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pulmonary heart disease/− nicotine
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A case report of cor pulmonale in a woman without exposure to tobacco smoke: an example of the risks of indoor wood burning.
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We present the case of a 67-year-old woman with chronic cor pulmonale. She never smoked tobacco and had no other risk factors for pulmonary disease. In developed nations, chronic obstructive lung disease and cor pulmonale are overwhelmingly associated with tobacco use. However, indoor air pollution,
[Special use of nicotine-k-strophanthin; chronic pulmonary heart disease].
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Measurement of nicotine in building air as an indicator of tobacco smoke levels.
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Humans apparently differ greatly in their sensitivity and tolerance to tobacco smoke, thereby creating conflicts in the workplace. Resolution of conflicts in a large office complex at the authors' institution required an objective measure of smoke levels. A gas chromatographic technique was devised
Effect of 2-5 years of tobacco smoking on ventilatory function tests.
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Smoking is a pernicious scourge of the world today. There is paucity of literature on the effect of acute smoking on ventilatory functions. The present work is undertaken to study the effect of 2-5 years of tobacco smoking on ventilatory functions. The study group consisted of 30 male, young,
[Smoking cessation in patients with cardiopulmonary diseases].
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OBJECTIVE
To determine the smoking cessation rates of outpatients with cardiopulmonary disease and the differences between non-cardiopulmonary diseases.
METHODS
Two hundred and two active smokers with comorbid diseases were prospectively evaluated between September 2004 and January 2008 in this
[Immunoglobulin A and tobacco antigens in patients with pneumopathy].
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Smoking is an addiction related with several cardiopulmonary diseases. Some compounds derived from tobacco combustion can be induce a response of secretory immune system, because antigen penetration is by respiratory tract epithelium, with IgA antibodies synthesis and immune complex (IC) generation.
Amount of nicotine and carbon monoxide inhaled by smokers of low-tar, low-nicotine cigarettes.
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In 76 smokers, correlations between plasma nicotine and alveolar carbon monoxide levels of the individual smoker and the nicotine and CO yields of his cigarette were very poor. In 24 smokers of low-nicotine, low-tar cigarettes, mean alveolar CO levels did not differ from those of smokers of regular
Active and passive tobacco smoke exposure: a comparison of maternal and child hair cotinine levels.
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The objective of this study was to compare tobacco smoke exposure in mothers and their healthy children less than 3 years old using hair cotinine (HC) levels as an objective long-term measure of exposure. Hair samples were obtained from mother/child pairs recruited from the Columbus Children's
Genomics of Particulate Matter Exposure Associated Cardiopulmonary Disease: A Narrative Review.
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Particulate matter (PM) exposure is associated with the development of cardiopulmonary disease. Our group has studied the adverse health effects of World Trade Center particulate matter (WTC-PM) exposure on firefighters. To fully understand the complex interplay between exposure, organism, and
[Chronic pulmonary heart disease and its risk factors among a worker population in Teheran].
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Because of the high frequency of chronic cor pulmonale in workers admitted to the cardiology department of the Khazaneh Hospital in Teheran, we studied the clinical aspect and the risk factors of this disease in 66 male patients. The average age of patients was 56.1 years and they often had a long
[Chronic cor pulmonale: a study of 34 cases in the Dakar University Hospital Center Cardiology Department].
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The authors report the results of a study on patients with chronic cor pulmonale hospitalized in the Departement of Cardiology (Dakar), from 1990 to 1998. The hospital prevalence is 0.9% with a male predominance (73.5%). The clinical signs were dominated by dyspnea and cough. The right heart failure
Electrocardiographic signs of chronic cor pulmonale in 40 376 patients with silicosis.
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The records of all patients who were examined for silicosis at the Fund of Occupational Diseases between 1972 and 1976 are reviewed. In 3627 cases the mechanographical record was incomplete leaving 40 376 patients in the study. Electrocardiographic signs of chronic cor pulmonale (C.C.P.) were
Thromboembolic injury and systemic toxicity induced by nicotine in mice.
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Nicotine is involved in the pathogenesis of hematological and cardiopulmonary diseases. The understanding of the pathophysiological mechanisms underlying these undesirable effects is however unclear. Cigarette smoking, nicotine gums and patches are common sources for nicotine ingestion. We have
Acute exposure to environmental tobacco smoke and heart rate variability.
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Environmental tobacco smoke (ETS) has been associated with cardiovascular mortality. Pathophysiologic pathways leading from ETS exposure to cardiopulmonary disease are still being explored. Reduced cardiac autonomic function, as measured by heart rate variability (HRV), has been associated with
Gene variance in the nicotinic receptor cluster (CHRNA5-CHRNA3-CHRNB4) predicts death from cardiopulmonary disease and cancer in smokers.
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BACKGROUND
Genetic variation in the cluster on chromosome 15, encoding the nicotinic acetylcholine receptor subunits (CHRNA5-CHRNA3-CHRNB4), has shown strong associations with tobacco consumption and an additional risk increase in smoking-related diseases such as chronic obstructive pulmonary
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