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Page 1 de 252319 résultats
[Tumor necrosis factor alpha (TNF alpha)].
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The role of tumor necrosis factor-alpha (TNF-alpha) in skeletal muscle regeneration. Studies in TNF-alpha(-/-) and TNF-alpha(-/-)/LT-alpha(-/-) mice.
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The role of tumor necrosis factor-alpha (TNF-alpha), an important mediator of the inflammatory response after injury, was investigated in regenerating skeletal muscle. The pattern of expression of TNF-alpha during muscle regeneration was examined by immunohistochemistry in tissue sections of
Rapid genotyping for tumor necrosis factor-alpha (TNF-alpha)-863C/A promoter polymorphism that determines TNF-alpha response.
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Tumor necrosis factor-alpha (TNF-alpha) plays a protective role in acute viralmyocarditis in mice: A study using mice lacking TNF-alpha.
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BACKGROUND
It has been reported that tumor necrosis factor-alpha (TNF-alpha) is expressed in the heart with viral myocarditis and that its expression aggravates the condition. The pathophysiological effects of TNF-alpha on viral myocarditis, however, have not been fully elucidated.
RESULTS
To
Tumour necrosis factor-alpha (TNF-alpha) polymorphisms-857C/A and -863C/A are associated with TNF-alpha secretion from human adipose tissue.
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Soluble tumor necrosis factor-alpha (TNF-alpha) receptors in human colostrum and milk bind to TNF-alpha and neutralize TNF-alpha bioactivity.
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We used column chromatography, affinity binding, and bioassay methods to address whether the soluble tumor necrosis factor (TNF)-alpha receptors present in human colostrum and milk bind to and modify TNF-alpha bioactivity. In gel chromatography experiments, soluble TNF-alpha receptor I (sTNFRI) and
Tumour necrosis factor-alpha (TNF-alpha) in human toxoplasmosis.
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Tumour necrosis factor alpha (TNF-alpha) is a cytokine that is secreted by activated macrophages and monocytes. This work studied TNF-alpha level in sera of patients with toxoplasmosis. Twenty eight female patients (12 of whom had obstetric troubles) and 17 healthy controls were the subjects in this
Tumor necrosis factor-alpha (TNF-alpha) in pleural fluids.
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Our study was undertaken to investigate the role of tumor necrosis factor-alpha (TNF-alpha) at the site of disease activity in tuberculous pleuritis (TP). Concentrations of TNF-alpha were measured directly by radioimmunoassay (RIA) in the pleural fluid (PF) and plasma (P) of patients with TP (n =
Ageing, tumour necrosis factor-alpha (TNF-alpha) and atherosclerosis.
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Ageing is associated with increased inflammatory activity in the blood. The purpose of this study was to investigate if age-related increased plasma levels of TNF-alpha were associated with atherosclerosis in a cohort of 130 humans aged 81 years. The elderly cohort had increased circulating levels
[Biology of tumor necrosis factor type alpha (TNF-alpha)].
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Tumor necrosis factor type alpha (TNF-alpha) is one of 22 TNF superfamily ligands. Its synthesis is regulated by expression of gene localized on chromosome 6. During posttranslation modification the proteolytic cleavage of the 76-aminoacid domain from the membrane-bound protein occurs with final
[Cachexia, malignancy and tumor necrosis factor alpha (TNF-alpha)].
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Cachexia is a common finding in various diseases such as chronic renal failure, HIV infection, malignancies, chronic obstructive pulmonary disease, congestive heart failure and rheumatoid arthritis. It is estimated that 30% of patients with malignancies will appear with cachexia, and up to 80% of
Mechanisms of tumor necrosis factor-alpha (TNF-alpha) hyperalgesia.
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Activation of immune cells by pathogens induces the release of a variety of proinflammatory cytokines, including IL-1 beta and TNF-alpha. Previous studies using IL-1 beta have demonstrated that this cytokine can alter brain function, resulting in a variety of 'illness responses' including increased
[Creation of monoclonal antibodies against tumor necrosis factor-alpha (TNF-alpha) and transforming growth factor alpha (TFG-alpha), their definition and possible use].
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These antibodies were characterised immunoserologically and immunohistochemically. The antibodies proved suitable in the detection of TNF-alpha and TGF-alpha production by different cells and tissues. These anti-TNF-alpha specific monoclonal antibodies were able to inhibit the cytotoxic effect of
Prognostic relevance of transforming growth factor alpha (TGF-alpha) and tumor necrosis factor alpha (TNF-alpha) detected in breast cancer tissues by immunohistochemistry.
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The function of different growth factors in the development and progression of malignant tumors and the role of cytotoxic cytokines in the host response generated against neoplasms have been recently studied. Anti-TGF-alpha and anti-TNF-alpha monoclonal antibody families have been developed and
The effect of a metalloproteinase inhibitor (GI5402) on tumor necrosis factor-alpha (TNF-alpha) and TNF-alpha receptors during human endotoxemia.
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Tumor necrosis factor-alpha (TNF-alpha) is released from the cell surface by cleavage of pro-TNF-alpha by metalloproteinases (MPs). In cell cultures, inhibition of MPs has been found not only to reduce the release of TNF-alpha, but also to enhance the surface expression of TNF-alpha and TNF-alpha
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