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uremia/triglyceride
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Effect of acute uremia on triglyceride kinetics in the rat.
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Plasma triglyceride (TG) levesl were elevated 24 hr after the production of acute uremia in rats. The effect of acute uremia on TG production rate was estimated by determining the rate of TG accumulation following Triton WR 1339 inhibition of lipoprotein removal, by measuring hepatic TG secretion
Apolipoprotein C-III, hypertriglyceridemia and triglyceride-rich lipoproteins in uremia.
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Apolipoprotein C-III (ApoC-III) plays an important role in the metabolism of triglyceride-rich lipoproteins and is known to be elevated in patients with uremia. To investigate the role of apoC-III in uremic dyslipidemia, we examined apoC-III, triglyceride levels and lipoprotein particles containing
Hypertriglyceridemia in uremia and the use of triglyceride turnover to define pathogenesis.
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The effects of chronic uremia and dexamethasone on triglyceride kinetics in the rat.
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[Results of a multicenter study on the effect on serum triglycerides and cholesterol administration of carnitine in chronic uremia patients undergoing periodic hemodialysis treatment].
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Lipoprotein metabolism in patients with chronic uremia. Effect of hemodialysis on serum lipoproteins and postheparin plasma triglyceride lipases.
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Lipid and carbohydrate metabolism in uraemia.
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Lipid and carbohydrate metabolism variables were studied in twenty-eight patients with chronic renal failure (mean GFR 7.7 +/- 2.5 ml/min) and uraemic symptoms. 71% of the patients had hypertriglyceridaemia (greater than or equal to 2.2 mmol/l). Total serum cholesterol was normal while VLDL
The short term effects of bezafibrate on the hypertriglyceridaemia of moderate to severe uraemia.
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Hypertriglyceridaemia, an atherogenic risk factor, is a well recognised complication of uraemia, and is present in the earliest stages of the disease. Bezafibrate is an effective hypolipidaemic agent, and its effect in moderate to severe uraemia is documented in this study. Significant reductions in
Atherosclerosis in uremia: possible roles of hyperparathyroidism and intermediate density lipoprotein accumulation.
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Cardiovascular motality is high in patients with chronic renal failure treated with dialysis, and secondary hyperparathyroidism may promote atherosclerogenesis. Recent studies have revealed advanced atherosclerosis in hemodialysis patients by using high-resolution B-mode ultrasonography. Multiple
[Diabetes and atherosclerotic coronary damage in uremia].
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BACKGROUND
Cardiovascular risk factors are common findings in uraemics, but the impact of each single factor on the development of atherosclerosis is still a matter of debate.
METHODS
In order to evaluate the relationship between diabetes and ischaemic heart disease (IHD) in uraemia, we carried out
Effects of intravenous nutrition on lipoprotein metabolism, body composition, weight gain and uremic state in experimental uremia in rats.
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The effect on serum lipids, lipoprotein fractions, body composition, weight gain and uremic state of including fat in intravenous nutrition was evaluated in rats with chronic uremia. Uremic rats were given high energy (1385 kJ.kg body weight-1.day-1), low nitrogen (0.6 g N.kg body weight-1.day-1)
New insights into uremia-induced alterations in metabolic pathways.
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OBJECTIVE
This article summarizes recent studies on uremia-induced alterations in metabolism, with particular emphasis on the application of emerging metabolomics technologies.
RESULTS
The plasma metabolome is estimated to include more than 4000 distinct metabolites. Because these metabolites can
Lecithin-cholesterol acyltransferase (LCAT) activity in chronic uremia.
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High plasma concentrations of triglycerides and low plasma concentrations of esterified cholesterol and lysolecithin, with an impaired rate of VLDL and LDL catabolism, have been reported in chronic uremic patients. An important contribution to these abnormalitites might be an impaired activity of
Effect of chronic uremia on plasma lipids and the aortic accumulation of cholesterol in hypercholesterolemic rabbits.
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14 rabbits were made uremic by extensive cauterization and subsequent removal of the contralateral kidney, resulting in a mean plasma creatinine concentration four times the value observed in a sham-operated control group. The animals were killed after 10 weeks of uremia. During the last 7 weeks the
[Plasma levels of free fatty acids, triglycerides and cholesterol in acute subrenal kidney failure].
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The plasma triglyceride, cholesterol, and free fatty acid levels were determined in patients with acute subrenal kidney failure. Like in chronic uraemia, the levels were higher than normal. The findings were confirmed in animal experiments which showed that the plasma lipid fractions will rise after
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