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[Effects of low molecular organic acids on nitrogen accumulation, nodulation, and nitrogen fixation of soybean (Glycine max L.) under phosphorus deficiency stress].
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A greenhouse sand culture experiment was conducted to study the effects of citric acid, oxalic acid, malic acid, and their mixture on the nitrogen accumulation, nodulation, and nitrogen fixation of soybean. After the application of test low molecular weight organic acids, the nitrogen accumulation
Loci and candidate gene identification for resistance to Sclerotinia sclerotiorum in soybean (Glycine max L. Merr.) via association and linkage maps.
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Soybean white mold (SWM), caused by Sclerotinia sclerotiorum ((Lib.) W. Phillips), is currently considered to be the second most important cause of soybean yield loss due to disease. Research is needed to identify SWM-resistant germplasm and gain a better understanding of the genetic and molecular
Identification of mono-, oligo-, and polysaccharides secreted from soybean roots.
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The mist culture system was conducted to study secreted polysaccharides from soybean ( Glycine max) roots grown for 15 days. Roots were rinsed with distilled water (DW) for 15 min, then with 30 mM oxalic acid (OXA) for 15 min to remove ionically bound sugar. Released sugars were further fractionated
Improving nutritional quality and fungal tolerance in soya bean and grass pea by expressing an oxalate decarboxylase.
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Soya bean (Glycine max) and grass pea (Lathyrus sativus) seeds are important sources of dietary proteins; however, they also contain antinutritional metabolite oxalic acid (OA). Excess dietary intake of OA leads to nephrolithiasis due to the formation of calcium oxalate crystals in kidneys. Besides,
Enhanced resistance to sclerotinia stem rot in transgenic soybean that overexpresses a wheat oxalate oxidase.
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Sclerotinia stem rot (SSR), caused by the oxalate-secreting necrotrophic fungal pathogen Sclerotinia sclerotiorum, is one of the devastating diseases that causes significant yield loss in soybean (Glycine max). Until now, effective control of the pathogen is greatly limited by a lack of strong
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