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tachycardia/hypoxia
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Hypoxia and tachycardia during endoscopic retrograde cholangiopancreatography: detection by pulse oximetry.
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Oxygen saturation and pulse rate were recorded using pulse oximetry during 50 consecutive endoscopic retrograde cholangiopancreatography (ERCP) examinations. Oxygen saturation dropped to below 90% in 22 patients at some point during the procedure. Multiple regression analysis revealed that
Fetal seizures causing increased heart rate variability during terminal fetal hypoxia.
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Fetal seizures together with both abnormal breathing movements and fluctuations in fetal blood pressure and heart rate resulting in increased fetal heart rate variability have been observed in brain-damaged fetal sheep shortly after an asphyxial insult. We report a clinical example of convulsions
On the mechanisms by which hypoxia eliminates digitalis-induced tachyarrhythmias.
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The mechanism by which hypoxia abolishes the tachyarrhythmias induced by cardiac steroids was studied in cardiac Purkinje and ventricular muscle fibres. In muscle fibres, hypoxia reduces excitability markedly, reduces the size and increases the time to peak of the oscillatory potentials (Vos) and of
A Case of Fetal Tachycardia after Electroconvulsive Therapy: A Possible Effect of Maternal Hypoxia and Uterine Contractions.
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Electroconvulsive therapy (ECT) is considered to be an effective and safe treatment for depression in pregnant women in that it avoids the risk of psychotropic pharmacotherapy. However, clinicians should be cautious about the adverse effects in the fetus, such as fetal cardiac arrhythmia. Most of
Paradoxical fall of tachycardia- and hypoxia-induced coronary flow under conditions of severe coronary stenosis in dogs.
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It has been stated that the coronary flow paradoxically falls in response to tachycardia if the coronary artery is stenotic and "compliant". To clarify this, we measured coronary vascular resistance by cannulating the left anterior descending coronary artery in open-chest dogs. In constant flow
Hypoxic tachycardia in hypoxia-acclimated rats.
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Acute hypoxia (9.5% O2) increased the heart rate (HR) in hypoxia (12% O2 for 2 months)-acclimated (HX) rats, while it decreased HR in unacclimated (CT) rats. After treatment with propranolol, acute hypoxia decreased HR to the same extent in CT and HX rats. After treatment with atropine, acute
Inhibitory effect of anoxia on reperfusion- and digitalis-induced ventricular tachyarrhythmias.
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In the isolated rat heart, anoxia or ischemia do not induce important ventricular tachyarrhythmias (VTAs). During the 1st min of reperfusion, VTAs are frequent. The frequency and severity of VTAs during reperfusion depend on the duration and the extent of the myocardial damage. Anoxia abolishes
Effect of acute hypoxia on human fetal heart rate. The significance of increased heart rate variability.
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The fetal heart rate response to acute hypoxemia was observed during an accidental drop in oxygen concentration inspired by the mother in labor. There was a marked increase in FHR variability in the early stage of hypoxia and also during the recovery phase. While an increase in fetal heart
Coronary vasodilator responses to hypoxia and induced tachycardia before and after lidoflazine.
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Effect of intranasal oxygen on hypoxia and tachycardia during endoscopic cholangiopancreatography.
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Decelerations, tachycardia, and decreased variability: have we overlooked the significance of longitudinal fetal heart rate changes for detecting intrapartum fetal hypoxia?
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Conditioning of tachycardia produced by histotoxic anoxia.
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The effect of hypoxia on experimental ventricular tachycardia.
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Minimal and deep sedation during ablation of ventricular tachycardia.
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BACKGROUND
Catheter ablation is a curative treatment option for ventricular premature contractions (VPC) and ventricular tachycardia (VT). Procedures require different sedation levels, depending on duration, ablation approach and patient characteristics. The aim of our study was to evaluate
[Ventricular tachycardia on arrival at the operating room in a patient with bilateral recurrent nerve paralysis].
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A 66-year-old man developed dyspnea at rest due to bilateral recurrent nerve paralysis. He was rushed into the operating room for emergency tracheostomy. He developed ventricular tachycardia (VT) due to hypoxemia before induction of general anesthesia. After tracheal intubation, VT returned to
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