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polyuria/vomissement
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[Loss of weight (emaciation), nausea, emesis, dizziness and polyuria: (malignant neuroglioma)].
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Fanconi syndrome and severe polyuria: an uncommon clinicobiological presentation of a Gitelman syndrome.
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BACKGROUND
Gitelman syndrome is an autosomal recessive tubulopathy characterized by hypokalemia, hypomagnesemia, metabolic alkalosis and hypocalciuria. The majority of patients do not present with symptoms until late childhood or adulthood, and the symptoms are generally mild. We report here the
A young man with polyuria and lethargy.
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We report a 20-year-old man who presented to our emergency room with a history of polyuria, weakness, constipation, nausea, and vomiting of two months duration. History and clinical examination revealed a significant weight loss and mild hepatomegaly. Laboratory investigations revealed hypokalemia,
Noncardiogenic pulmonary edema complicating diabetic ketoacidosis.
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OBJECTIVE
To alert physicians to the possibility of pulmonary edema as a complication of diabetic ketoacidosis.
METHODS
We report a case of adult respiratory distress syndrome after resuscitative efforts to compensate the first episode of diabetic ketoacidosis in a previously healthy young
Identification of five novel arginine vasopressin gene mutations in patients with familial neurohypophyseal diabetes insipidus.
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Familial neurohypophyseal diabetes insipidus (FNDI) is a genetic disorder presenting with polyuria and polydipsia and is caused by mutations in the arginine vasopressin-neurophysin II (AVP-NPII) gene. The clinical manifestations of this disorder vary greatly depending on different mutations. The
Desmopressin toxicity due to prolonged half-life in 18 patients with nocturnal enuresis.
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OBJECTIVE
Desmopressin has been used extensively for primary nocturnal enuresis and it is associated with a low incidence of adverse effects. The only reported serious side effect is seizure or altered levels of consciousness resulting from water intoxication, which has been reported for the nasal
Renal dysplasia in a Shih Tzu dog in Japan.
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A 5-month-old, male, Shih Tzu dog manifesting polyuria and polydipsia since 2-month-old was presented to our hospital with additional clinical complaints of vomiting and depression during recent a few days. Despite the symptomatic therapy for chronic renal failure, he died on the day after
Arterial blood gas and acid-base values in dogs with various diseases and signs of disease.
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Results of arterial blood gas and acid-base analysis on initial samples prior to therapy were reviewed for 220 dogs admitted to the University of Georgia Veterinary Teaching Hospital. Acidemia or alkalemia was detected in 61 of 220 dogs (28%). The most common acid-base abnormality was metabolic
Hereditary renal tubular disorders in Turkey: demographic, clinical, and laboratory features.
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BACKGROUND
The Turkish Renal Tubular Disorders Working Group aimed to form a patient registry database and gathered demographic, clinical, and laboratory data in various hereditary renal tubular disorders (HRTDs).
METHODS
A questionnaire comprising HRTDs was sent to the centers. The cohort was
End-to-end ureteral anastomosis and double-pigtail ureteral stent placement for treatment of iatrogenic ureteral trauma in two dogs.
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METHODS
A 6-month-old spayed female Soft-Coated Wheaten Terrier and 8-month-old spayed female Shih Tzu were referred because of complications related to inadvertent ureteral ligation and transection during recent ovariohysterectomy.
RESULTS
The Soft-Coated Wheaten Terrier had a 2-day history of
Risperidone-induced type 2 diabetes presenting with diabetic ketoacidosis.
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A 28-year-old male presented with 2 days of vomiting and abdominal pain, preceded by 2 weeks of thirst, polyuria and polydipsia. He had recently started risperidone for obsessive-compulsive disorder. He reported a high dietary sugar intake and had a strong family history of type 2 diabetes mellitus
The essentials of calcium, magnesium and phosphate metabolism: part II. Disorders.
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OBJECTIVE
To review the components of calcium, phosphate and magnesium metabolism that are relevant to the critically ill patient, in a two-part presentation.
METHODS
A review of articles reported on calcium, phosphate and magnesium disorders in the critically ill patient.
RESULTS
Abnormal calcium
Cystinosis in Eastern Turkey.
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BACKGROUND
This study was conducted to investigate CTNS (cystinosin, lysosomal cystine transporter) gene mutations and the clinical spectrum of nephropathic cystinosis among patients diagnosed with the disease in a single center in Turkey.
METHODS
Patients' clinical and laboratory data were
Profound hypernatremia due to central diabetes insipidus.
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Diabetes insipidus is a rare endocrine disorder in paediatric patients. Polyuria is a cardinal manifestation that is extremely difficult to recognize in diapered infants. Careful urine quantification is the key to diagnosis in appropriate clinical setting. We report a case of a 4 months old infant
[Distal hereditary renal tubular acidosis, diagnosis in siblings: About 2 pediatric cases].
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Distal renal tubular acidosis is the most common type of renal tubular acidosis in pediatrics and can be hereditary. It is due to an inability in the kidney to excrete hydrogen ion (H+), in the absence of deterioration of renal function, and it is accompanied by hyperchloremic metabolic acidosis
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