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glutathione reductase/hémorragie
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Picroliv modulates antioxidant status and down-regulates AP1 transcription factor after hemorrhage and resuscitation.
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Resuscitation from hemorrhagic shock initiates profound changes in the liver that are likely to contribute to end organ damage and resultant dysfunction after shock. Extensive research in this area has indicated the potential of free radical scavenging strategy for better management of the
Inhibition of BECN1 Suppresses Lipid Peroxidation by Increasing System Xc- Activity in Early Brain Injury after Subarachnoid Hemorrhage.
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Lipid peroxidation plays a crucial role in early brain injury (EBI) after subarachnoid hemorrhage (SAH), and cystine/glutamate antiporter system Xc- has been proved to be associated with glutathione (GSH) synthesis, which protects cells against oxidative damage. Antioxidant
Oxygen-derived free radicals in kidney in experimental hemorrhagic shock treated by PAF-receptor antagonist BN 52021.
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Renal damage in rat hemorrhagic shock model was assessed by estimation of the reactive oxygen metabolites generation (malondialdehyde measured as thiobarbituric acid reactive substances) and antioxidative potential (activity of Cu-, Zn- superoxide dismutase, activity of glutathione reductase and
Comparison of protein carbonyl and antioxidant levels in brain tissue from intracerebral haemorrhage and control cases.
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BACKGROUND
In an attempt to develop a clearer understanding of the pathological mechanisms underlying intracerebral haemorrhage (ICH), the objective of this investigation was to obtain evidence for free radical-induced oxidative damage in brain tissue following intracerebral
[Effect of pyruvate on metabolic processes in erythrocytes after acute massive blood loss].
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It is shown in the in vitro experiments that addition into the incubation medium of 10 mumol pyruvate is accompanied by the inhibition of the lactate dehydrogenase activity. The level of 2.3-biphosphoglycerate, ATP, lactate and hemoglobin increases by 92%, 265%, 93% and 56%, respectively. The acute
Plasma values of oxidants and antioxidants in acute brain hemorrhage: role of free radicals in the development of brain injury.
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The levels of oxidants xanthine oxidase (XO), nitric oxide (NO), and malondialdehyde (MDA) and of the antioxidant enzymes superoxide dismutase (SOD), glutathione peroxidase (GSH-Px), and glutathione reductase (GRD) were determined in plasma within 24 h after onset of hemorrhagic stroke in 17
Role of reperfusion with polyelectrolyte solution and BN 52021 in hemorrhagic shock-induced changes in rat lungs.
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The aim of this study was to test whether reperfusion with isotonic standard polyelectrolyte solution alone has any influence on the redox balance in the lung during hemorrhagic shock, and whether reperfusion and BN 52021, used together have better protective effect on free radical lung injury. The
Protective effect of aminoguanidine against oxidative stress and bladder injury in cyclophosphamide-induced hemorrhagic cystitis in rat.
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Cyclophosphamide (CP) is an antineoplastic agent that is used for the treatment of many neoplastic diseases. Hemorrhagic cystitis (HC) is a major dose limiting side effect of CP. Recent studies show that aminoguanidine, an inhibitor of inducible nitric oxide synthase is a potent antioxidant and
BN 52021, PAF-receptor antagonist, improves diminished antioxidant defense system of lungs in experimentally induced haemorrhagic shock.
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The purpose of our experiments was to determine whether BN 52021, PAF-receptor antagonist, has any influence on the production of reactive oxygen species in the lung induced by haemorrhagic shock. The results showed, that the activities of Cu,Zn-superoxide dismutase (SOD) and glutathione peroxidase
Diallyl Disulfide Prevents Cyclophosphamide-Induced Hemorrhagic Cystitis in Rats through the Inhibition of Oxidative Damage, MAPKs, and NF-κB Pathways.
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This study investigated the possible effects and molecular mechanisms of diallyl disulfide (DADS) against cyclophosphamide (CP)-induced hemorrhagic cystitis (HC) in rats. Inflammation response was assessed by histopathology and serum cytokines levels. We determined the protein expressions of nuclear
Pyruvate alleviates lipid peroxidation and multiple-organ dysfunction in rats with hemorrhagic shock.
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OBJECTIVE
Pyruvate can reduce lipid peroxidation, which plays a critical role in organ injury, in various models. However, it is not fully understood if this inhibition occurs in resuscitation of hemorrhagic shock (HS). This study examines effects of pyruvate Ringer solution (PR) in this respect in
Histopathological and biochemical changes in goldfish kidney due to exposure to the herbicide Sencor may be related to induction of oxidative stress.
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Molecular mechanisms of toxicity by the metribuzin-containing herbicide Sencor to living organisms, particularly fish, have not yet been extensively investigated. In the present work, we studied the effects of 96 h exposure to 7.14, 35.7, or 71.4 mg L(-1) of Sencor (corresponding to 5, 25, or 50 mg
Protective effects of pine bark extract on hexavalent chromium-induced dermatotoxicity in rats.
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The present study investigated the protective effects of pine bark extract (PBE) against hexavalent chromium [Cr(VI)]-induced dermatotoxicity in rats. Skin reactions were evaluated by visual inspection, histopathological changes and oxidative stress parameters. Topical application of Cr(VI) produced
A Study on Neuroprotective Effects of Curcumin on the Diabetic Rat Brain.
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The present study was aimed to study the neuroprotective therapeutic effect of curcumin on the male albino rat brain. Subarachnoid hemorrhage leads to severe mortality rate and morbidity, and oxidative stress is a crucial factor in subarachnoid hemorrhage. Therefore, we investigated the effect of
Mechanism for the protective effect of diallyl disulfide against cyclophosphamide acute urotoxicity in rats.
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This study investigated the protective effects of diallyl disulfide (DADS) against cyclophosphamide (CP)-induced acute urotoxicity in rats. CP caused severe hemorrhagic cystitis as shown by significant increases in bladder weight, edema, and hemorrhage as well as increased urinary bladder epithelial
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