chlamydia infections/hypoxie

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Chlamydia pneumoniae enhances Interleukin 8 (IL-8) production with reduced azithromycin sensitivity under hypoxia.

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Obligate intracellular bacterium Chlamydia pneumoniae causes respiratory tract infections such as community-acquired pneumonia. During infection, C. pneumoniae induces inflammatory responses in host cells and the oxygen concentration at the infection sites subsequently decreases. Because hypoxic

Hypoxia Promotes Chlamydia Trachomatis L2/434/Bu Growth in Immortal Human Epithelial Cells via Activation of the PI3K-AKT Pathway and Maintenance of a Balanced NAD ⁺/NADH Ratio

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Chlamydia trachomatis LGV (CtL2) causes systemic infection and proliferates in lymph nodes as well as genital tract or rectum producing a robust inflammatory response, presumably leading to a low oxygen environment. We therefore assessed how CtL2 growth in immortal human epithelial cells adapts to

Activities of first-choice antimicrobials against gamma interferon-treated Chlamydia trachomatis differ in hypoxia.

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Gamma interferon (IFN-γ)-mediated host responses play a central role in resolving genital Chlamydia trachomatis infections but may also result in persistence of the pathogen, which shows reduced susceptibility to antimicrobials. The antichlamydial function of IFN-γ is oxygen dependent, and the

Outbreak of Chlamydia pneumoniae infection in a Japanese nursing home, 1999-2000.

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OBJECTIVE To identify risk factors for infection and severe illness due to Chlamydia pneumoniae. METHODS To identify risk factors for infection, we conducted a case-control study among nursing home residents who had onset of symptoms during December 1, 1999, to February 20, 2000. To identify risk

Host immune responses after hypoxic reactivation of IFN-γ induced persistent Chlamydia trachomatis infection.

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Genital tract infections with Chlamydia trachomatis (C. trachomatis) are the most frequent sexually transmitted disease worldwide. Severe clinical sequelae such as pelvic inflammatory disease (PID), tubal occlusion, and tubal infertility are linked to inflammatory processes of chronically infected

[A case of severe interstitial pneumonia probably due to Chlamydia pneumoniae].

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A 64-year-old male was admitted to our hospital with dyspnea and high fever. The patient had a relative bradycardia and severe hypoxemia. Velcr rales were heard throughout the entire lung fields. Leucocytosis was absent. Chest X-ray showed bilateral diffuse reticular shadows. Corticosteroid pulse

[A case of severe Chlamydia pneumoniae pneumonia requiring mechanical ventilation and complicated with disseminated intravascular coagulation].

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A previously healthy 48-year-old man presented to his primary care physician with high fever, dry cough and dyspnea. Pneumonia was diagnosed and intravenous administration of imipenem/cilastatin was begun, but his respiratory condition worsened and he was admitted to our hospital with severe

Chlamydia pneumoniae directly interferes with HIF-1alpha stabilization in human host cells.

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Chlamydiaceae are obligate intracellular bacteria that cause endemic trachoma, sexually transmitted diseases and respiratory infections. The course of the diseases is determined by local inflammatory immune responses and the propensity of the pathogen to replicate within infected host cells. Both

Host metabolism promotes growth of Chlamydia pneumoniae in a low oxygen environment.

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Chlamydia pneumoniae infections of the respiratory tract are common and are associated with acute and chronic diseases such as community-acquired pneumonia (CAP) and chronic obstructive pulmonary disease (COPD). Recent studies have shown that reduced environmental oxygen availability promotes

The danger signal adenosine induces persistence of chlamydial infection through stimulation of A2b receptors.

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Infections with intracellular bacteria such as chlamydiae affect the majority of the world population. Infected tissue inflammation and granuloma formation help contain the short-term expansion of the invading pathogen, leading also to local tissue damage and hypoxia. However, the effects of key

[Severe chlamydia pneumoniae pneumonia requiring mechanical ventilation and steroid therapy in an elderly patient].

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A 75-year-old man first developed dyspnea and low-grade fever in late March. A chest X-ray film showed infiltration in the right lower lung field and blood gas analysis revealed severe hypoxemia. Accordingly, he was diagnosed as having pneumonia and was admitted to our hospital on March 11, 2003.

[Chlamydia trachomatis infection in the first year of life].

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BACKGROUND Chlamydia trachomatis (C. trachomatis) is the most frequent bacterial sexual infection in women. Pregnant women screening is controversial, with asymptomatic colonization occurring in 2-20%, and 40-70% newborns can be infected. OBJECTIVE The aim of this work is to characterise C.

Hypoxia abrogates antichlamydial properties of IFN-γ in human fallopian tube cells in vitro and ex vivo.

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IFN-γ has an important role in the adaptive immune response against intracellular pathogens. In urogenital tract (UGT) infections with the obligate intracellular pathogen Chlamydia trachomatis, IFN-γ-mediated control of chlamydial growth implies the JAK-STAT signaling cascades and subsequent

[Respiratory infection caused by Chlamydia pneumoniae].

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BACKGROUND Chlamydia pneumoniae is a pathogen usually associated with respiratory pathology. It has been estimated that this agent causes 5 to 15% of all pneumonias but its incidence among hospitalised children with respiratory infections is unknown. OBJECTIVE Characterization of the Chlamydia

Fatal hemorrhagic pneumonia concomitant with Chlamydia pneumoniae and parainfluenza virus 4 infection.

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BACKGROUND Cases of fatal hemorrhagic pneumonia need to be investigated for highly contagious viral causes. While not all hemorrhagic pneumonias are caused by very contagious agents, the etiology must be correctly determined in order to administer appropriate patient care. OBJECTIVE To determine

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chlamydia infections/hypoxie

Chlamydia pneumoniae enhances Interleukin 8 (IL-8) production with reduced azithromycin sensitivity under hypoxia.

Hypoxia Promotes Chlamydia Trachomatis L2/434/Bu Growth in Immortal Human Epithelial Cells via Activation of the PI3K-AKT Pathway and Maintenance of a Balanced NAD +/NADH Ratio

Activities of first-choice antimicrobials against gamma interferon-treated Chlamydia trachomatis differ in hypoxia.

Outbreak of Chlamydia pneumoniae infection in a Japanese nursing home, 1999-2000.

Host immune responses after hypoxic reactivation of IFN-γ induced persistent Chlamydia trachomatis infection.

[A case of severe interstitial pneumonia probably due to Chlamydia pneumoniae].

[A case of severe Chlamydia pneumoniae pneumonia requiring mechanical ventilation and complicated with disseminated intravascular coagulation].

Chlamydia pneumoniae directly interferes with HIF-1alpha stabilization in human host cells.

Host metabolism promotes growth of Chlamydia pneumoniae in a low oxygen environment.

The danger signal adenosine induces persistence of chlamydial infection through stimulation of A2b receptors.

[Severe chlamydia pneumoniae pneumonia requiring mechanical ventilation and steroid therapy in an elderly patient].

[Chlamydia trachomatis infection in the first year of life].

Hypoxia abrogates antichlamydial properties of IFN-γ in human fallopian tube cells in vitro and ex vivo.

[Respiratory infection caused by Chlamydia pneumoniae].

Fatal hemorrhagic pneumonia concomitant with Chlamydia pneumoniae and parainfluenza virus 4 infection.

La base de données d'herbes médicinales la plus complète soutenue par la science

Hypoxia Promotes Chlamydia Trachomatis L2/434/Bu Growth in Immortal Human Epithelial Cells via Activation of the PI3K-AKT Pathway and Maintenance of a Balanced NAD ⁺/NADH Ratio