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West Nile virus infections.
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Since its introduction to the United States in 1999, West Nile virus (WNV) has become endemic in North America and has emerged as the most common cause of epidemic meningoencephalitis in North America and the leading cause of arboviral encephalitis in the United States. West Nile virus is maintained
[Pathomorphology of West Nile fever].
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8 patients, aged from 61 to 70 years, died from serous meningoencephalitis during the outburst of the West Nile fever in Volgograd in 1999-2001, were studied morphologically. Serous meningoencephalitis with necrotic vasculitis is a characteristic feature of this fever with degenerative changes and
[Tungiasis in Bangou (West Cameroon)].
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Tungiasis is an ectoparasitosis caused by penetration into the skin of the sand or chigger flea, Tunga penetrans. The epidemiology in Cameroon of this disease, often associated with poverty, is unknown. Accordingly, in August 2008, we conducted a study in Bangou, in Western Cameroon, among a sample
Anti-tumour necrosis factor therapy in the West Midlands.
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West Nile virus in farmed alligators.
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Seven alligators were submitted to the Tifton Veterinary Diagnostic and Investigational Laboratory for necropsy during two epizootics in the fall of 2001 and 2002. The alligators were raised in temperature-controlled buildings and fed a diet of horsemeat supplemented with vitamins and minerals.
Does tumor necrosis factor protect against lupus in west Africans?
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West Nile virus encephalitis in a dog.
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In November 2002, a 2-year-old, spayed Maltese Terrier in central Mississippi was presented for an acute illness characterized by uncontrolled hyperactivity that rapidly progressed to generalized tremors, ataxia, and intermittent hyperthermia. Postmortem examination after a 2-week course revealed
Pathogenicity of West Nile virus in chickens.
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In the fall of 1999, West Nile virus (WNV) was isolated for the first time in the Western Hemisphere during an outbreak of neurologic disease in humans, horses, and wild and zoo birds in the northeastern United States. Chickens are a potential reservoir for WNV, and little is known about the
Hereditary copper toxicosis in West Highland white terriers.
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Histologic, histochemical and atomic absorption studies on liver tissue from 71 West Highland white terriers are reported. Twenty-seven dogs had histologically normal liver and copper concentration comparable to mongrel control dogs. Forty-four dogs had hepatic copper concentrations up to 22 times
Variations in biological features of West Nile viruses.
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Pathological findings in humans, horses, and birds with West Nile (WN) encephalitis show neuronal degeneration and necrosis in the central nervous system (CNS), with diffuse inflammation. The mechanisms of WN viral penetration of the CNS and pathophysiology of the encephalitis remain largely
Acute infectious oral necrosis among young children in the Gambia, West-Africa.
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[Rheumatic diseases profile of 13517 West African patients].
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OBJECTIVE
To determine the patterns of rheumatic diseases in patients attending the rheumatology unit of the Lomé Tokoin teaching hospital.
METHODS
Medical records of patients seen over 16 years period were studied transversally. All the patients suffering of rheumatic disease were including in the
Sheep pox: experimental studies with a west african isolate.
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Under conditions of a maximum security laboratory, four cross-bred sheep were inoculated intradermally only or intradermally and intratracheally with a West African isolate of sheep pox virus. All sheep had increased temperature and depression by the fourth or fifth day after infection. Nasal and
Regulation of immune recognition molecules by flavivirus, West Nile.
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We have shown the flaviviruses can up-regulate the cell surface expression of the immune recognition molecules, major histocompatability complex class-I and class-II (MHC-I, MHC-II), ICAM-1, VCAM, and E-selectin, in an interferon-independent and tumor necrosis factor-independent manner. This
East-west fusion-necrosis and apoptosis acting in concert by demethylcantharidin-integrated platinum complexes.
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The different types of cell death occurring in HCT116 colorectal cancer cell after the treatment of cisplatin, carboplatin, oxaliplatin, DMC, Pt(NH3)2(demethylcantharidin:DMC) and Pt(R,R-1,2-diaminocyclohexane: DACH)(DMC) were examined. Pt(NH3)2(DMC) and Pt(R,R-DACH)(DMC) are the two DMC-integrated
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