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Evidence of involvement of tumor necrosis factor in adverse reactions during treatment of kidney allograft rejection with antithymocyte globulin.
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Serial plasma concentrations of the pyrogenic cytokines tumor necrosis factor and interleukin-1 beta were measured during treatment of acute renal allograft rejection with antithymocyte globulin in 7 consecutive kidney transplant recipients. TNF and IL-1 beta were measured with specific
Alpha-globulins suppress human leukocyte tumor necrosis factor secretion.
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The secretion of tumor necrosis factor (TNF) by human peripheral blood mononuclear cells was suppressed by either whole human plasma alpha-globulins or purified alpha 1-acid-glycoprotein, alpha 1-antitrypsin and alpha 2-macroglobulin in a concentration-dependent manner. alpha 1-Antitrypsin was found
Anthropometric, computed tomography and fat cell data in an obese population: relationship with insulin, leptin, tumor necrosis factor-alpha, sex hormone-binding globulin and sex hormones.
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OBJECTIVE
To correlate anthropometric, computed tomography and fat cell data from abdominal regions with the levels of serum insulin, C-peptide, leptin, tumor necrosis factor-alpha (TNF-alpha), testosterone, 17beta-estradiol, androstenedione, dehydroepiandrosterone sulphate (DHEA-S) and sex
Anti-tumour necrosis factor antibody treatment does not change serum levels of cortisol binding globulin in patients with rheumatoid arthritis but it increases androstenedione relative to cortisol.
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BACKGROUND
Cortisol binding globulin (CBG) is produced by liver cells and is inhibited by proinflammatory cytokines such as interleukin (IL) 6. CBG serum levels are typically low during prolonged inflammatory processes. Thus, observed changes of cortisol during anti-tumour necrosis factor (TNF)
Avascular necrosis after treatment of aplastic anaemia with antilymphocyte globulin and high-dose methylprednisolone.
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Avascular necrosis of bone (AVN) occurring in patients with aplastic anaemia (AA) treated with antilymphocyte globulin (ALG) followed by high-dose methylprednisolone (HDMP) has been studied retrospectively. Out of 49 patients treated at two centres, seven have developed AVN at a median of 14 months
Potential role of tumor necrosis factor-α in downregulating sex hormone-binding globulin.
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Low plasma sex hormone-binding globulin (SHBG) levels are associated with obesity and predict the development of type 2 diabetes. The reason why obese individuals have low circulating SHBG has been attributed to hyperinsulinemia, but no mechanistic evidence has been described. The aim of the current
[Elevation of antihemophilic globulin (factor 8) in lethal liver necrosis].
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Raised serum tumour necrosis factor in delayed antilymphocyte globulin adverse reactions.
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Local formation of gamma-globulin in the diseased liver, and its relation to hepatic necrosis.
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Liver histochemistry (mucopolysaccharides) and plasma alpha3-globulin; studies in diffuse massive (fulminant viral hepatitis) and zonal liver necrosis (toxic hepatitis).
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Coagulation changes during massive hepatic necrosis due to Amanita phalloides poisoning with special reference to antihemophilic globulin.
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Arteritis, carditis, glomerulonephritis and bilateral renal cortical necrosis induced in rabbits by injection of horse serum or bovine gamma globulin combined with killed group A Streptococci or Freund's adjuvant.
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Sex hormone binding globulin and anti-tumor necrosis factor-α therapy.
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Massive necrosis of the brain in rabies.
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A young man developed virologically proved hydrophobic rabies three months after being scratched on the cheek by a bat in Northern Alberta. He became comatose after 8 days and died 5 weeks after vigorous therapy with immune globulin, interferon and Vidarabine, and excellent maintenance of
[A case of acute retinal necrosis syndrome caused by herpes simplex virus type 1].
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A case of bilateral acute retinal necrosis syndrome accompanied by viral meningitis in a 46 year-old male is reported. The characteristic scattered yellowish-white retinal exudates and retinal detachment appeared in both eyes within 2 weeks, and after treatment with intravenous acyclovir and
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