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diabetic retinopathy/hypoxie
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MicroRNA-135b-5p promotes endothelial cell proliferation and angiogenesis in diabetic retinopathy mice by inhibiting Von Hipp-el-Lindau and elevating hypoxia inducible factor α expression
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Objective: This study was performed to investigate the effect of microRNA-135b-5p (miR-135b-5p) on endothelial cell proliferation and angiogenesis in diabetic retinopathy (DR) mice with the involvement of Von Hipp-el-Lindau protein (VHL) and hypoxia-inducible factor 1 α
[Study on risk factors of tissue hypoxia in patients with diabetic retinopathy].
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Risk factors of tissue hypoxia was investigated in 26 patients with diabetic retinopathy (DMR), who compared with 62 patients without DMR. significant higher incidence of DMR was found in patients with poor diabetic control (HbA 1 greater than or equal to 10%), with orthostatic hypotension (OH),
Time-dependent changes in hypoxia- and gliosis-related factors in experimental diabetic retinopathy.
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Diabetes causes various biochemical changes in the retina; long-term changes in the factors associated with hypoxia and gliosis have rarely been reported. The present study was conducted to explore the changes in these factors in a time-dependent manner in experimental diabetic retinopathy (DR).
Hypoxia-inducible factor-1α: A promising therapeutic target for vasculopathy in diabetic retinopathy
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Diabetic retinopathy (DR) is a serious condition that can cause blindness in diabetic patients. It is a neurovascular disease, but the pathogenesis leading to the onset of this disease is still not completely understood. However, hypoxia with subsequent neovascularization is a characteristic
Hypoxia and oxidative stress in the causation of diabetic retinopathy.
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Diabetic retinopathy is associated with hyperglycemia, and there is convincing evidence that oxidative stress (the overproduction of reactive oxygen species (ROS)) measured indirectly in patients, is related to the severity of diabetic complications. Also, reducing such stress by various means,
Nocturnal intermittent serious hypoxia and reoxygenation in proliferative diabetic retinopathy cases.
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OBJECTIVE
To clarify the relationship between evaluation items of sleep-disordered breathing and diabetic retinopathy in detail.
METHODS
Cross-sectional comparative study.
METHODS
Sixty-eight consecutive nonproliferative diabetic retinopathy and 151 proliferative diabetic retinopathy (PDR) cases who
Interactions between hyperglycemia and hypoxia: implications for diabetic retinopathy.
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The primary aim of these experiments was to assess in vitro effects of hyperglycemia (30 mmol/l glucose) and hypoxia (Po(2) = 36 torr) of 2-h duration, separately and in combination, on cytosolic and mitochondrial free NADH (NADHc and NADHm, respectively) in retinas from normal rats. NADH is the
Intravitreous vascular endothelial growth factor and hypoxia-inducible factor 1a in patients with proliferative diabetic retinopathy.
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OBJECTIVE
To determine vascular endothelial growth factor (VEGF) and hypoxia-inducible factor 1a (HIF-1a) in the vitreous fluid of patients with proliferative diabetic retinopathy (PDR).
METHODS
Observational case-control study.
METHODS
Serum and vitreous fluid samples were obtained during
Ischaemia-induced retinal neovascularisation and diabetic retinopathy in mice with conditional knockout of hypoxia-inducible factor-1 in retinal Müller cells.
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OBJECTIVE
Retinal Müller cells are known to produce inflammatory and angiogenic cytokines, which play important roles in diabetic retinopathy. Hypoxia-inducible factor (HIF)-1 has been shown to play a crucial role in retinal inflammation and neovascularisation. We sought to determine the role of
Proliferative diabetic retinopathy: predictive and preventive measures at hypoxia induced retinal changes.
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Retinal vasculature changes in diabetic patients are most common cause of blindness among eye diseases. Numerous studies have explored the role of the agiogenic factors in the progression of diabetic retinopathy (DR). The balance between angiogenic and antiangiogenic factors has a determining role
Affinity hypoxia as a pathogenetic factor of microangiopathy with particular reference to diabetic retinopathy.
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The microvascular complications in the retina and kidneys are preceded and accompanied by functional abnormalities. The appearance of the first retinal responses, venous dilatation associated with increased plasma permeation, suggest a state of tissue hypoxia located close to the venous
The potential association between obstructive sleep apnea and diabetic retinopathy in severe obesity-the role of hypoxemia.
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BACKGROUND
Obstructive sleep apnea (OSA) is common in obese patients with type 2 diabetes mellitus (DM) and may contribute to diabetic microvascular complications.
METHODS
To investigate the association between OSA, hypoxemia during sleep, and diabetic retinal complications in severe obesity. This
[Diabetic retinopathy. Pathophysiology and therapy of hypoxia-induced inflammation].
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Diabetic retinopathy is the most common chronic microvascular complication associated with diabetes mellitus. The development of diabetic retinopathy is a consequence of metabolic dysregulation. Hyperglycemia is a critical factor which is involved in basement membrane thickening, loss of pericytes
Role of oxidative stress, inflammation, hypoxia and angiogenesis in the development of diabetic retinopathy.
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Diabetic retinopathy (DR) is a retinal disease which is one of the most severe complications occuring due to diabetes mellitus and is a major cause of blindness. Patients who have diabetes mellitus for number of years develop characteristic group of lesions in the retina which leads to Diabetic
Macular edema-ischemia makes a vicious cycle in diabetic retinopathy : role of retinal hypoxia in diabetic macular edema.
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