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peritonitis/hypoxia
Linkki tallennetaan leikepöydälle
Sivu 1 alkaen 63 tuloksia
Hepatic cellular hypoxia in murine peritonitis.
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Reduced oxygen consumption and lactic acidosis were observed frequently in patients with peritonitis. This study was designed to evaluate whether reduced oxygen consumption is secondary to deficient oxygen delivery or is a function of primary injury to mitochondria. Peritonitis was produced in rats
Sepsis is associated with altered cerebral microcirculation and tissue hypoxia in experimental peritonitis.
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OBJECTIVE
Alterations in cerebral microvascular blood flow may develop during sepsis, but the consequences of these abnormalities on tissue oxygenation and metabolism are not well defined. We studied the evolution of microvascular blood flow, brain oxygen tension (PbO2), and metabolism in a
[Initial reduction of the lung volume and capillary escape syndrome in gram negative peritonitis].
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Report on a female patient aged 29 with gramnegative bacterial peritonitis due to perforation of a postpyloric ulcer. A reduction of lung volume was observed, followed after 24 h by non cardiogenic interstitial and alveolar edema resulting in severe hypoxemia and hypercapnia with metabolic acidosis
Blockade of transdiaphragmatic lymphatic absorption reduced systemic inflammatory response syndrome during experimental peritonitis: evaluation with body oxygen kinetics in rats.
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OBJECTIVE
To assess the effect of blockade of transdiaphragmatic lymphatic absorption of infected peritoneal fluid on systemic inflammatory response syndrome during experimental peritonitis by evaluating body oxygen kinetics in rats.
METHODS
Randomised controlled experimental study.
METHODS
Teaching
Intramucosal pH and pCO(2) do not strictly correlate with intestinal energy metabolism in experimental peritonitis.
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This study aimed to investigate tissue hypoxia on the cellular level in sepsis. Eighteen pigs weighing 18-27 kg were studied. Intramucosal-arterial PCO(2) gradient (PCO(2)-gap) and intramucosal pH (pH(i)) were calculated using tonometry. A blind loop of the small intestine was constructed for
Perforation Peritonitis at High Altitude.
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Trekkers going to high altitude can suffer from several ailments both during and after their treks. Gastro-intestinal symptoms including nausea, vomiting, and abdominal pain are common in high altitude areas of Nepal due to acute mountain sickness or due to a gastro-intestinal illness. Occasionally,
[The effectiveness of the gas-exchange function of the lungs in diffuse peritonitis].
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Using a Douglas-Holden open system, gas exchange disturbances were studied and the role of gas exchange parameters in determination of gas exchange lung function was assessed in 39 patients with generalized peritonitis (GP). It has been established that in patients with GP the postoperative period
[The use of endovascular laser irradiation of the blood in the complex treatment of acute pancreatitis and peritonitis in experimental and clinical conditions].
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Experimental (118 experiments) and clinical (30 patients) investigations have shown a positive effect of endovascular irradiation of blood with the helium-neon and ultraviolet lasers on recovery of homeostasis in pancreatitis and peritonitis. The procedure stimulates the erythrocyte bioenergetics,
[Functional state of the lungs in toxemia in peritonitis and acute intestinal obstruction].
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On the basis of examination of 214 patients, it was established that the pronounced intoxication syndrome developing in peritonitis and acute ileus led to disorders in the non-respiratory pulmonary functions. This contributes to the development of arterial hypoxemia, hypocapnia and acute respiratory
Meconium peritonitis in stillbirths.
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Meconium peritonitis is a sterile, chemical peritonitis resulting from perforation of the bowel in perinatal life. In stillbirths meconium peritonitis is extremely rare. We report the autopsy findings in three fetuses ranging from 21-39 weeks gestation in which meconium peritonitis was identified.
[The concentration of end products of purine metabolism as possible signs of tissue hypoxia in critically ill surgical patients].
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The study was performed on 26 critically ill patients with hemorrhagic pancreonecrosis and disseminated suppurative peritonitis. Central hemodynamic and oxygen transport parameters have been determined in all the patients. To assess the degree of tissue hypoxia end products of ATP catabolism
Evaluation of the role of cellular hypoxia in sepsis by the hypoxic marker [18F]fluoromisonidazole.
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Underlying cellular hypoxia, which may be difficult to detect, has been postulated to be a major cause of morbidity and mortality in sepsis. We employed the novel hypoxic marker [18F]fluoromisonidazole to determine whether cellular hypoxia was present in a peritonitis model of sepsis in the rat. A
[Disorders of purine metabolism in patients with peritonitis complicated by sepsis].
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Progressive hypoxia and cell destruction leading to increased production of active oxygen forms by xanthinoxidase and manifesting by an increased level of uric acid in the blood in parallel with inhibited pentose cycle reaction due to low activity of glucose-6-phosphate dehydrogenase determine the
Peritonitis in Rwanda: Epidemiology and risk factors for morbidity and mortality.
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Few studies discuss causes and outcomes of peritonitis in low-income settings. This study describes epidemiology of patients with peritonitis at a Rwandan referral hospital. Identification of risk factors associated with mortality and unplanned reoperation could improve management of
[The state of the microsomal oxidative system in the rat liver during fecal peritonitis].
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Content and activity of cytochrome P-450, activity of main microsomal enzymes as well as content of glycogen, lactic and pyruvic acids were studied in liver tissue of rats within 24, 48 and 72 hrs after simulation of acute fecal peritonitis. Inhibition of the enzymatic activity of microsomal
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