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10 résultats
Method and reagent for monitoring apoptosis and distinguishing apoptosis from necrosis
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BACKGROUND OF THE INVENTION
1. Technical Field of the Invention
This invention relates generally to cytoenzymology, and more particularly to methods and reagents used in cytoenzymology.
2. Discussion of the Background Art
Apoptosis, or programmed cell death, is a process that involves the activation
Nucleic acid encoding modified human tnf.alpha. (tumor necrosis factor alpha) receptor
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The present invention relates to recombinant proteins and their use.
Tumour necrosis factor-.alpha. (TNF.alpha.) is a potent cytokine which elicits a broad spectrum of biological responses. TNF.alpha. causes the cytolysis or cytostasis of many tumour cell lines in vitro, induces the haemorrhagic
Soluble receptor BR43x2
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BACKGROUND OF THE INVENTION
Cellular interactions which occur during an immune response are regulated by members of several families of cell surface receptors, including the tumor necrosis factor receptor (TNFR) family. The TNFR family consists of a number of integral membrane glycoprotein receptors
Methods for inhibiting B lymphocyte proliferation with soluble ztnf4 receptor
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BACKGROUND OF THE INVENTION
Cellular interactions which occur during an immune response are regulated by members of several families of cell surface receptors, including the tumor necrosis factor receptor (TNFR) family. The TNFR family consists of a number of integral membrane glycoprotein receptors
DNA molecule encoding TNF binding ligands and vectors and host cells containing the DNA molecule
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FIELD OF THE INVENTION
The present invention relates to ligands to Tumor Necrosis Factor receptors (TNF-Rs) which inhibit the effect of TNF but not its binding to the TNF-Rs, as well as to ligands interacting with other receptors of the TNF/NGF family.
BACKGROUND OF THE INVENTION
Tumor necrosis
TNF ligands
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FIELD OF THE INVENTION
The present invention relates to ligands to Tumor Necrosis Factor receptors (TNF-Rs) which inhibit the effect of TNF but not its binding to the TNF-Rs, as well as to ligands interacting with other receptors of the TNF/NGF receptor family.
BACKGROUND OF THE INVENTION
Tumor
Compositions and methods for modulating responses mediated or associated with BTLA activity
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TECHNICAL FIELD
The invention relates to polypeptides that include a binding site for immunoregulatory molecule B-T lymphocyte attenuator (BTLA). Furthermore, the invention relates to ligands, such as antibodies, that bind to a binding site for immunoregulatory molecule B-T lymphocyte attenuator
Method of inhibiting the cytocidal effect of TNF with TNF receptor-specific antibodies
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FIELD OF THE INVENTION
The present invention relates to ligands to Tumor Necrosis Factor receptors (TNF-Rs) which inhibit the effect of TNF but not its binding to the TNF-Rs, as well as to ligands interacting with other receptors of the TNF/NGF family.
BACKGROUND OF THE INVENTION
Tumor necrosis
TNF receptor-specific antibodies
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FIELD OF THE INVENTION
The present invention relates to ligands to Tumor Necrosis Factor receptors (TNF-Rs) which inhibit the effect of TNF but not its binding to the TNF-Rs, as well as to ligands interacting with other receptors of the TNF/NGF family.
BACKGROUND OF THE INVENTION
Tumor necrosis
Cyclic, cystein-free protein
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The present invention relates to a cyclic protein free from cysteines which can be used as a medicament, e.g., for activating epithelial ion channels, for improving the lung function as well as for treating oedemas such as pulmonary oedemas.
The fluid transport through cell layers and tissue is
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