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Fat necrosis of transverse colon and necrotizing pancreatitis in a patient with acute lymphoblastic leukemia (ALL): cause of massive ascites and high fever.
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We report a nine-year-old boy with acute lymphoblastic leukemia L3 (ALL-L3) and necrotizing pancreatitis in which necrosis was not limited to the pancreas. As our patient had a defective inflammatory process as a result of underlying malignant disease and neutropenia, the peripancreatic collection
Control by hyperthermia of ornithine decarboxylase in Ehrlich ascites tumor cells.
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The effect of hyperthermia on the activity and the messenger RNA levels of ornithine decarboxylase (ODC), which has a rapid rate of turnover in cultured cells, was studied in Ehrlich ascites tumor cells. When the cells were incubated at 42 degrees C, elevation of ODC activity by a change of the
[The recovery parameters of DNA strand breaks of Ehrlich ascites cells after the combined action of ionizing radiation and hyperthermia].
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A mathematical model of DNA strand breaks postirradiation repair and the methodology allowing to differentiate the mechanism of inhibition of DNA strand breaks recovery after combined actions of ionizing radiation and hyperthermia have been described in this paper. Using this model and the results
A rare case of typhoid presenting with fever, ascites, hyponatremia, thrombocytopenia, mesenteric lymphadenitis, and multi-drug resistance.
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A rare case of typhoid presenting with thrombocytopenia, hyponatremia, ascites mesenteric adenitis, and multi-drug resistance is being presented in this article. An 8-year-old girl was admitted with a history of fever, vomiting, abdominal pain and loose stools. Clinical examination revealed fever
Influence of hyperthermia and acidosis on the altered red blood cell osmotic fragility in mice with Ehrlich ascites tumor.
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Advanced cancer affects many body functions including red cell production and survival. The resulting anemia is probably due to membrane alterations. Red cells of Ehrlich ascites tumor (EAT)-bearing mice (EAT-RC) are known to be osmotically more stable than those of normal animals (N-RC). We
Enhancement of the in vitro cytotoxicity of bouvardin by verapamil alone and combined with hyperthermia in Sarcoma 180 and Ehrlich ascites carcinoma cells.
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The cytotoxic effect of bouvardin (BVD) a protein synthesis inhibitor was studied separately and in combination with verapamil (VRP), a vasodilator and hyperthermia (43 degrees C) against Sarcoma 180 (S 180) and Ehrlich ascites carcinoma (EAC) tumour cells in vitro. S 180 cells exhibited natural
Effect of hyperthermia and environmental acidity on the proteolytic activity in murine ascites tumor cells.
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The influence of hyperthermia and environmental pH on proteolytic activity was studied in murine ascites tumor cells in vitro. PNJ ascites tumor cells were incubated with [125I]cytochrome c at 42.5 or 37 degrees C in a modified Krebs-Ringer buffer adjusted to pH 7.2 or 6.4. Incubation at normal
Promotive effects of hyperthermia on the inhibition of DNA synthesis in ehrlich ascites tumor cells by eicosapentaenoic and docosahexaenoic acids.
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OBJECTIVE
To evaluate inhibitory effects of eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA) on DNA synthesis in combination with hyperthermia in vitro.
METHODS
A suspension of Ehrlich ascites tumor cells (EAT) was mixed with DHA or EPA in a glass tube, heated at 37 degrees C, 40 degrees
Enhancement of doxorubicin-induced cytotoxicity by hyperthermia in Ehrlich ascites cells.
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Hyperthermia (HPT) at 43 degrees C for 30 min increased the cytotoxic activity of doxorubicin against the growth of Ehrlich ascites carcinoma cells. There was more delay in tumor growth with 89% inhibition in the tumor volume and 90% increase in the survival of the tumor-bearing animals compared to
Right atrial myxoma presenting as post partum ascites and fever of unknown origin.
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A case is reported of right atrial myxoma presenting clinically as ascites and fever of unknown origin. An extensive work-up, including laboratory investigations, X-ray imaging, laparoscopy and laparotomy, failed to explain the clinical picture which was manifested post partum. Echocardiography was
Sonographic evidence of ascites, pleura-pericardial effusion and gallbladder wall edema for dengue fever.
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BACKGROUND
Radiographic findings of dengue fever have not yet been clearly elucidated in relation to clinical and serological findings, despite the fact that two-fifths of the world population lives in areas where the virus is endemic. The current study is a retrospective analysis of
Atypical presentation of intrahepatic cholangiocarcinoma---Fever and ascites in a postpartum lady.
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Intrahepatic cholangiocarcinoma is an uncommon malignancy which usually occurs in the 7th decade. Here we present a postpartum patient with fever, hepatomegaly, and ascites, who was diagnosed to have metastatic intrahepatic cholangiocarcinoma.
Effect of hyperthermia on electron transport in Ehrlich ascites tumor mitochondria.
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The effect of hyperthermia (1 hr, 41 degrees C) on the functional properties of Ehrlich ascites tumor mitochondria was investigated. Mitochondria isolated from Ehrlich ascites tumor after exposure of whole cells to 41 degrees C for 1 hr still phosphorylate and maintain a normal acceptor control
Familial Mediterranean fever with chronic ascites: a case report and a review of literature.
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Familial Mediterranean fever (FMF) is an autosomal recessive inflammatory disease especially seen in Turks, Sephardic Jews, Armenians, and Arabs. Peritoneal and pleural inflammation, arthritis, erysipelas-like erythema, and arthralgia are well-known features of FMF. A small amount of peritoneal
A rare cause of ascites: Familial Mediterranean fever.
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Familial Mediterranean fever is an autosomal recessive disorder characterized by sporadic, paroxysmal attacks of fever and serosal inflammation. In Familial Mediterranean fever, peritoneal effusion during abdominal attacks is usually mild, is not detected by clinical evaluation, and disappears
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