hemifacial spasm/hémorragie

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Treatment of Venous Hemorrhage Between Vestibulocochlear Nerve and Hypertrophic Flocculus During Microvascular Decompression Procedure for Hemifacial Spasm.

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Hemifacial spasm is a kind of painless, intermittent, involuntary, and irregular unilateral facial muscles convulsion. Microvascular decompression has become the standard surgical procedure for hemifacial spasm after years of popularization and development. In the article, the authors described in

Treatment of Venous Hemorrhage Between Vestibulocochlear Nerve and Hypertrophic Flocculus During Microvascular Decompression Procedure for Hemifacial Spasm.

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Delayed Facial Palsy After Microvascular Decompression for Hemifacial Spasm.

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To explore the risk factors of delayed facial paralysis (DFP) after microvascular decompression (MVD) for hemifacial spasm (HFS) METHODS: :A retrospective study was conducted on 636 patients who had undergone MVD for HFS by the same neurosurgery department of China-Japan Friendship Hospital from

[A case of hemifacial spasm associated with a cerebellar hematoma].

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This is a report of a hemifacial spasm associated with a cerebellar hematoma. A 60 year-old female was admitted to our hospital due to severe vertigo and nausea. On admission, the neurological examination showed drowsy conscious level, cerebellar sign dominant on the left side, and left hemifacial

Hemifacial spasm developed after contralateral vertebral artery ligation.

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Although the mechanism of hemifacial spasm (HFS) is not yet well established, vascular compression of the facial nerve root exit zone and hyperexcitability of the facial nucleus have been suggested. We report a case of HFS in the setting of coinciding intracranial hemorrhage (ICH) of the pons and

Hemifacial spasm caused by CP angle AVM associated with ruptured aneurysm in the feeding artery--case report.

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A 66-year-old male presented with clinical features of hemifacial spasm. Cerebral angiograms disclosed an arteriovenous malformation (AVM) in the cerebellopontine angle. The hemifacial spasm was caused by a dilated feeding artery of the AVM compressing the facial nerve at the root exit zone. Surgery

Dissecting aneurysm of the vertebral artery developed after microvascular decompression for hemifacial spasm.

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CONCLUSIONS We report a rare case of a ruptured de novo dissecting aneurysm induced by ethyl 2-cyanoacrylate. A 39-year-old woman underwent microvascular decompression for left hemifacial spasm. The offending vessel was left posterior inferior cerebellar artery (PICA). Left vertebral artery (VA) was

Delayed dural arteriovenous fistula after microvascular decompression for hemifacial spasm.

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Dural arteriovenous fistula (AVF) is very rare, acquired lesion that may present with intracranial hemorrhage or neurological deficits. The etiology is not completely understood but dural AVF often has been associated with thrombosis of the involved dural sinuses. To our knowledge, this is the first

Hemifacial spasm and recurrent stroke due to vertebrobasilar dolichoectasia coexisting with saccular aneurysm of the basilar artery: a case report.

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We describe a patient presented with sequential events of hemifacial spasm, cerebral infarction and fatal subarachnoid hemorrhage. All of them are seemingly separate entities. Radiological examination revealed that the cause was vertebrobasilar dolichoectasia (VBD) coexisting with a giant saccular

Nitric oxide metabolites in the cisternal cerebral spinal fluid of patients with subarachnoid hemorrhage.

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OBJECTIVE To investigate nitric oxide (NO) metabolism after subarachnoid hemorrhage (SAH). METHODS We measured the concentrations of the NO metabolites, nitrite and nitrate, in cerebrospinal fluid (CSF) obtained from the cisternal drainage of patients with SAH. Studies were performed for 31 patients

Synchronous bilateral hemifacial spasm: case-report and literature review.

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Bilateral hemifacial spasm (biHFS) is an infrequent cranial nerve disorder that causes patients to suffer from severe psychological stress, and there are no reported cases of synchronous biHFS. In this study, a 46-year-old right-handed woman was diagnosed with a synchronous biHFS. After one

[Microneurosurgery for the trigeminal neuralgia and hemifacial spasm and glosspharyngeal neuralgia with endoscopic].

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OBJECTIVE Discussing the use of endoscopy in the operation of microvascular decompression of cranial nerves to treat trigeminal neuralgia (TN), hemifacial spasm (HFS), glosspharyngeal neuralgia (GN) and some tumors of the cerebellopontine angle (CPA). METHODS Since 2006, 973 cases (including 420

Serious complications of microvascular decompression operations for trigeminal neuralgia and hemifacial spasm.

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Serious complications of microvascular decompression operations for trigeminal neuralgia or hemifacial spasm are reported. Among 278 patients who underwent microvascular decompression, 9 serious complications were observed: 1 intracerebellar hematoma with acute hydrocephalus, 1 cerebellar swelling

Microvascular decompression for treating hemifacial spasm: lessons learned from a prospective study of 1,174 operations.

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The authors critically analyzed a large series of patients with hemifacial spasm (HFS) and who underwent microvascular decompression (MVD) under a prospective protocol. We describe several "lessons learned" that are required for achieving successful surgery and proper postoperative management. The

Postoperative complications of microvascular decompression for hemifacial spasm: lessons from experience of 2040 cases.

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Microvascular decompression (MVD) is an effective and safe treatment option that offers the prospect of definitive cure for hemifacial spasm (HFS). However, there are potential risks of complications for MVD associated with retromastoid suboccipital craniectomy (RmSOC) and cranial nerves in

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