hyperinsulinism/palavik

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Leht 1 alates 17 tulemused

Anemia and neutropenic fever with high dose diazoxide treatment in a case with hyperinsulinism due to Munchausen by proxy.

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BACKGROUND The etiology of hyperinsulinemic hypoglycemia in adolescents is similar to that of adults. Patients resistant to medical treatment may undergo pancreatectomy. Diazoxide is the mainstay of medical treatment. Rarely bone marrow suppression is reported due to diazoxide. METHODS An adolescent

Hyperinsulinism with fatal postoperative hyperthermia.

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Hyperinsulinemia elicited by interleukin-1 and nonlethal endotoxemia in rats.

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Nonlethal endotoxemia was produced in conscious fasted rats by the intravenous (i.v.) administration of Salmonella enteritidis lipopolysaccharide (LPS) at a dose of 30 micrograms/100 g together with the typical acute-phase response of fever at 4 hr post-LPS. Also at 4 hr post-LPS both

[Study on the mechanism of glucose intolerance and insulin resistance in epidemic hemorrhagic fever].

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Intravenous glucose tolerance test (ivGTT) and insulin releasing test (IRT) were performed in 32 cases with epidemic hemorrhagic fever (EHF). 23 patients had impaired glucose tolerance, 17 patients showed hyperinsulinemia in fasting state, 12 patients had flat insulin response curves and most

Central interleukin 1-elicited hyperinsulinemia is mediated by prostaglandins but not autonomics.

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This laboratory previously reported that centrally administered interleukin 1 (IL-1) in fasted pentobarbital-anesthetized rats elicited significant hyperinsulinemic and febrile responses. In characterizing this putative central mechanism for the regulation of pancreatic insulin secretion,

Nutritional Quality during Development Alters Insulin-Like Peptides' Expression and Physiology of the Adult Yellow Fever Mosquito, Aedes aegypti.

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Mosquitoes have distinct developmental and adult life history, and the vectorial capacity of females has been shown to be affected by the larval nutritional environment. However, little is known about the effect of developmental nutrition on insulin-signaling and nutrient storage. In this study, we

[Arterial hypertension, its metabolic aspects and kidney function in patients after hemorrhagic fever with renal syndrome].

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OBJECTIVE To characterize late convalescence after hemorrhagic fever with renal syndrome (HFRS), i.e. metabolic disorders and their relation with arterial pressure (AP) and renal function. METHODS 202 HFRS convalescents were followed up with measurements of AP, purin, carbohydrate and lipid

Effect of glucose infusion on the concentration of individual serum free amino acids during sandfly fever in man.

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After an intravenous glucose load in man, total serum amino acid concentrations are rapidly depressed and remain below baseline values for at least 2 to 3 hr after serum glucose and insulin have returned to preload concentrations. Despite the presence of basal hypoaminoacidemia, a decreased glucose

Euglycemic hyperinsulinemia alters the response to orthostatic stress in older adults with type 2 diabetes.

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OBJECTIVE Insulin has opposing influences on blood pressure by simultaneously increasing adrenergic activity and vasodilatating peripheral blood vessels. In this study, we sought to determine whether hyperinsulinemia affects tilt table responses in older adults with type 2 diabetes not complicated

Euglycemic hyperinsulinemia augments the cytokine and endocrine responses to endotoxin in humans.

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Type 2 diabetes is associated with biochemical evidence of low-grade inflammation, and experimental studies have suggested that both insulin and glucose affect inflammatory responses. To determine the effect of in vivo changes in glucose availability and plasma insulin concentrations in humans, we

Seizures and epilepsy in hypoglycaemia caused by inborn errors of metabolism.

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OBJECTIVE The aim of the study was to characterize seizures and epilepsy related to hypoglycaemia. METHODS We analyzed the files of 170 consecutive patients referred for hypoglycaemia (onset 1h to 4y) caused by inborn errors of metabolism (glycogen storage disease type I, fatty acid β-oxidation

The mitochondrial uncoupling protein-2: current status.

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In eukaryotic cells ATP is generated by oxidative phosphorylation, an energetic coupling at the mitochondrial level. The oxidative reactions occurring in the respiratory chain generate an electrochemical proton gradient on both sides of the inner membrane. This gradient is used by the ATPsynthase to

Role of prostaglandins in mediating alterations in glucose metabolism during acute endotoxemia in the rat.

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We determined the role of prostanoids in mediating alterations in glucose metabolism during lipopolysaccharide (LPS)-induced (1 mg/kg; LD10) acute endotoxemia in chronically catheterized awake rats. Basal glucose turnover (Rt, infusion of [5-3H]glucose), in vivo insulin action on overall glucose

Ion channels in disease.

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Diseases as different as cardiac arrhythmias, epilepsy, myotonia, malignant hyperthermia, familial hyperinsulinism, and Bartter syndrome have all been linked to mutations in genes encoding ion channels. This has been made possible by an exciting and fruitful collaboration between clinicians,

Effects of an endotoxin challenge on growth performance, carcass accretion rates, and serum hormone and metabolite concentrations in control pigs and those treated with recombinant porcine somatotropin.

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Barrows were restrictively fed starting at 20 kg BW to determine the effects of endotoxin on growth performance of control and somatotropin-treated pigs. The following treatments were used: 1) daily i.m. vehicle injection until 55 kg BW; 2) daily i.m. injections of 100 micrograms of recombinant

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