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hyperventilation/infarci

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Facilitation through hyperventilation of therapeutic effect of pyrithioxin in cerebral infarct patients.

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A method of treatment consisting in administration of a neurodynamic drug, pyrithioxin, combined with a short period of hyperventilation (HV) was applied in cerebral infarct patients with hemiplegia. The combination was superior to pyrithioxin alone for the motor recovery of these patients. In some

Possible role of coronary spasm in acute myocardial infarction precipitated by hyperventilation.

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Acute myocardial infarction was precipitated by hyperventilation in a 65 year old man. His coronary arteriogram in the chronic phase showed almost normal coronary arteries. Injection of acetylcholine (50 micrograms) into the left coronary artery induced spasm of the circumflex artery with chest pain

Hyperventilation-induced ST segment elevation mimicking acute myocardial infarction in a comatose patient with tracheostomy.

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Controlled hyperventilation leading to respiratory alkalosis may induce coronary artery spasm. This manoeuvre is currently used in the diagnosis of Prinzmetal's angina. We describe the case of a comatose patient with tracheostomy in whom hyperventilation, caused by excessive bronchial secretion

Aspirin, hyperventilation, and cerebellar infarction in sickle cell disease.

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Aspirin ingestion was followed by hyperventilation, cerebellar signs, and fatal brain stem dysfunction in a patient with sickle cell disease. Autopsy showed a swollen, recently infarcted cerebellum with tonsillar herniation compressing the medulla. We propose that hypocapnea from aspirin-induced

Chronic dyspnea and hyperventilation in an awake patient with small subcortical infarcts.

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A 79-year-old woman presented with chronic dyspnea and hyperventilation. There was no evidence of pulmonary disease. Hyperventilation persisted during sleep and after high-dose administration of a narcotic. A head MRI revealed bilateral medial thalamic infarctions. Central neurogenic

[Controlled hyperventilation in the therapy of acute myocardial infarction].

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Two patients with anterior wall infarction were hyperventilated under control with the aim of the induction of an inverse steal-phenomenon. Simultaneously the registration of the ST-dynamics was performed by means of precordial mapping. In a region of an arterial carbon dioxide partial pressure up
To assess contribution of dynamic (vasospastic) stenosis to pathogenesis of anginal attacks in postmyocardial patients, 148 patients 15-4 months after the infarction were examined using bicycle exercise, Holter ECG monitoring and hyperventilation test (HVT). The relations between painless myocardial

Non Q-wave myocardial infarction following hyperventilation test.

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We report a case of acute myocardial infarction following a hyperventilation test performed at coronary angiography. The potential pathophysiological mechanisms and clinical implications are discussed.

Hyperventilation and myocardial infarction.

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Chest pain that is associated with hyperventilation is often considered to be benign and noncardiac in nature. While not commonly recognized, hyperventilation can provoke coronary vasospasm. We report a man who presented with hyperventilation and developed myocardial infarction. In the setting of

[Central neurogenic hyperventilation associated with a pontine infarction].

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Central neurogenic hyperventilation in patients with a normal level of consciousness is uncommon. This condition occurs in bilateral pontine tegmental lesions, particularly tumors such as CNS lymphomas and glioma, as well as traumatic lesions. The physiopathological mechanisms are unknown and no

Hyperventilation in patients who have sustained myocardial infarction after a work injury.

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Patients who present with acute myocardial infarction after a work injury (AMI-WI) often report symptoms consistent with chronic hyperventilation which date back as far as the work injury itself, rather than to the AMI. The aim of the study was to test the hypothesis that hyperventilation

Hyperventilation in normoxia following myocardial infarction in rats: a shift in the set point of the hypoxic ventilatory response.

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OBJECTIVE The peripheral chemoreflex is augmented in heart failure, and it may contribute to sympathoexcitation. This study aimed to investigate both the chemoreflex and the cardiac sympathetic nerve activity in the acute-stage post-myocardial infarction. METHODS Myocardial infarction was induced in

Effect of delayed hyperventilation on experimental cerebral infarction.

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The effect of hyperventilation on subsequent cerebral infarction.

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Effects of hyperventilation, hypocapnia, and acetazolamide on experimental cerebral ischemia and infarction.

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