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heart failure/nikotiin
Link salvestatakse lõikelauale
Leht 1 alates 326 tulemused
National Rate of Tobacco and Substance Use Disorders Among Hospitalized Heart Failure Patients.
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Electronic cigarette use among heart failure patients: Findings from the Population Assessment of Tobacco and Health study (Wave 1: 2013-2014).
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Usefulness of a history of tobacco and alcohol use in predicting multiple heart failure readmissions among veterans.
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Multiple hospital readmissions for heart failure (HF) are progressively increasing and may be related to continued tobacco and alcohol use. To study this relation, we conducted a retrospective chart audit of all veterans discharged with HF at a large Veterans Administration (VA) facility from 1997
Smokeless tobacco (snus) and risk of heart failure: results from two Swedish cohorts.
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BACKGROUND
Oral moist snuff (snus) is discussed as a safer alternative to smoking, and its use is increasing. Based on its documented effect on blood pressure, we hypothesized that use of snus increases the risk of heart failure.
METHODS
Two independent Swedish prospective cohorts; the Uppsala
Tobacco smoking in patients with heart failure and coronary artery disease: A 20-year experience at Duke University medical center
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Smoking is associated with incident heart failure (HF), yet limited data are available exploring the association between smoking status and long-term outcomes in HF with reduced vs. preserved ejection fraction (i.e., HFrEF vs. HFpEF).
Methods:
Tobacco smoking and the risk of heart failure: A systematic review and meta-analysis of prospective studies.
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Background We conducted a systematic review and meta-analysis to clarify the association between smoking and the risk of developing heart failure. Methods PubMed and Embase databases were searched up to 24 July 2018. Prospective studies were included if they reported adjusted relative risk (RR)
Factors affecting theophylline clearances: age, tobacco, marijuana, cirrhosis, congestive heart failure, obesity, oral contraceptives, benzodiazepines, barbiturates, and ethanol.
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Studies on water excretion following intravenous hydration and the administration of pitressin or nicotine in congestive heart failure.
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Differential effect of nicotine on plasma norepinephrine levels in normal humans and in patients with congestive heart failure.
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FDA in the 21st Century: Focus on Tobacco Policies and Heart Failure Prevention.
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Molecular basis of calpain cleavage and inactivation of the sodium-calcium exchanger 1 in heart failure.
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Cardiac sodium (Na(+))-calcium (Ca(2+)) exchanger 1 (NCX1) is central to the maintenance of normal Ca(2+) homeostasis and contraction. Studies indicate that the Ca(2+)-activated protease calpain cleaves NCX1. We hypothesized that calpain is an important regulator of NCX1 in response to pressure
Nicotine plus a high-fat diet triggers cardiomyocyte apoptosis.
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Cigarette smoking is an important risk factor for diabetes, cardiovascular disease and non-alcoholic fatty liver disease. The health risk associated with smoking can be aggravated by obesity. Smoking might also trigger cardiomyocyte (CM) apoptosis. Given that CM apoptosis has been implicated as a
The prevalence, aetiology and treatment of congestive cardiac failure in Antigua and Barbuda.
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A retrospective review of the cases of congestive heart failure admitted to Holberton Hospital in Antigua in 1995 and 1996 was undertaken. Two hundred and ninety-three (293) patients were identified by International Statistical Classification of Diseases, 10th revision (ICD-10) coding as having
Nicotine inhibits cardiac apoptosis induced by lipopolysaccharide in rats.
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OBJECTIVE
Apoptosis develops in several heart diseases, but the therapeutic options are limited. It was hypothesized that nicotine, which inhibits apoptosis in several cells, inhibits cardiac apoptosis induced by lipopolysaccharide (LPS).
BACKGROUND
Over-the-counter nicotine produces sustained
Human Immunodeficiency Virus and Heart Failure in Low- and Middle-Income Countries.
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Successful combination therapy for human immunodeficiency virus (HIV) has transformed this disease from a short-lived infection with high mortality to a chronic disease associated with increasing life expectancy. This is true for high- as well as low- and middle-income countries. As a result of this
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