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apudoma/sérotonine
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[A case of malignant pancreatic apudoma producer of serotonin and pancreatic polypeptide].
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Taking as their starting point the observation of a pancreatic malignant endocrine neoplasm with mixed production of serotonin and pancreatic polypeptide, the authors go on to review the literature on the diagnosis and treatment of pancreatic apudomas. The possibility of performing an extempore
[Primary hepatic apudoma with serotonin syndrome].
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Primary hepatic apudoma associated with serotonin syndrome was recorded in a man of 55 followed up for 6 years. Clinically, there was a progressive enlargement of the liver with varicosis of venae subcutaneae abdominis and venae esophageae. There were also weak signs of carcinoid syndrome. The
[Inhibition of pancreatic apudoma-dependent hypersecretion of gastrin, glucagon and serotonin using somatostatin-14].
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Iodine-123 MIBG imaging in a generalized pancreatic polypeptide-gastrin-serotonin secreting tumor.
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The usefulness of radio-metaiodobenzylguanidine (MIBG), a specific radiopharmaceutical agent for scintigraphic imaging and treatment of phaeochromocytoma and neuroblastoma, has been extended to the location of carcinoid tumors. Scintigraphic evaluation with I-123 MIBG in a patient with a
Relationships between neural crest cells and serotonin in newborn mice.
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The experimental newborn mice injected intraperitoneally with serotonin within 24 h after birth and survived up to 2-days showed multiple APUDomas, hyperplasia, heterotopic melanin pigmentation and the cell death of neural crest cells. Further, the degeneration occurred in the Auerbach, Meisner
Effects of catecholamine and serotonin in central nervous system in newborn mice with special reference to neural crest cells; presumptive evidence of neural crest origin.
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Catecholamine and serotonin injected i.p. to new-born mice caused gliomas, ependymomas and choroid plexus papilloma together with multiple APUDomas within 48 hours after injection. We consider that an error in neural crest DNA may be caused through an adenylate-cyclic AMP system and Ca++ during
[A poorly differentiated apudoma of the gallbladder].
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Apudoma was found in the gall bladder removed in a 76-year-old woman because of the chronic calculous cholecystitis exacerbation. Carcinoid syndrome was absent clinically. Histologically, the tumour was a poorly differentiated carcinoid with areas of small cell and polymorphic carcinoma.
[Apudoma of the prostate].
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Prostatic apudomas are represented by well-, moderately- and poorly differentiated variants. Histologic differentiation determines various functional properties of tumor cells. Tumor cells of well-differentiated apudomas produce serotonin, less frequently calcitonin, poorly differentiated ones
Pancreatic polypeptide as screening marker for pancreatic polypeptide apudomas in multiple endocrinopathies.
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Prospective screening was carried out in 12 members of three families with multiple endocrine adenopathies, type I (MEA,I) and in 14 patients with no multiple endocrine adenopathies with and without other endorcinopathies. Elevated basal and responsive (after a meal) plasma concentrations of a
Dopamine, norepinephrine and serotonin production by an intestinal carcinoid tumor.
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Substantial amounts of both dopamine and norepinephrine in addition to serotonin were found in a mesenteric metastasis of an ileal carcinoid tumor. Correspondingly, the norepinephrine-synthesizing enzymes were present in the tumor tissue and tyrosine hydroxylase was found in amounts substantially
[Transient diabetes insipidus following removal of a medullary thyroid carcinoma (author's transl)].
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Medullary thyroid carcinoma (MTC) is a known apudoma producing calcitonin, prostaglandins and serotonin. It can present itself as a familial or sporadic form or as part of a multiple endocrine adenomatosis. We present here the case of a patient admitted with a four-year history of diarrhea,
Carcinoid syndrome: an unusual cause of diarrhoea.
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A 9 year old girl underwent laparotomy because of intermittent diarrhoea, present since infancy. Histology of a mass at the head of pancreas and multiple hepatic nodules suggested an apudoma. Plasma serotonin and urinary excretion of 5 hydroxy indole acetic acid were raised. The child is
Physiology of the APUD system.
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The characteristic biochemical pathway of the APUDoma cell, namely amine precursor uptake and decarboxylation, are illustrated by the examples of serotonin and catecholamine metabolism. Increasing understanding of the origins of APUDomas as well as the biochemistry and physiology of the hormones
[A 2-component tumor of the breast with carcinoid syndrome].
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The structures of a well-differentiated apudoma were combined with the foci of colloid carcinoma in a mammary gland tumour described. Two types of cells with argyrophil grains are distinguished depending on the argyrophil grain size, localization in the tumour cell cytoplasm and the ability to react
Regulatory peptides and other neuroendocrine markers in medullary carcinoma of the thyroid.
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Medullary thyroid carcinoma (MTC) is an APUDoma (APUD refers to amine precursor uptake and decarboxylation) arising from the parafollicular cells. Diarrhoea has been reported in some 30% of patients, variously attributed to excess production of calcitonin (CT), serotonin (5-HT), vasoactive
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