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[Alpha globulins and alpha globulin fractions of glycoproteins in acute myocardial infarct].
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[Lipoprotein (alpha) as a risk factor for myocardial infarction].
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Serum lipoprotein (alpha) [Lp(alpha)]was determined in 105 cases of myocardial infarction survivors (MIS). Lp(alpha) level in MIS was much more elevated as compared with that in healthy subjects and in a hypertension group. Lp(alpha) had no relation with apolipoprotein B and AI, cholesterol,
Differential expression of alpha 1, alpha 3 and alpha 5 integrin subunits in acute and chronic stages of myocardial infarction in rats.
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OBJECTIVE
Anchoring cardiac myocytes to extracellular matrix, which is mediated mainly by integrins on their surfaces, is important for maintaining the architecture of myocardial tissues and transmitting mechanical force. We evaluated the expression of alpha integrin subunits on myocytes and the
SERUM ALPHA-HYDROXYBUTYRATE DEHYDROGENASE: A NEW TEST FOR MYOCARDIAL INFARCTION.
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A prevention trial on cerebral infarction with dl-alpha-tocopheryl nicotinate--rationales for trial. The Alpha-Tocopheryl Nicotinate Prevention Study Group.
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Changes in plasma level of alpha-atrial natriuretic polypeptide (alpha-ANP) and responsiveness of the aorta to exogenous alpha-ANP subsequent to myocardial infarction in rats.
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The changes in plasma level of alpha-atrial natriuretic polypeptide (alpha-ANP) and the relaxing responses to exogenous alpha-ANP of strips of rat aorta pretreated with methoxamine were examined at one, four and eight weeks after myocardial infarction induced by left coronary ligation. Responses to
Alpha-adrenergic blockade in myocardial infarction.
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A study of lipid peroxide and alpha tocopherol in acute myocardial infarction.
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Thirty patients of acute myocardial infarction proven by electrocardiography (ECG) and enzymes were included in the study. All of them received streptokinase. A single lead showing the largest ST elevation, a proportional value for the shift in ST segment > or = 0.5 was taken as criteria for
Utility of alpha-blockade in a hypotensive pheochromocytoma patient with myocardial infarction.
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OBJECTIVE
The aim of this case study is to emphasize the importance of α-blockade in managing a rare complication of an untreated pheochromocytoma.
METHODS
A 41-year-old man with previous bilateral pheochromocytoma presented with chest pain. He was suffering from cardiac failure and persistent
Kinetics of tumor necrosis factor alpha in plasma and the cardioprotective effect of a monoclonal antibody to tumor necrosis factor alpha in acute myocardial infarction.
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BACKGROUND
Inflammation plays a critical role in acute myocardial infarction (AMI) and tumor necrosis factor alpha (TNF-alpha) is a potent inflammatory trigger. This study was designed to examine the kinetics of TNF-alpha in plasma in patients with AMI and the potential benefit of inhibition of
Alpha 1-antitrypsin in acute myocardial infarction.
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Alpha 1-antitrypsin serum levels were measured in 48 patients with acute myocardial infarction and in 19 control patients either with coronary heart disease without necrosis, or with neither coronary disease nor inflammation. Alpha 1-antitrypsin was significantly raised in the group of patients with
Serum alpha-hydroxybutyric dehydrogenase in myocardial infarction.
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[Serum alpha-amino nitrogen in myocardial infarction].
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SERUM "ALPHA-HYDROXYBUTYRATE DEHYDROGENASE". A CLINICAL AND LABORATORY EVALUATION IN PATIENTS WITH MYOCARDIAL INFARCT.
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Circulating alpha-actin protein in acute myocardial infarction.
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We used Western-blot analysis to investigate the possible presence in the bloodstream of the contractile protein alpha-actin in 70 patients diagnosed with acute myocardial infarction on the basis of clinical, electrocardiographic and laboratory (creatine kinase and lactate dehydrogenase) criteria.
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