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lactate dehydrogenase/atrophy
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Leht 1 alates 797 tulemused
Lactate dehydrogenase and susceptibility to deterioration of mild COVID-19 patients: a multicenter nested case-control study
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Background: Coronavirus disease 2019 (COVID-19) has infected more than 4 million people within 4 months. There is an urgent need to properly identify high-risk cases that are more likely to deteriorate even if they present mild diseases
[FRACTIONATION OF SOLUBLE PROTEINS OF LACTATE DEHYDROGENASE AND GLUTAMIC-OXALOACETIC TRANSAMINASE OF LIVER TISSUE OF NORMAL SUBJECTS AND IN SUBJECTS AFFECTED BY ACUTE ATROPHY].
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Lactate dehydrogenase (LDH) isoenzymes in the retina of the sheep and changes associated with progressive retinal degeneration (Bright Blindness).
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[Changes in lactate dehydrogenase, glutamate-cxaloacetate transaminase and glutamate-pyruvate transaminase in several muscular atrophies. I. Changes following tenotomy].
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The activites of lactic dehydrogenase (LDH), glutamicoxalactic transaminase (GOT) and glutamic-pyruvic transaminase (GPT) are investigated within variable periods following tenotomy of m. gastrocnemius in the rat. A gradual fall of total activities is established, mostly significant for LDH, and
[Changes in lactate dehydrogenase, glutamate-oxaloacetate transmainase and glutamate-pyruvate transmainase activity in several muscular atrophies (I. Changes following denervation)].
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Changes in lactate dehydrogenase(LDH) isoenzymes associated with retinal degeneration of ewes affected with bright blindness.
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NEUROCHEMICAL STUDIES ON THE INHERITED RETINAL DEGENERATION OF THE RAT. I. LACTATE DEHYDROGENASE IN THE DEVELOPING RETINA.
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Lactate dehydrogenase forms in denervation and disuse atrophy of red and white muscle.
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Lactate dehydrogenase isoenzymes in muscular development of man and in nerve degeneration.
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MOLECULAR CHANGES OF LACTATE DEHYDROGENASE IN THE DEGENERATING PERIPHERAL NERVE.
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Regulation of membrane-mediated chronic muscle degeneration in dystrophic hamsters by calcium-channel blockers: diltiazem, nifedipine and verapamil.
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Membrane-mediated excessive intracellular calcium accumulation (EICA) is a fundamental pathogenetic event associated with chronic muscle degeneration in patients with Duchenne muscular dystrophy (DMD), and in animals with hereditary muscular dystrophy (HMD). Because of potential Ca(2+)-channel
Acute Babesia bovis infections: plasma creatine kinase, lactate dehydrogenase and creatinine levels and associated muscle damage.
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Two groups of five two-year-old Hereford steers were infected with two virulent strains of B. bovis. The plasma concentrations of creatine kinase (CK), lactate dehydrogenase (LDH), and creatinine rose early in infection and showed highly significant increases terminally. Extensive skeletal muscle
Animal models of amyotrophic lateral sclerosis and the spinal muscular atrophies.
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The causes of human amyotrophic lateral sclerosis (ALS) and the spinal muscular atrophies (SMA) are, almost without exception, unknown. This ignorance has stimulated the search for animal models to obtain insight into the etiology, pathogenesis and biochemical mechanisms underlying the human
Glutamine synthetase protects against neuronal degeneration in injured retinal tissue.
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The neurotransmitter glutamate is neurotoxic when it is accumulated in a massive amount in the extracellular fluid. Excessive release of glutamate has been shown to be a major cause of neuronal degeneration after central nervous system injury. Under normal conditions, accumulation of synaptically
[Histochemical study on disuse atrophy of skeletal muscle in rabbit (author's transl)].
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Histological and histochemical study was carried out to investigate disuse atrophy on skeletal muscle in adult albino rabbits. Untreated anterior tibial muscle and gastrocnemius muscle were studied. Three fiber types were recognized with myosin ATPase reaction, viz. type I, IIA and IIB fibers under
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