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arenaria grandiflora/syöpä
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Sivu 1 alkaen 45 tuloksia
Effects of gonadal neoplasms on oogenesis in softshell clams, Mya arenaria.
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The average prevalence of gonadal neoplasms in softshell clams, Mya arenaria, from Whiting Bay, Washington County, Maine, was 19.4% in 1994. Monthly prevalences ranged from 10 to 26.7%. Neoplasms ranged in intensity from few, small foci of undifferentiated germ cells (Stage 1), to 50-100% of gonadal
Soft shell clams Mya arenaria with disseminated neoplasia demonstrate reverse transcriptase activity.
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Disseminated neoplasia (DN), a proliferative cell disorder of the circulatory system of bivalves, was first reported in oysters in 1969. Since that time, the disease has been determined to be transmissible through water-borne exposure, but the etiological agent has not been unequivocally identified.
Lack of detection of a putative retrovirus associated with haemic neoplasia in the soft shell clam Mya arenaria.
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Haemic neoplasia (HN) is a leukemia-like disease that affects at least 20 species of marine bivalves including soft shell clam, Mya arenaria. Since the disease was discovered in 1969, the etiology remains unknown. A retroviral etiology has been suggested based on the detection of reverse
Disseminated neoplasia in the soft-shell clam Mya arenaria: membrane lipid composition and functional parameters of circulating cells.
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In a previous study we compared lipid composition and functional parameters of circulating cells from Cerastoderma edule affected or not by disseminated neoplasia (neoplastic cells vs hemocytes) (Le Grand et al. Chem Phys Lipids 167:9-20 2013). Neoplastic cells presented morpho-functional
Prevalence of neoplasia in 10 New England populations of the soft-shell clam (Mya arenaria).
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Neoplasia was a prevalent disease of the soft-shell clam and was found in widely geographically distinct sites in New England. Two types of neoplasms were recognized. Most were of hematopoietic origin, except in clams from Maine, which also had gonadal neoplasms. Both types were apparently malignant
Potential link between exposure to fungicides chlorothalonil and mancozeb and haemic neoplasia development in the soft-shell clam Mya arenaria: a laboratory experiment.
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The aetiology of haemic neoplasia (HN) is unknown, so far but many causative factors are suggested such as viral, pollution and genetics. The aim of this study was to determine if, under chronic exposure, two major pesticides (chlorothalonil and mancozeb) which are used in potato production could
Selective initiation and transmission of disseminated neoplasia in the soft shell clam Mya arenaria dependent on natural disease prevalence and animal size.
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Disseminated neoplasia, a diffuse tumor of the hemolymph system, is one of the six most destructive diseases among bivalve mollusk populations, characterized by the development of abnormal, rounded blood cells that actively proliferate. Though the specific etiology of disseminated neoplasia in Mya
Experimental field studies with Mya arenaria (Bivalvia) on the induction and effect of hematopoietic neoplasia.
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A field experiment was conducted from 1991 to 1992 to examine induction and impact of hematopoietic neoplasia on the marine bivalve Mya arenaria in southeastern Massachusetts. Clams were collected from Little Buttermilk Bay and separated into three size classes (20-29, 30-39, and 40-49 mm shell
Field and laboratory comparisons of mortality in normal and neoplastic Mya arenaria.
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The results of a 6-month mark and recapture experiment involving approximately 900 adult Mya arenaria demonstrated that under natural conditions, significantly higher (P much less than .001, chi 2 test) mortality occurred among animals with neoplasia than those diagnosed as normal. Using a blood
Reverse transcriptase activity in tissues of the soft shell clam Mya arenaria affected with haemic neoplasia.
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Since all retroviruses possess reverse transcriptase (RT) enzyme, reverse transcriptase activity has been the main supportive evidence of retroviral etiology of haemic neoplasia (HN) in soft shell clams, Mya arenaria. The objective of the present study was to search for a putative retrovirus in
Unique antigens on neoplastic cells of the soft shell clam Mya arenaria.
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Soft shell clams (Mya arenaria) are commonly found in coastal waters of both the eastern and western United States. These invertebrates, which have an open circulatory system, may develop neoplasms of the haemolymph which ultimately kill the host. In this study we have 1) recorded the prevalence of
Assessment of haemic neoplasia in different soft shell clam Mya arenaria populations from eastern Canada by flow cytometry.
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Diagnosis of haemic neoplasia (HN) in the soft shell clam, Mya arenaria, is often achieved by hematocytology and histology. Since neoplastic cells display tetraploid DNA contents, haemocyte cell cycle analysis was developed for use as a diagnosis tool. The aim of this study was to assess the
Reverse transcriptase activity associated with haemic neoplasia in the soft-shell clam Mya arenaria.
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Reverse transcriptase (RT) activity has been reported in bivalves affected by haemic neoplasia (HN). Since all retroviruses have RT, detection of RT activity was regarded as evidence for the retroviral etiology of HN. This study investigates the relationship between RT levels and the progress of HN
Immunophenotyping of Mya arenaria neoplastic hemocytes using propidium iodide and a specific monoclonal antibody by flow cytometry.
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Disseminated neoplasia (DN) is a disorder referred to as hemic neoplasia (HN) in the soft-shell clam Mya arenaria. Traditionally, diagnosis is performed by hematocytology or histology. The intensity of the disease is generally given as the percentage of transformed neoplastic cells out of total
Transcriptome analysis of neoplastic hemocytes in soft-shell clams Mya arenaria: Focus on cell cycle molecular mechanism.
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In North America, a high mortality of soft-shell clams Mya arenaria was found to be related to the disease known as disseminated neoplasia (DN). Disseminated neoplasia is commonly recognized as a tetraploid disorder related to a disruption of the cell cycle. However, the molecular mechanisms by
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