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charcoal/سویا

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مقالاتآزمایشات بالینیحق ثبت اختراع
صفحه 1 از جانب 31 نتایج

Phytotoxic Responses of Soybean (Glycine max L.) to Botryodiplodin, a Toxin Produced by the Charcoal Rot Disease Fungus, Macrophomina phaseolina.

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Toxins have been proposed to facilitate fungal root infection by creating regions of readily-penetrated necrotic tissue when applied externally to intact roots. Isolates of the charcoal rot disease fungus, Macrophomina phaseolina, from soybean plants in Mississippi produced a phytotoxic

Plant growth-promotion (PGP) activities and molecular characterization of rhizobacterial strains isolated from soybean (Glycine max L. Merril) plants against charcoal rot pathogen, Macrophomina phaseolina.

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Charcoal rot disease, caused by the fungus Macrophomina phaseolina, leads to significant yield losses of soybean crops. One strategy to control charcoal rot is the use of antagonistic, root-colonizing bacteria. Rhizobacteria A(5)F and FPT(7)21 and Pseudomonas sp. strain GRP(3) were characterized for

Toxin Production in Soybean (Glycine max L.) Plants with Charcoal Rot Disease and by Macrophomina phaseolina, the Fungus that Causes the Disease.

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Charcoal rot disease, caused by the fungus Macrophomina phaseolina, results in major economic losses in soybean production in southern USA. M. phaseolina has been proposed to use the toxin (-)-botryodiplodin in its root infection mechanism to create a necrotic zone in root tissue

Pelleting of soybean (Glycine max) seeds with activated charcoal and humus.

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First Report of Charcoal Rot Caused by Macrophomina phaseolina in Soybean in New York.

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Charcoal rot of soybean (Glycine max (L.) Merr.), incited by Macrophomina phaseolina (Tassi) Goidanich, is commonly found in much of the southern soybean production region of the United States, where it can be a major contributor to yield loss in warm, dry seasons (4). The disease has also been

First Report of Charcoal Rot Caused by Macrophomina phaseolina on Mungbean in China.

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Mungbean (Vigna radiata L.), an important leguminous food crop in China, is popularly grown in arid regions. The total area of mungbean production is 8.0 × 105 ha. In August and September 2010, wilted symptoms were observed in mungbean plants in Yulin, Shaanxi Province and Datong, Shanxi Province.

First Report of Charcoal Rot (Macrophomina phaseolina) on Soybean in Minnesota.

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In August 1999, soybean (Glycine max (L.) Merr.) plants exhibiting symptoms of charcoal rot were observed near Zumbrota, MN. Symptoms included shrunken, unfilled pods, and brown, wilted leaves attached to dead petioles and stems (1). When stems of symptomatic soybean plants were split, areas of

First Report of Charcoal Rot on Soybean Caused by Macrophomina phaseolina in North Dakota.

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In late August 2002, patches of soybean (Glycine max) plants in a field in Richland County, ND exhibited symptoms and signs of charcoal rot and died prematurely. Dead plants had a silvery-gray appearance, and black microsclerotia (76 ± 28 μm in diameter) were present in the vascular tissue of the

Effects of elevated O3 exposure on seed yield, N concentration and photosynthesis of nine soybean cultivars (Glycine max (L.) Merr.) in Northeast China.

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Nine soybean cultivars widely cultivated in Northeast China were investigated in present study to assess their O3 sensitivities on the basis of the response of photosynthesis and seed yield to ambient and future ozone (O3) concentrations, and determine whether the effects of O3 vary with the

Soybean-Macrophomina phaseolina-Specific Interactions and Identification of a Novel Source of Resistance.

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Charcoal rot, caused by the fungus Macrophomina phaseolina, is an economically important disease of soybean (Glycine max) worldwide. Objectives of the present research were to (i) study the genetic and pathogenic diversity in a collection of M. phaseolina isolates from Argentina and Paraguay and

Transcriptional Changes in Mycorrhizal and Nonmycorrhizal Soybean Plants upon Infection with the Fungal Pathogen Macrophomina phaseolina.

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Macrophomina phaseolina is a soil-borne fungal pathogen with a wide host range that causes charcoal rot in soybean [Glycine max (L.) Merr.]. Control of the disease is a challenge, due to the absence of genetic resistance and effective chemical control. Alternative or complementary measures are

Harpin-inducible defense signaling components impair infection by the ascomycete Macrophomina phaseolina.

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Soybean (Glycine max) infection by the charcoal rot (CR) ascomycete Macrophomina phaseolina is enhanced by the soybean cyst nematode (SCN) Heterodera glycines. We hypothesized that G. max genetic lines impairing infection by M. phaseolina would also limit H. glycines parasitism, leading to

Increase of apoplastic ascorbate induced by ozone is insufficient to remove the negative effects in tobacco, soybean and poplar.

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Apoplastic ascorbate (ASCapo) is an important contributor to the detoxification of ozone (O3). The objective of the study is to explore whether ASCapo is stimulated by elevated O3 concentrations. The detoxification of O3 by ASCapo was quantified in tobacco (Nicotiana L), soybean (Glycine max (L.)

Feeding and maturation by soybean looper (Lepidoptera: Noctuidae) larvae on soybean affected by weed, fungus, and nematode pests.

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Feeding and maturation by the soybean looper, Pseudoplusia includens (Walker) (Lepidoptera: Noctuidae), were investigated in a 2-yr study on 'Davis' soybean, Glycine max (L.) Merr., grown alone and combined with the weed hemp sesbania, Sesbania exaltata (Raf.) Rybd. ex. A. W. Hill, the root-knot

Maturation of soybean somatic embryos and the transition to plantlet growth.

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The maturation of soybean (Glycine max L. Merr.) somatic embryos was characterized. Maturation was assayed by evaluating the ability of somatic embryos to make the transition to a plantlet through a germination-like process. Somatic embryos were organized from cotyledons of immature soybean embryos.
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