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essential hypertension/nikotiini

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Sivu 1 alkaen 55 tuloksia

Effects of passive exposure to tobacco, socioeconomic status and a family history of essential hypertension on lipid profiles in children.

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There is abundant evidence that the atherosclerotic process begins in childhood. Dyslipidemia is a major risk factor for atherosclerosis in adults and children. In the present study, we measured serum lipoprotein concentrations in 194 healthy children aged between 4 to 14 years. Children were

[Evaluation of plasma endothelin-1 concentration in tobacco smoking patients with essential hypertension].

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An evident influence of nicotine on vasoconstrictive and mitogenic peptide-- endothelin-1 (ET-1) synthesis and expression of its receptors was observed in experimental investigations. The aim of this study was ET-1 concentration assessment in patients with essential arterial hypertension, regarding

In vitro effect of nicotine on red blood cell deformability in untreated and treated essential hypertension.

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The in vitro effect of nicotine (10(-7) mol l-1) on red blood cell (RBC) deformability in essential hypertension, efficacy of antihypertensive treatment (with the combination of enalapril, nifedipine and indapamid) on hypertension-induced decrease in RBC deformability and interaction between

Influence of hypertension, obesity and nicotine abuse on quantitative and qualitative changes in acute-phase proteins in patients with essential hypertension.

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BACKGROUND Hypertension is a powerful risk factor for cardiovascular disease and frequently occurs in conjunction with obesity. Accumulative evidence suggests a link between inflammation and hypertension. The aim of study was to evaluate whether blood pressure, obesity and smoking may influence

[Evaluation of salt consumption, physical activity, stress, tobacco and oral contraceptives in the epidemiology of essential hypertension].

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In the cross section study of a sample of 1,915 newly diagnosed cases of hypertension and 8,043 healthy individuals, a comparative analysis is made of different risk factors which can influence blood pressure counts. An association among the statistics for an excessive salt intake, stress, when the

Acute effects of low dose nicotine gum on platelet function in non-smoking hypertensive and normotensive men.

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Twenty non-smoking middle-aged men with mild untreated essential hypertension were compared to age-matched controls (n = 22) in a double-blind placebo controlled study. Plasma and urinary concentrations of the platelet-specific protein beta-thromboglobulin (beta-TG), platelet count and mean platelet

Functional promoter polymorphisms govern differential expression of HMG-CoA reductase gene in mouse models of essential hypertension.

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3-Hydroxy-3-methylglutaryl-coenzyme A [HMG-CoA] reductase gene (Hmgcr) is a susceptibility gene for essential hypertension. Sequencing of the Hmgcr locus in genetically hypertensive BPH (blood pressure high), genetically hypotensive BPL (blood pressure low) and genetically normotensive BPN (blood

[Influence of social support on patients with essential hypertension].

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OBJECTIVE To determine whether social support affects blood pressure control in patients diagnosed with essential Hypertension. METHODS Observational, prospective study, with a year's follow-up. SERRING: Mariñamansa-A Cuña Health Centre, Ourense (Galicia), Spain. METHODS 236 patients diagnosed with

Influence of combination of captopril and hydrochlorothiazide on plasma lipids, lipoproteins and apolipoproteins in primary hypertension.

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The effects of the combination of captopril and hydrochlorothiazide at modest doses of plasma lipids, lipoproteins and apolipoproteins were investigated in 139 patients with mild to moderate primary hypertension. Following an eight week wash-out period and a 4 week period of monotherapy with either

Significance of blood cadmium concentrations in patients with renal disorders or essential hypertension and the normal population.

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Cadmium concentrations in whole blood have been determined in normal control subjects, in patients with untreated essential hypertension or with treated essential hypertension, and in those with acute or chronic renal disorders. High cadmium concentrations were not found in the patients with

Effect of smoking on the course of essential hypertension: a follow-up study of a group composed predominantly of women.

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BACKGROUND The aim of this study was to compare the clinical course of essential hypertension between tobacco smokers and non-smokers. METHODS 64 health service workers with essential hypertension were studied. The assessment consisted of anamnesis, physical examination, fundoscopic examination,

Association of angiotensin-converting enzyme and angiotensin-converting enzyme-2 gene polymorphisms with essential hypertension in the population of Odisha, India.

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BACKGROUND Hypertension is a serious health issue worldwide and essential hypertension, which includes 90-95% of the cases, is influenced by both genetic and environmental factors. Identification of these factors may help in control of this disease. The Insertion/Deletion (I/D) polymorphism in

Environmental Tobacco Smoke Exposure Estimated Using the SHSES Scale and Epicardial Adipose Tissue Thickness in Hypertensive Patients

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The aim of the study was to assess the relationship between environmental tobacco smoke exposure (ETS) and epicardial adipose tissue thickness (EATT) in hypertensive patients. A total of 96 patients with essential hypertension were recruited for this study. The group consisted of 48 females and 48
The aim of the study was to determine the relationship between exposure to cigarette smoke and the morphology of atherosclerotic plaques in the extracranial arteries assessed by computed tomography angiography in patients with hypertension. The study included 61 hypertensive patients: 17 active
Four consecutive 24-h urine samples were collected from 134 male and 134 female placebo-treated patients in the Medical Research Council Trial for Mild Hypertension. Similar samples were collected from age and sex-matched normotensive controls. On the fourth day noradrenaline excretion was 22.05 +/-
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