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hemangioma/l tyrosine
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Gene therapy for inhibition of angiogenesis
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FIELD OF THE INVENTION
The present invention relates to methods of gene therapy for inhibiting angiogenesis associated with tumor growth, inflammation, psoriasis, rheumatoid arthritis, hemangiomas, diabetic retinopathy, angiofibromas, and macular degeneration.
This invention also relates to animal
Substituted phenylacrylonitrile compounds and compositions thereof for the treatment of disease
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1. INTRODUCTION
The present invention relates to novel compounds capable of modulating and/or regulating tyrosine kinase signal transduction. The Applicants have demonstrated that the murine fetal liver kinase 1 (FLK-1) receptor and its non-murine counterparts, including the human Kinase
Compounds for the treament of disorders related to vasculogenesis and/or angiogenesis
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1. INTRODUCTION
The present invention relates to novel compounds capable of modulating and/or regulating tyrosine kinase signal transduction. The Applicants have demonstrated that the murine fetal liver kinase 1 (FLK-1) receptor and its non-murine counterparts, including the human Kinase
Compounds for the treatment of disorders related to vasculogenesis and/or angiogenesis
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1. INTRODUCTION
The present invention relates to novel compounds capable of modulating and/or regulating tyrosine kinase signal transduction. The Applicants have demonstrated that the murine fetal liver kinase 1 (FLK-1) receptor and its non-murine counterparts, including the human Kinase
Compounds for the treatment of disorders related to vasculogenesis and/or angiogenesis
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1. Introduction
The present invention relates to novel compounds capable of modulating and/or regulating tyrosine kinase signal transduction. The Applicants have demonstrated that the murine fetal liver kinase 1 (FLK-1) receptor and its non-murine counterparts, including the human Kinase
Quinazoline compounds and compositions thereof for the treatment of disease
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1. INTRODUCTION
The present invention relates to novel compounds capable of modulating and/or regulating tyrosine kinase signal transduction. The Applicants have demonstrated that the murine fetal liver kinase 1 (FLK-1) receptor and its non-murine counterparts, including the human Kinase
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