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post-traumatic stress disorder/hypoxia
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[The anxyolytic effect of mild hypobaric hypoxia in a model of post-traumatic stress disorder in rats].
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The impact of mild hypobaric hypoxia on the development of anxiety-like state in rats in experimentally simulated human post-traumatic stress disorder was studied. Three-trial exposure to mild hypobaric hypoxia (360 mm Hg for 2 hours daily, for 3 days) in preconditioning or post-conditioning mode
Acclimatization to Middle Attitude Hypoxia Masks the Symptoms of Experimental Posttraumatic Stress Disorder, but Does Not Affect Its Pathogenetic Mechanisms.
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The effects of acclimatization to middle attitude hypoxia on the resistance to acute emotional stress were studied on the model of posttraumatic stress disorder in rats. Anxyolitic, but not anxiogenic effect was observed in acclimatized rats. However, acclimatized rats with posttraumatic stress
Intermittent hypoxia improves behavioral and adrenal gland dysfunction induced by posttraumatic stress disorder in rats.
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Nonpharmacological treatments of stress-induced disorders are promising, since they enhance endogenous stress defense systems, are free of side effects, and have few contraindications. The present study tested the hypothesis that intermittent hypoxia conditioning (IHC) ameliorates behavioral,
Post-traumatic hypoxia exacerbates brain tissue damage: analysis of axonal injury and glial responses.
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Traumatic brain injury (TBI) resulting in poor neurological outcome is predominantly associated with diffuse brain damage and secondary hypoxic insults. Post-traumatic hypoxia is known to exacerbate primary brain injury; however, the underlying pathological mechanisms require further elucidation.
Post-traumatic hypoxia exacerbates neurological deficit, neuroinflammation and cerebral metabolism in rats with diffuse traumatic brain injury.
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BACKGROUND
The combination of diffuse brain injury with a hypoxic insult is associated with poor outcomes in patients with traumatic brain injury. In this study, we investigated the impact of post-traumatic hypoxia in amplifying secondary brain damage using a rat model of diffuse traumatic axonal
Post-traumatic hypoxia exacerbates neuronal cell death in the hippocampus.
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Hypoxia frequently occurs in patients with traumatic brain injury (TBI) and is associated with increased morbidity and mortality. This study examined the effects of immediate or delayed post-traumatic hypoxia (fraction of inspired oxygen [FiO(2)] 11%) on acute neuronal degeneration and long-term
Photoacoustic imaging of synovial tissue hypoxia in experimental post-traumatic osteoarthritis.
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OBJECTIVE
This pilot study aimed to investigate the feasibility of non-invasively assessing synovial tissue hypoxia in vivo using photoacoustic (PA) imaging in a post-traumatic osteoarthritis model and explore its correlation with OA severity.
METHODS
The three-dimensional vasculature structure and
Post-traumatic hypoxia is associated with prolonged cerebral cytokine production, higher serum biomarker levels, and poor outcome in patients with severe traumatic brain injury.
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Secondary hypoxia is a known contributor to adverse outcomes in patients with traumatic brain injury (TBI). Based on the evidence that hypoxia and TBI in isolation induce neuroinflammation, we investigated whether TBI combined with hypoxia enhances cerebral cytokine production. We also explored
[First clinical results of a treatment of post-traumatic and postoperative cerebral hypoxia with CO2-O2 enriched air].
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[Post-traumatic and hemorrhagic refractory hypoxia].
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Posthypoxic ascites: a cause of post-traumatic abdominal distention.
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A previously unreported etiology for post-traumatic abdominal distention was observed in a 5-year-old girl who suffered only head trauma and a prolonged period of hypoxemia. When there is a rapid accumulation of peritoneal fluid containing a high concentration of protein but negative for amylase, no
Ischemic injury-specific gene expression in the rat spinal cord injury model using hypoxia-inducible system.
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METHODS
A spinal cord injury and in vitro neural hypoxia models were used to evaluate the hypoxia responsive gene expression.
OBJECTIVE
To limit the risk of unwanted overexpression of therapeutic genes, we developed a hypoxia-inducible gene therapy system using the erythropoietin (Epo) enhancer and
Secondary hypoxia exacerbates acute disruptions of energy metabolism in rats resulting from fluid percussion injury.
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The purpose of these experiments was to determine whether secondary hypoxia exacerbates the metabolic consequences of fluid percussion injury (FPI). In Experiment I, rats were trained to press a lever for their entire daily ration of food at any time during a 12-h light/dark cycle and run in an
[Hypoxic postconditioning corrects behavioral abnormalities in a model of post-traumatic stress disorder in rats].
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Protective effects of the novel technique of hypoxic postconditioning with a hypobaric hypoxia paradigm were studied in "stress-restress" model ofposttraumatic stress disorder in rats. It was shown that repeated (3 times) exposure of rats that survived after severe traumatic stress to mild hypobaric
Intracranial pressure changes following traumatic brain injury in rats: lack of significant change in the absence of mass lesions or hypoxia.
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Traumatic brain injury (TBI) often causes raised intracranial pressure (ICP), with >50% of all TBI- related deaths being associated with this increase in ICP. To date, there is no effective pharmacological treatment for TBI, partly because widely used animal models of TBI may not replicate many of
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