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peritonitis/triglyceride
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Página 1 desde 73 resultados
Medium-chain triglyceride supplementation exacerbates peritonitis-induced septic shock in rats: role on cell membrane remodeling.
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OBJECTIVE
Lipid emulsions for parenteral nutrition interfere with immunity and may alter the cell plasma membrane and microparticle release, thus modulating their biological effects. Our aim was to evaluate the effect of two lipid emulsions for parenteral nutrition containing either a mixture of
Hepatic histological lesions during acute experimental peritonitis in the rat.
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During acute experimental peritonitis in the rat, the main histological lesions in the liver are represented by venous stasis in the central lobular vein and in the sinusoids; by glycogen mobilization and intracytoplasmic accumulation of triglycerides in the hepatocytes; by activation and
Congenital chyloperitoneum: direct comparison of medium-chain triglyceride treatment with total parenteral nutrition.
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A severe case of congenital chyloperitoneum was managed over a prolonged period by permanent drainage and replacement of lymphatic loss by fresh frozen plasma, resulting in normal development of the infant. The average daily drainage of 360 ml during a period of a medium-chain-triglyceride (MCT)
Metabolic differences between persistent and routine peritonitis in CAPD.
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Changes in 10 metabolic parameters (body weight, blood hemoglobin, and serum albumin, urea, creatinine, cholesterol, triglycerides, potassium, calcium and phosphorus) were compared in 28 episodes of routine peritonitis and 27 episodes of persistent peritonitis. These infections occurred in 20 CAPD
[Lipid metabolism during hemosorption in experimental peritonitis].
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Increase of lipolytic hormones and cAMP was revealed in experimental peritonitis. The amount of free fatty acids and the total protein content in the blood increased on the average by 50 and 40%, respectively. The blood free fatty acid content reduced to control values after hemosorption, the
[Enteral therapy of the syndrome of intestinal insufficiency in patients with peritonitis].
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Sixty five patients with peritonitis were examined and divided into two groups. The operative intervention in all the patients included transnasal intubation of the small intestine. The basic enteral therapy used in the first group of patients was made with the glucose-saline solution and transition
Effect of toll-like receptor agonists on the formation of macrophage/foam cells upon acute peritonitis in mice.
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We studied effects of zymosan, double-stranded RNA, LPS of E. coli and bacterial CpG DNA, agonists of toll-like receptor TLR2, TLR3, TLR4 and TLR9, respectively, on the formation of macrophage/foam cells 24 h after induction of acute peritonitis. Administration of agonists led to transformation of
[Metabolic correction of the lipid-transport system in experimental diffuse purulent peritonitis].
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The research was performed in 55 male chinchilla rabbits. For the first time the effect of metabolic preparations "citoflavin" and "neoton" of the protein-lipid spectrum of blood was studied in experimental diffuse purulent peritonitis. The development of diffuse purulent peritonitis caused negative
[Use of liquid sorbents based on chitosan for treatment of diffuse forms of peritonitis].
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Chitosan-based gelatinous affinity sorbent containing 9 mg/ml of polymyxin B was injected into the infected peritoneal cavity for twenty-four hours for detoxification in diffuse peritonitis. The clinical assay of blood plasma (Spectrum Abbot, USA) revealed that the lavage of the infected abdomen by
[Lipid peroxidation products in experimental peritonitis].
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Oxygen free radicals attribute to the peroxidation of lipids forming the cellular wall. These processes are manifested by means of phospholipid (PL), triglyceride (TG), and cholesterol (CH) concentration changes. Simultaneously, as a result of arachidonic acid peroxidation, conjugated dienes (CD)
[Evaluation of the usefulness of the physico-chemical study of ascitic fluid in patients with chronic hepatopathies, neoplasms and spontaneous peritonitis].
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In order to separate malignancy from chronic liver disease, the ascitic fluid (AF) of 45 in-patients was studied prospectively. Protein, cholesterol, triglycerides, LDH and glucose concentrations were determined in AF. Ascites/Serum (A/S) protein and LDH ratios were also established. Spontaneous
Clinical characteristics and outcomes of "silent" and "non-silent" peritonitis in patients on peritoneal dialysis.
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OBJECTIVE
We compared the clinical characteristics and outcomes of "silent" peritonitis (meaning episodes without fever and abdominal pain) and "non-silent" peritonitis in patients on peritoneal dialysis (PD).
METHODS
Our cohort study collected data about all peritonitis episodes occurring between
Nutrition and delayed hypersensitivity during continuous ambulatory peritoneal dialysis in relation to peritonitis.
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Anthropometric measurements, sixteen specific plasma proteins, triglycerides, cholesterol, urea and creatinine were measured at 4-monthly intervals for 1 year in 15 patients on CAPD. Delayed hypersensitivity skin tests were performed on 11 patients at the start and after 4 and 12 months. Body weight
Relation of demographic, clinic and biochemical parameters to peritonitis in peritoneal dialysis.
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The relation of various demographic, clinical and biochemical parameters of peritoneal dialysis patients with peritonitis and other infections was evaluated. The age, gender, peritoneal dialysis (PD) period, educational status, peritonitis, exit site score, serum albumin, C-reactive protein (CRP),
Adipose triglyceride lipase acts on neutrophil lipid droplets to regulate substrate availability for lipid mediator synthesis.
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In humans, mutations in ATGL lead to TG accumulation in LDs of most tissues and cells, including peripheral blood leukocytes. This pathologic condition is called Jordans' anomaly, in which functional consequences have not been investigated. In the present study, we tested the hypothesis that ATGL
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