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angioedema/albumine
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C1 inactivator protein complexed with albumin in plasma from a patient with angioneurotic edema.
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Albumin plasma exchange for life-threatening angioedema with normal C1-inhibitor.
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Urticaria and angioedema during insemination with fluid containing bovine serum albumin.
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Response of variant hereditary angioedema phenotypes to danazol therapy. Genetic implications.
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Hereditary angioedema (HAE), an auto-somal dominant disorder characterized by attacks of episodic edema is associated with decreased functional levels of the C1 esterase inhibitor. Approximately 85% of patients have lowered antigen levels of a normal inhibitor protein. 15% of patients have normal or
Allergic urticaria and angioedema caused by a hemostatic sponge of bovine fibrin used in tooth extraction.
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We report the case of a 54-year-old woman with no history of atopic diseases or drug allergies who developed a generalized anaphylactic reaction with urticaria and angioedema a few hours after tooth extraction. The skin scratch and RAST tests demonstrated IgE-mediated hypersensitivity to the product
Severe anaphylactic reaction to bovine serum albumin at the first attempt of artificial insemination.
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A 33-year-old woman without history of previous atopic diseases or drug allergies developed a severe anaphylactic reaction with asthma, vomiting, itching, generalized urticaria, and angioedema during artificial insemination with her husband's sperm. The sperm-processing medium contained bovine serum
Angioneurotic edema with acquired C1- inhibitor deficiency and autoantibody to C1- inhibitor: response to plasmapheresis and cytotoxic therapy.
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A patient with severe acquired angioneurotic edema had essentially no C1- inhibitor activity in his serum and nearly died of cardiopulmonary arrest during an acute episode of facial, oral, and pharyngeal edema. This patient had an antibody directed against C1- inhibitor and C1- inhibitor-anti-C1-
Recurrent angioedema caused by circulating immune complexes containing antibodies against bovine proteins.
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A 27-year-old woman with severe recurrent angioedema and urticaria since 5 years was found to have high levels of circulating immune complexes in the peripheral blood. These immune complexes contained antibodies against bovine serum albumin. Elimination of bovine products from the diet resulted in
Immunoreactive precipitation of C1 inhibitor protein from plasma of normal subjects and of patients with hereditary angioedema after isoelectric focusing.
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C1-inhibitor is an acid glycoprotein, isoelectric point 3.5-3.6. Plasma of some patients with a variant form of hereditary angioedema contains high levels of functionless C1-inhibitor-albumin complex with an isoelectric point at 4.5-4.6. Therapy with Danazol, which increases C1-inhibitor levels,
Delayed type III hypersensitivity reaction with acute kidney injury in two dogs following administration of concentrated human albumin during treatment for hypoalbuminemia secondary to septic peritonitis
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Objective: To describe 2 dogs with acute kidney injury secondary to type III hypersensitivity reaction to 25% human serum albumin (HSA).
Case series summary: Two dogs were
Administration of 5% human serum albumin in critically ill small animal patients with hypoalbuminemia: 418 dogs and 170 cats (1994-2008).
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OBJECTIVE
To describe the administration of 5% human serum albumin (HSA) in 588 critically ill hypoalbuminemic dogs and cats, and report outcome to discharge, changes in albumin concentration, and adverse effects during hospitalization.
METHODS
Retrospective clinical study.
METHODS
Private emergency
Angioedema and IgE antibodies to aspirin: a case report.
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We present a case of a patient who developed two episodes of angioedema after taking acetylsalicylic acid orally. Specific IgE antibodies to acetylsalicylic human serum albumin (ASA-HSA) were found. A control group of 37 patients with aspirin intolerance was studied and evidence of specific IgE
Novel pathogenic mechanism and therapeutic approaches to angioedema associated with C1 inhibitor deficiency.
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BACKGROUND
Activation of bradykinin-mediated B2 receptor has been shown to play an important role in the onset of angioedema associated with C1 inhibitor deficiency. This finding has led to the development of novel therapeutic drugs such as the B2 receptor antagonist icatibant. However, it is
A randomized, controlled trial to study the efficacy and safety of C1 inhibitor concentrate in treating hereditary angioedema.
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BACKGROUND
No effective treatment exists in the United States for acute attacks of hereditary angioedema (HAE).
METHODS
To evaluate the efficacy and safety of C1 inhibitor concentrate in treating HAE, a large primary care and referral center hospital conducted a randomized, placebo-controlled,
Quantitative studies of phagocytosis. Kinetic effects of cations and heat-labile opsonin.
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Kinetic analysis of the initial ingestion rate of albumin-coated paraffin oil particles by human granulocytes and rabbit alveolar macrophages was undertaken to study the mechanism of action of cations and of heat-labile opsonin on engulfment. The rate of uptake of the particles was stimulated by
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