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atrioventricular block/hypoxie
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[RESPIRATORY ARRHYTHMIA AND RESPIRATORY ATRIOVENTRICULAR BLOCK IN HYPERCAPNIA AND HYPOXIA].
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[The electric excitability of the dog heart in normal stroke sequence, after artificially produced atrioventricular block and in anoxia].
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Hypoxia on hippocampal slices from mice deficient in dystrophin (mdx) and isoforms (mdx3cv).
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Slices from control C57, mdx, and mdx3cv mice were made hypoxic until both field excitatory postsynaptic potential (fEPSP) and presynaptic afferent volley (AV) disappeared (H1). After reoxygenation and recovery of fEPSP, a second and longer hypoxic test (H2) lasted 3 minutes beyond the time required
Enhanced arrhythmogenicity of Freon 113 by hypoxia in the perfused rat heart.
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The interaction of Freon 113 (1,1,2-trichloro-1,2,2-trifluoroethane) and hypoxia on the heart conduction system was studied using electrocardiogram monitoring of isolated perfused rat hearts. Freon 113 (0.2 mM) alone elicited significant atrioventricular conduction delay (p less than 0.05) and heart
Epileptic seizures secondary to high degree atrioventricular block without escape rhythm.
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Differentiation between cardiac and neurological origin of syncope may be challenging. Prolonged cerebral hypoxia secondary to cardiac arrhythmias may lead to epileptic seizures. Moreover, partial epileptic seizures by themselves can trigger cardiac arrhythmias. Herein, we present a case of partial
[Patent foramen ovale presented hypoxemia with cardiac sarcoidosis].
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A 53-year-old woman with a double, double, double (DDD) pacemaker due to complete atrioventricular block was admitted to our hospital with a diagnosis of congestive heart failure. At the time of admission, she was in a hypoxic state with cyanosis and clubbed finger. The ultrasonic cardiogram showed
Functional and electrophysiological effects of quinacrine on the response of ventricular tissues to hypoxia and reoxygenation.
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This study examined the effects of quinacrine on the functional and electrophysiological responses of isolated guinea pig hearts and isolated canine papillary muscle and Purkinje fibre preparations. A dose-response relationship for quinacrine (0.01-10.0 micrograms/mL) was studied in isolated guinea
Developmental changes in cardiac recovery from anoxia-reoxygenation.
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The developing cardiovascular system is known to operate normally in a hypoxic environment. However, the functional and ultrastructural recovery of embryonic/fetal hearts subjected to anoxia lasting as long as hypoxia/ischemia performed in adult animal models remains to be investigated. Isolated
Role of adenosine as mediator of bradyarrhythmias during hypoxia in isolated guinea pig hearts.
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Tissue concentrations of adenosine, an endogenous metabolite with negative chronotropic and dromotropic actions, are known to increase when myocardial oxygen supply is reduced. In this study the concentrations of endogenous adenosine released during a period of hypoxic perfusion were measured to
Endogenous adenosine, A1 adenosine receptor, and pertussis toxin sensitive guanine nucleotide binding protein mediate hypoxia induced AV nodal conduction block in guinea pig heart in vivo.
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OBJECTIVE
The aim was to study the mechanism of global hypoxia induced atrioventricular (AV) nodal conduction block in vivo using a guinea pig model.
METHODS
Animals subjected to 10 min periods of global hypoxia, induced by decremental changes in O2 content in the inhaled gas mixture, were randomly
Evidence for adenosine mediation of atrioventricular block in the ischemic canine myocardium.
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Adenosine levels in oxygen-deprived myocardium can rise to 10- 100 microM concentrations known to cause atrioventricular (AV) conduction delay and block. We reported that the AV conduction delay and block caused by hypoxia is markedly attenuated by 10 microM aminophylline, and adenosine competitive
Effects of ouabain and hypoxia on the cardiac stimulation threshold in the dog (with special reference to changes in extra- and intracellular potassium concentrations).
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The influence on the cardiac stimulation threshold of changes in the myocardial intra/extracellular potassium ratio induced with ouabain and hypoxia was investigated in 9 dogs with total atrioventricular block. In spite of significant elevations of extracellular potassium and reductions of
Role of aminophylline in atropine resistant atrioventricular block.
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Atrioventricular (AV) block may be induced by ischaemia as a result of production of adenosine, a metabolite that accumulates during hypoxia and ischaemia. Adenosine antagonism has been shown to reverse experimental AV node block in dogs. Recently, theophylline has been shown to be highly effective
Changes in the fetal heart rate and ECG during hypoxia.
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Previous reports on the fetal hypoxic bradycardia in animals have indicated, that there is vagal influence, especially when asphyxia is induced by umbilical cord occlusion. In the present study hypoxia was induced via the mother, thus keeping the fetal circulation intact. The experiments were
Interactive effects of mercury exposure and hypoxia on ECG patterns in two Neotropical freshwater fish species: Matrinxã, Brycon amazonicus and traíra, Hoplias malabaricus.
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Hypoxia and mercury contamination often co-occur in tropical freshwater ecosystems, but the interactive effects of these two stressors on fish populations are poorly known. The effects of mercury (Hg) on recorded changes in the detailed form of the electrocardiogram (ECG) during exposure to
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