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The activation of spinal N-methyl-D-aspartate receptors may contribute to degeneration of spinal motor neurons induced by neuraxial morphine after a noninjurious interval of spinal cord ischemia.

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We investigated the relationship between the degeneration of spinal motor neurons and activation of N-methyl-d-aspartate (NMDA) receptors after neuraxial morphine following a noninjurious interval of aortic occlusion in rats. Spinal cord ischemia was induced by aortic occlusion for 6 min with a

Intravenous infusion of dexmedetomidine can prevent the degeneration of spinal ventral neurons induced by intrathecal morphine after a noninjurious interval of spinal cord ischemia in rats.

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BACKGROUND In recent studies, we demonstrated that neuraxial morphine after noninjurious spinal cord ischemia in the rat could induce spastic paraplegia and degeneration of selective spinal ventral neurons. Our objective was to investigate the impact of dexmedetomidine infusion on the degeneration

Morphine-mediated deterioration of oxidative stress leads to rapid disease progression in SIV/SHIV-infected macaques.

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Oxidative stress is well documented in HIV infection, but the effect of concomitant substance abuse is largely unknown. We studied oxidative stress in our macaque model of morphine abuse and AIDS. In plasma, we found an approximately 50% decrease in catalase activity with morphine dependence that

Morphine deteriorates spatial memory in sodium salicylate treated rats.

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Tolerance and cross-tolerance for the effects of morphine (M) and sodium salicylate on nociception and learning were examined. The anti-nociceptive effects were measured by using the classic tail flick (TF) and hot plate (HP) tests and learning was measured with the Morris water maze (MWM).

Morphine contributed to the deterioration of cancer via miR-543/MARCKS/FcγR-mediated phagocytosis pathway.

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It has been confirmed that morphine was detrimental to patients with cancers. Hence, we aimed to reveal a certain mechanism of morphine in cancer development.Microarray and GSEA analysis were utilized to seek for differently expressed genes and

Epidural use of morphine in managing the pain of carneous degeneration of a uterine leiomyoma during pregnancy.

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Intrathecal administration of morphine, but not small dose, induced spastic paraparesis after a noninjurious interval of aortic occlusion in rats.

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We sought to investigate the dose-response relationship for the effect of intrathecal morphine on the transient spastic paraparesis after short-lasting spinal ischemia in rats. Spinal ischemia was induced by aortic occlusion for 6 min with a balloon catheter in rats previously implanted with an

Morphine infusions after pediatric cranial surgery: a retrospective analysis of safety and efficacy.

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BACKGROUND Morphine infusions are avoided after cranial surgery in children due to concerns of masking neurological deterioration and because cranial surgery is considered less painful than other surgical procedures. Studies indicate that up to 60% of patients experience moderate to severe pain

Thoracic epidural morphine in the palliation of chest wall pain secondary to relapsing polychondritis.

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Relapsing polychondritis is a rare disease of unknown etiology characterized pathologically by degeneration of the chondrocyte and replacement with fibrous connective tissue. The following case report presents the pain management of a 34-yr-old man suffering from intractable pain secondary to

Reassessing morphine effects in cats: III. Responses of intact, caudate nuclei-lesioned and hemispherectomized animals following chronic administration and precipitated withdrawal.

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Behavioral response to low doses of morphine (2.0 to 3.0 mg/kg, IP) administered for up to 15 days, and responses to subsequent naloxone challenges, were measured in intact, unilaterally and bilaterally caudate-lesioned (acaudate) cats, and in hemispherectomized cats using a video time-sampling

Morphine-induced transactivation of HIV-1 LTR in human neuroblastoma cells.

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Infection by human immunodeficiency virus (HIV) is followed in many cases by a clinically quiescent or latent phase that appears to continue as long as host antiviral defense is intact. This has raised the possibility that certain host susceptibility factors (i.e., environmental cofactors) might

Skeletal muscle circulation during sufentanyl and morphine anesthesia in propranolol treated dogs.

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Sufentanyl is a new, potent, short-acting, fentanyl-like morphinomimetic. In the present study we compared the effects of high doses of sufentanyl and morphine on the peripheral circulation in beta-blocked dogs. Skeletal muscle surface pH (m-pH) was recorded continuously as an index of the

Catecholamine and serotonin depletion from rat spinal cord: effects on morphine and footshock induced analgesia.

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Using a methodology that causes a selective degeneration of spinal cord catecholaminergic or serotoninergic pathways but not those of the brain, it has been possible to study more precisely the role played by the spinal cord monoaminergic systems that underly the mechanism through which morphine and

Enhancement of tumor growth by morphine and its possible mechanism in mice.

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The effect of morphine on tumor growth of EL-4 leukemia in C57BL/6 mice and of Sarcoma 180 carcinoma in ddY mice was studied. Local subcutaneous tumor growth was enhanced by morphine (10 mg/kg, s.c.) given daily for 10d. This effect was inhibited by preadministration of the opioid antagonist

Disappearance of gamma-aminobutyric acid (GABA)B receptor-mediated cAMP suppression in mouse cerebrum after chronic treatment with morphine.

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The effects of chronic morphine administration (dependence) and naloxone-induced withdrawal on cerebral GABAB receptor and its signal transduction system were examined. Alterations in receptor affinity and number and in the amount of Gi protein were determined by radioligand binding assay and

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