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diabetes insipidus/œdème

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Brain water accumulation (1.2%) with an accompanying increase in the sodium content was observed in Wistar rats as early as 1 hour after experimental subarachnoid hemorrhage (SAH). After 6 and 24 hours, the water content was 1.3 and 1.4%, respectively, higher than that of control animals. In
OBJECTIVE To evaluate the effect of idiopathic orthostatic edema and the effect of thyrotoxicosis on weight fluctuation and fluid retention in the presence of surgically induced panhypopituitarism and diabetes insipidus controlled with hormone replacement. METHODS Dextroamphetamine sulfate was used

[On the mechanism of appearance of edema in a rare type of diabetes insipidus].

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Diabetes insipidus associated with edema; report of a case, with discussion of the physiologic implications.

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[Induced diuresis for decrease of edema in diabetes insipidus with cardiac decompensation].

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Cerebral edema, diabetes insipidus, and sudden death during the treatment of diabetic ketoacidosis.

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[Acido-ketotic coma with diabetes insipidus of fatal outcome caused by cerbral edema].

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Partial nephrogenic diabetes insipidus associated with Castleman's disease.

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Nephrogenic diabetes insipidus (DI) secondary to a urinary tract obstruction is a rare condition. Herein, we report a case of partial nephrogenic DI due to obstructive uropathy in a patient with Castleman's disease.A 78-year-old man underwent computed

[MR imaging study of edema along the optic tract in patient with Rathke's cleft cyst].

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We report an 80-year-old woman who was admitted to our hospital with symptoms due to diabetes insipidus. Magnetic resonance (MR) imaging demonstrated a sellar/suprasellar cystic lesion with marginal enhancement and the thick pituitary stalk. The MR imaging depicted edema spreading along the optic

Neurogenic diabetes insipidus presenting in a patient with subacute liver failure: a case report.

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BACKGROUND To the best of our knowledge, this is the first report in the literature of development of neurogenic diabetes insipidus in a patient with subacute liver failure. METHODS A 25-year-old man presented with subacute liver failure. While awaiting a liver transplant, the patient developed

Central diabetes insipidus in pediatric severe traumatic brain injury.

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OBJECTIVE To determine the occurrence rate of central diabetes insipidus in pediatric patients with severe traumatic brain injury and to describe the clinical, injury, biochemical, imaging, and intervention variables associated with mortality. METHODS Retrospective chart and imaging

Diabetes insipidus in children with brain death.

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Central diabetes insipidus (DI) occurs in patients suffering from overwhelming CNS injuries leading to brain death. The purpose of this study was to describe the clinical presentation of DI in children with brain death. The medical records of 34 patients with a diagnosis of brain death were

Acute diabetes insipidus in severe head injury: a prospective study.

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BACKGROUND The incidence and risk factors for acute diabetes insipidus after severe head injury and the effect of this complication on outcomes have not been evaluated in any large prospective studies. METHODS We conducted a prospective study of all patients admitted to the surgical ICU of a Level I

Acute post-traumatic diabetes insipidus: treatment with continuous intravenous vasopressin.

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A young male presented within hours after closed head injury with hypotension, tachycardia, and polyuria. A diagnosis of post-traumatic diabetes insipidus was made. Although a rare entity, the rapid diagnosis of diabetes insipidus and early treatment with vasopressin may have been life-saving in
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