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diabetes insipidus/fièvre

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The different patterns of histiocytosis X are usually classified into three forms according to their clinical and pathological expression. One form is acute and disseminated ; the two others are chronic, unifocal or multifocal. Borderline cases and intermediate forms can be seen. For instance, onset

Diabetes insipidus: another cause of hormonal hyperthermia.

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Diabetes insipidus is not generally recognized as a possible cause of etiologically obscure fever. We describe a female patient who presented with fever and polyuria and was shown to have idiopathic partial neurogenic diabetes insipidus. The fever was subsequently demonstrated to be caused by

Dengue fever with diffuse cerebral hemorrhages, subdural hematoma and cranial diabetes insipidus.

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BACKGROUND Neurological manifestations in dengue fever occur in <1 % of the patients and known to be due to multisystem dysfunction secondary to vascular leakage. Occurrence of wide spread cerebral haemorrhages with subdural hematoma during the leakage phase without profound thrombocytopenia and

Hemorrhagic fever with renal syndrome accompanied by panhypopituitarism and central diabetes insipidus: a case report.

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Central diabetes insipidus (DI) was detected in a patient with hemorrhagic fever with renal syndrome (HFRS) who had been molecularly and serologically diagnosed with Hantaan virus infection. We recommend that clinicians differentiate central DI in HFRS patients with a persistent diuretic phase even

[Autopsy case of craniopharyngioma preceded by fever and diabetes insipidus].

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Typhus fever (Brill's disease) complicated by diabetes insipidus: report of a case.

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[Occult diabetes insipidus renalis as the cause of fever in an infant].

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Nephrogenic diabetes insipidus presenting as fever of unknown origin in the neonatal period.

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[Fever, cognition disorder and polyuria: diabetes insipidus caused by sarcoidosis].

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Cranial diabetes insipidus presenting as pyrexia of undetermined origin.

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[Nephrogenic diabetes insipidus as the cause of fever of unknown origin in infancy: report of a case].

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Identification of five novel arginine vasopressin gene mutations in patients with familial neurohypophyseal diabetes insipidus.

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Familial neurohypophyseal diabetes insipidus (FNDI) is a genetic disorder presenting with polyuria and polydipsia and is caused by mutations in the arginine vasopressin-neurophysin II (AVP-NPII) gene. The clinical manifestations of this disorder vary greatly depending on different mutations. The

Diabetes insipidus and anterior pituitary insufficiency as presenting features of Wegener's granulomatosis.

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Central diabetes insipidus (DI) is a rare complication of Wegener's granulomatosis (WG), which usually presents after pulmonary or kidney involvement. Anterior pituitary dysfunction secondary to WG has been extremely rare, documented in only three cases. We report a case of a 47-year-old

Giant cell myocarditis with central diabetes insipidus: A case report.

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A 51-year-old male, previously diagnosed with central diabetes insipidus due to lymphocytic hypophysitis, presented with fever and dyspnea for 1 week. On arrival, he exhibited hypotension (85/60 mmHg) and sinus tachycardia (110 bpm). His electrocardiogram revealed mild ST elevation on V2-V4.

Hypopituitarism associated with transient diabetes insipidus followed by an episode of painless thyroiditis in a young man.

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A 16-year-old male complained of a headache and a high fever followed by polyuria. The endocrinological studies showed he had hypopituitarism and central diabetes insipidus, and magnetic resonance imaging (MRI) revealed a pituitary mass. Diabetes insipidus gradually improved and hydrocortisone
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