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rhabdomyolysis/l tyrosine
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Comment: Management of De Novo Chronic Myelogenous Leukemia and Imatinib-Induced Acute Rhabdomyolysis With the Second-Generation Tyrosine Kinase Inhibitor Dasatinib.
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[Carbohydrate and nitrogenous metabolism condition in the rat tissue under experimental rhabdomyolysis].
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Some effects of glycerol injection on indices of the condition of the thiol-disulfide system as well as carbohydrate and nitrogen metabolism in rats in vivo were studied. A decrease was revealed in levels of non-protein SH-groups in the liver, kidney and heart, as well as of protein SH-groups in the
Selenium inhibits renal oxidation and inflammation but not acute kidney injury in an animal model of rhabdomyolysis.
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Acute kidney injury (AKI) is a manifestation of rhabdomyolysis (RM). Extracellular myoglobin accumulating in the kidney after RM promotes oxidative damage, which is implicated in AKI.
OBJECTIVE
To test whether selenium (Se) supplementation diminishes AKI and improves renal function.
RESULTS
Dietary
Treatment compliance and severe adverse events limit the use of tyrosine kinase inhibitors in refractory thyroid cancer.
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OBJECTIVE
The aim of the present study was to assess patient compliance with tyrosine kinase inhibitor (TKI) treatment used for refractory and progressive thyroid cancer, in addition to the efficacy and serious adverse events associated with these agents.
METHODS
We retrospectively analyzed data
Life-threatening disseminated enterovirus infection during combined rituximab and ibrutinib maintenance treatment for mantle cell lymphoma: a case report
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Background: Rituximab is a well-established component of treatment regimens for B-cell non-Hodgkin lymphoma. Rituximab binds the CD20 antigen on the surface of B lymphocytes, causing an enhanced clearance of malignant and benign B cells.
Characteristics and mechanism of formation of peroxide-induced heme to protein cross-linking in myoglobin.
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At acidic pH values heme-protein cross-linked myoglobin (Mb-H) forms as a product of a peroxide-induced ferric-ferryl redox cycle. There is evidence that this molecule acts as a marker for heme-protein-induced oxidative stress in vivo and may exacerbate the severity of oxidative damage due to its
Extracellular signal-regulated kinase mediates renal regeneration in rats with myoglobinuric acute renal injury.
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In vitro data support that extracellular signal-regulated kinase (ERK) and c-Jun N-terminal kinase (JNK), members of mitogen-activated protein (MAP) kinases, mediate the signal transduction pathways responsible for the cell proliferation. However, in vivo role of these MAP kinases is poorly
Clinical approach to the weak patient in the intensive care unit.
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Motor weakness in a patient in the intensive care unit (ICU) may be related to (1) pre-existing neuromuscular disorder that leads to ICU admission, (2) new-onset or previously undiagnosed neurological disorder, or (3) complications of non-neuromuscular critical illness. Neuromuscular syndromes
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