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disseminated intravascular coagulation/œdème
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Bacillus anthracis cell wall peptidoglycan but not lethal or edema toxins produces changes consistent with disseminated intravascular coagulation in a rat model.
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BACKGROUND
Disseminated intravascular coagulation (DIC) appears to be important in the pathogenesis of Bacillus anthracis infection, but its causes are unclear. Although lethal toxin (LT) and edema toxin (ET) could contribute, B. anthracis cell wall peptidoglycan (PGN), not the toxins, stimulates
Late-onset pulmonary edema and disseminated intravascular coagulation due to latex anaphylaxis.
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BACKGROUND
Anaphylaxis after the administration of tissue expanders has been reported. Late onset hypotension, thrombocytopenia, disseminated intravascular coagulation (DIC) and pulmonary edema due to the tissue expanders have not been reported.
METHODS
In this case report, late onset hypotension,
Falciparum malaria in an overseas traveler complicated by disseminated intravascular coagulation and pulmonary edema.
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A 24-year-old woman was infected with falciparum malaria during travel to Kenya, complicated by intravascular coagulation and pulmonary edema. She was successfully treated with anti-malarial drugs including chloroquine, quinine sulfate and pyrimethamine, with a combined regimen of heparin,
[Pulmonary edema due to shock, fat embolism, disseminated intravascular coagulation and post extracorporeal circulation].
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The appearance of lesional edema in the region of the alveolar wall constitues the initial phenomenon of involvement of the pulmonary parenchyma during shock lung, fat embolism, DIC and post perfusion lung syndrome. The alveolar septum reacts in a monomorphous manner to these various agressions,
[P. falciparum and P. malariae malaria complicated by pulmonary edema with disseminated intravascular coagulation].
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Pulmonary edema and disseminated intravascular coagulation after intravenous abuse of d-propoxyphene (darvon).
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Hydrops fetalis attributable to intrauterine disseminated intravascular coagulation.
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Hyperthermia, pulmonary edema, and disseminated intravascular coagulation in an 18-year-old military recruit.
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Disseminated intravascular coagulation and fetal hydrops in a newborn infant in association with a chorangioma of placenta.
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Disseminated intravascular coagulation in the pathogenesis of adult respiratory distress syndrome: 2. Experimental study.
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The role of disseminated intravascular coagulation (DIC) in the pathogenesis of adult respiratory distress syndrome (ARDS) was studied in the experimental animals. ARDS was simulated in dogs by the administration of various doses of Escherichia coli endotoxin (Difco). The alveolar surface activity
[Disseminated intravascular coagulation and its pathogenetic significance in meningoencephalitis].
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Combined investigation was performed in 370 patients with meningoencephalitis caused by different etiological factors. This comprised the clinical, laboratory, pathological (in 20 deceased patients) studies aimed at elucidating the role of cerebral microcirculation (CM) disorders in the pathogenesis
Consumptive coagulopathy in utero associated with multiple vascular malformations.
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We report a postmature male infant who died at age 2 h of asphyxiating pulmonary hemorrhage. Autopsy found edema, serous ascites, and vascular malformations within myocardium, lung, mediastinal soft tissue, thoracic-wall skeletal muscle, spleen, thyroid and adrenal glands, and pancreas. The
Disseminated Intravascular Coagulation-like Reaction after Rituximab Infusion in a Patient with Nephrotic Syndrome.
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Several case series have suggested that rituximab is efficacious in adult patients with minimal change disease. We herein report a case of disseminated intravascular coagulation-like reaction after rituximab infusion in a patient with nephrotic syndrome. A 58-year-old Japanese man with minimal
miR-96 and miR-330 overexpressed and targeted AQP5 in lipopolysaccharide-induced rat lung damage of disseminated intravascular coagulation.
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Disseminated intravascular coagulation (DIC) is a severe clinical condition that can lead to or aggravate the development of multiple organ dysfunction syndrome. Of all types of organ damage, lung damage is the most frequent and most severe. In DIC patients, lung damage is primarily characterized by
Case report--a neonate with nonimmune hydrops fetalis.
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A post-dated intra-uterine growth retarded male Malay baby was born to a 30-year-old mother gravida II by Caesarean section. Her previous pregnancy ended in still-birth. The baby was severely asphyxiated at birth. He was intubated and immediately admitted to the neonatal intensive care unit. He had
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