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laryngismus/œdème
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Pulmonary edema complicated by post-extubation laryngospasm: a case report.
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A case of post-extubation laryngospasm complicated by negative pressure pulmonary edema in a 26 year-old healthy male undergoing right herniorrhaphy is presented. Immediately after extubation laryngeal stridor was noted, which rapidly developed in severe laryngospasm. Two minutes after the
Naloxone administration and laryngospasm followed by pulmonary edema.
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A 50-year-old woman underwent laryngoscopy. Postoperatively she received naloxone and was extubated. She developed severe laryngospasm and one hour later pulmonary edema. Both naloxone administration and laryngospasm can provoke pulmonary edema; the pathophysiology is discussed. It is suggested that
Laryngospasm induced pulmonary edema.
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Two case reports of laryngospasm-induced pulmonary edema following general anesthesia are presented. Therapy consisted of immediate reintubation, application of positive pressure ventilation, and diuresis. This phenomenon should be recognized rapidly with appropriate therapy instituted immediately
[Two cases of acute pulmonary edema secondary to laryngospasm].
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We report two cases of acute pulmonary edema attributable to episodes of postextubation laryngospasm appearing when two young patients awoke after appendectomies under general anesthesia. Both patients improved in less than 48 hours with diuretics, oxygen therapy and liquid restriction, making
Acute Postobstructive Pulmonary Edema Following Laryngospasm in Elderly Patients: A Case Report.
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Only a few cases regarding postobstructive pulmonary edema following laryngospasm in older patients aged more than 60 years have been reported; however, acute pulmonary edema or pulmonary hemorrhage would be more deadly to elderly patients who have cerebrovascular disease than young healthy adults.
[Postoperative laryngospasm may induce pulmonary edema. An unusual or overlooked complication?].
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Young and healthy patients in whom laryngospasm secondary to postoperative extubation is encountered run a risk of developing pulmonary edema. The mechanism behind the edema is thought to be a strongly negative intrathoracic pressure generated by the patient's forced inspiration against a closed
Postoperative negative pressure pulmonary edema following repetitive laryngospasm even after reversal of neuromuscular blockade by sugammadex: a case report.
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Laryngospasm, an occlusion of the glottis, can occur at any time during anesthesia, and is associated with serious perioperative complications such as hypoxia, hypercabia, aspiration, bronchospasm, arrhythmia, prolonged recovery, cardiac collapse, and eventually catastrophic death. Importantly,
Laryngospasm-induced pulmonary edema.
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Pulmonary edema after relief of airway obstruction due to laryngospasm is an uncommon but recognized entity. The authors report a case of a previously healthy young man who developed pulmonary edema after relief of laryngospasm following extubation of the trachea. Pulmonary edema after relief of
Laryngospasm-induced pulmonary edema.
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Laryngospasm developed in a 33-year-old woman following attempted endotracheal intubation. Following establishment of the airway, the patient developed pulmonary edema which was successfully treated by conventional means. The sequence of events suggests that laryngospasm precipitated the development
Laryngospasm and noncardiogenic pulmonary edema.
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The incidence of laryngospasm in the PACU after extubation remains low despite the number of extubations that are performed annually. A study of general anesthesia patients conducted by Olsson and Hallen in 1977 & reported by Holmes et al (Am J Sports Med 19:365-371, 1991) showed the incidence of
Laryngospasm-induced pulmonary edema.
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Laryngospasm is one of the more common forms of airway obstruction encountered by an anesthetist. Therapy usually is straightforward, with resolution of the obstruction normally occurring within minutes. In some cases, however, the patient's vigorous inspiratory efforts may lead to a rapidly
Acute pulmonary edema after laryngospasm.
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Pulmonary edema may develop in healthy patients after anesthesia. Even in adult patients it is important to ascertain the depth of anesthesia before extubation. Too early extubation can result in laryngospasm, followed by increased inspiratory effort and significant rises in pulmonary capillary
Postobstructive pulmonary edema after laryngospasm in the otolaryngology patient.
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BACKGROUND
Post-obstructive pulmonary edema (PPE) is an uncommon complication which develops immediately after the onset of acute airway obstruction such as laryngospasm or epiglottitis (type I) or after the relief of chronic upper airway obstruction such as adenotonsillar hypertrophy (type
Laryngospasm-induced pulmonary edema: case report.
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Laryngospasm during the emergent phase of anesthesia is a respected complication well known to any PACU nurse. One complication of laryngospasm is noncardiac pulmonary edema (NCPE). NCPE can be a catastrophic complication of anesthesia. A case report is presented to illustrate the signs and symptoms
Review of laryngospasm and noncardiogenic pulmonary edema.
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Laryngospasm is an emergency situation that requires rapid identification and resolution of the obstructed glottis. Although there is a low incidence of laryngospasm, it is important to remember that any patient has the potential for post-extubation laryngospasm. Nurses must know about the causes,
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