number of potential adverse effects associated with their use. Their widespread administration can convey a false sense of security and failure to appreciate possible
Acute toxic methemoglobinemia following dental analgesia.
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This communication reports a case of acute toxic methemoglobinemia associated with hypoxia and general motor seizures following dental anesthesia using nitrous oxide and prilocaine hydrochloride. The dosage of prilocaine hydrochloride used was 5.6% of the recognized toxic dosage associated with
Dapsone induced methemoglobinemia : Intermittent vs continuous intravenous methylene blue therapy.
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OBJECTIVE
The study compares the decline in blood methemoglobin (MetHb) level in children of dapsone intoxication treated with intermittent and continuous methylene blue therapy.
METHODS
Eleven children with history of accidental dapsone ingestion and suggestive clinical features of dapsone
[Late effects of toxic methemoglobinemia].
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A psychological testing rendered it possible to reveal some disorders of the central nervous system in a chemical industry worker who suffered from a serious intoxication with chloronitrobenzene a year ago. Such symptoms as loss of consciousness, convulsions and serious methemoglobinemia were
[Congenital enzymopenic methemoglobinemia and its pharmacogenetic aspects].
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The authors report on their experience in nearly 10 years follow-up of four families with hereditary enzymopenic methemoglobinemia. In patients a dangerous elevation of methemoglobin level was never observed, what is probably due to the information of the patients and the family doctor about the
Hemolysis and methemoglobinemia secondary to rasburicase administration.
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OBJECTIVE
To report a case of hemolytic anemia and methemoglobinemia developing after rasburicase administration to a patient with glucose-6-phosphate dehydrogenase (G6PD) deficiency.
METHODS
A 50-year-old African American man was hospitalized with new onset seizure, diabetic ketoacidosis,
[Pregnancy in hereditary enzymatic methemoglobinemia].
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The authors describe a case of successful pregnancy in a patent with hereditary enymopenic methemoglobinemia. The course of pregnancy was followed up by the use of ultrasound and the control of CTG, estriol, and pregnanediol. No signs of the intrauterine fetal threat were noticed. The methemoglobin
Central Nervous System Symptoms Due to Transient Methemoglobinemia in a Child With G6PD Deficiency.
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The authors herein report a 5-year-old child who presented with massive hemolysis, irritability, and cyanosis. The final diagnosis was glucose-6-phosphate dehydrogenase deficiency with associated central nervous system symptoms probably because of concomitantly acquired methemoglobinemia following
A Thai boy with hereditary enzymopenic methemoglobinemia type II.
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Individuals with methemoglobin exceeding 1.5 g/dl have clinically obvious central cyanosis. Hereditary methemoglobinemia is due either to autosomal dominant M hemoglobins or to autosomal recessive enzymopenic methemoglobinemia. Four types of enzymopenic methemoglobinemia have been described. In
Celecoxib-induced methemoglobinemia.
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OBJECTIVE
To report a case of acute methemoglobinemia in a patient treated with celecoxib for osteoarthritis.
METHODS
A 72-year-old African American man developed an acute confusional state (ACS) one month after receiving celecoxib for osteoarthritis of his knee joints. There was no other
Massive sulfasalazine and paracetamol ingestion causing acidosis, hyperglycemia, coagulopathy, and methemoglobinemia.
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METHODS
Reports of acute toxicity following sulfasalazine ingestion are rare. A case of an acute ingestion of sulfasalazine 50 g and paracetamol 50 g resulting in severe lactic acidosis, seizures, coagulopathy, hyperglycemia, ketosis, and methemoglobinemia is reported. Despite the ingestion of a
Methemoglobinemia induced by topical vaginal sulfanilamide cream in a patient with cervical cancer: a case report.
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BACKGROUND
Methemoglobinemia is a rare disorder most commonly associated with the ingestion or topical application of an offending exogenous agent. The clinical consequences of acute methemoglobinemia can be devastating and include lethargy, headache, and dyspnea and, as methemoglobin concentrations
Benzocaine-adultered street cocaine in association with methemoglobinemia.
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A 27-year-old man with a history of cocaine abuse ingested a large quantity of street cocaine in an apparent suicide attempt. Shortly thereafter, he developed tonic-clonic seizures and became cyanotic. An arterial blood gas sample, collected in the emergency department, appeared chocolate-brown and
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