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hypophosphatemia/carbohydrate
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Carbohydrate induced hypophosphatemia.
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[Hypophosphatemia and refeeding syndrome: a severe and underdiagnosed adverse effect].
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A 68-year-old woman was hospitalised because of generalised weakness and development of confusional state, related to severe hyponatremia, probably due to an eating disorder with malnutrition. During the first days of hospitalisation the patient eats surprising large amounts of food. The worsening
[Diabetes mellitus and carbohydrate metabolism in primary hyperparathyroidism].
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It is generally known that patients with primary hyperparathyroidism (pHPT) feature disturbances in carbohydrate metabolism and hypertension. The incidence and prevalence of frank diabetes mellitus is significantly increased in these patients. The etiology and pathogenesis of the vascular and
Refeeding syndrome and hypophosphatemia.
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Hypophosphatemia is a potentially life-threatening complication of reinstating nutrition in a malnourished patient. Refeeding syndrome is a term that refers to various metabolic abnormalities that may complicate carbohydrate administration in subnourished patient populations. Hypophosphatemia is the
Endocrine crises. Hypophosphatemia and hyperphosphatemia.
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Pathophysiology, clinical sequelae, and treatment for hypophosphatemia and hyperphosphatemia are discussed. Hypophosphatemia results from a variety of conditions including malnutrition, carbohydrate refeeding, acid-base disorders, and hormonal and drug effects. Patients suffering from severe
[The carbohydrate-energy metabolic and hemodynamic status of children with diffuse suppurative peritonitis in the early postoperative period].
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Thirty-three children with grave peritonitis were examined 16-24 h after operation. The groups were matched for the hemodynamic status. Group 1 were patients with moderate hyperdynamic status, group 2 those with the extreme hyperdynamic status, and group 3--patients with hypodynamic status. The
[Bodybuilding: hypokalemia and hypophosphatemia].
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In preparing for competitive body building, body builders--in addition to continuous and hard muscle training--engage in stringent dietetic manipulations: the first few months of hypercaloric nutrition, rich in proteins, are devoted to the build-up of muscle mass. A second phase of reduced caloric
Hypophosphatemia following self-treatment for anorexia nervosa.
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Hypophosphatemia is recognized as a serious complication of hospital treatment for patients with anorexia nervosa but may also occur prior to medical intervention.
METHODS
This paper reports a case of severe hypophosphatemia in a patient who consumed large quantities of carbohydrates following a
Hypophosphatemia and rhabdomyolysis.
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Clinical observations suggest that overt rhabdomyolysis may occur if severe hypophosphatemia is superimposed upon a pre-existing subclinical myopathy. To examine this possibility, a subclinical muscle cell injury was induced in 23 dogs by feeding them a phosphorus- and calorie-deficient diet until
Profound hypophosphatemia in the course of acute renal failure.
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Four of 19 patients with acute renal failure (ARF) requiring dialytic therapy were hypophosphatemic (serum phosphate, 0.8 +/- 0.8 mg/dL) during their illness. The hypophosphatemia could not be attributed to differences in the etiology of renal failure, frequency of dialysis, or residual renal
Hypophosphatemia in hospitalized patients.
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Hypophosphatemia is common in hospitalized patients and occurs under a variety of circumstances other than parathyroid hormone excess. Charts of 100 inpatients with hypophosphatemia were reviewed and the patients divided into five groups on the basis of serum phosphate level: 18, 2.1 to 2.4 mg/dL;
Severe hypophosphatemia following the institution of enteral feedings.
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Although severe hypophosphatemia has been recognized in refeeding syndromes, it is not a commonly reported complication of enteral nutrition. The present study was designed to identify cases of severe hypophosphatemia (less than 0.32 mmol/L [less than 1.0 mg/dL]) related to the administration of
Hypophosphatemia as unusual cause of ARDS in Cushing's syndrome secondary to ectopic CRH production. A case report.
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Hypophosphatemia is an unusual cause of acute respiratory distress syndrome (ARDS). We describe a hypophosphatemia-related ARDS case report of a 50-year-old woman with ACTH dependent Cushing's syndrome secondary to ectopic CRH production. The patient clinically showed hypotension tachypnea and
Peripheral insulin resistance in primary hyperparathyroidism.
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Carbohydrate metabolism was investigated in 9 patients with symptomatic primary hyperparathyroidism. Before and after parathyroidectomy intravenous and oral glucose tolerance test, tolbutamide test, arginine infusion test and insulin tolerance test were performed. During intravenous and oral glucose
Efficacy and safety of iron isomaltoside (Monofer(®)) in the management of patients with iron deficiency anemia.
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New intravenous (IV) iron preparations should ideally be capable of delivering a wide dosing range to allow iron correction in a single or low number of visits, a rapid infusion (doses up to 1,000 mg must be administered over more than 15 minutes and doses exceeding 1,000 mg must be administered
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