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wallerian degeneration/fièvre
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Ataxia and paralysis associated with bovine ephemeral fever infection.
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Three adult cattle that had been ataxic for 5 to 7 months and a bull that had been paralysed for 24 days following bovine ephemeral fever infection were studied clinically and pathologically. Severe bilaterally symmetrical Wallerian degeneration was demonstrated in the spinal cords of all 4 cattle;
Viral neurotropism, peripheral neuropathy and other morphological abnormalities in bovine ephemeral fever virus-infected downer cattle.
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OBJECTIVE
This study assessed the neurotropism of bovine ephemeral fever (BEF) virus (BEFV) and described histomorphological abnormalities of the brain, spinal cord and peripheral nerves that may causally contribute to paresis or paralysis in BEF.
METHODS
Four paralysed and six asymptomatic but
Bilateral middle cerebral artery thromboembolic occlusion. Could maternal hyperthermia be a detrimental factor?
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We describe a six-month-old girl with microcephaly, developmental delay, truncal hypotonia, left pyramidal signs, partial seizures and myoclonic spasms, born to a feverish mother. MRI showed bilateral vascular lesions in the territory of the middle cerebral arteries, prevalent in the right
[An autopsy case of an extensive epidural spinal abscess demonstrating necrotizing poliomyelopathy].
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A 59-year-old man with a history of diabetes mellitus (NIDDM) presented with fever, back pain and weakness in the left lower limb. Three weeks later he suddenly developed flaccid paraklegia, a sensory deficit below the abdomen and sphincter dysfunction. MR images of the spinal cord showed an
Sensorimotor neuropathy in hemophagocytosis syndrome.
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Hemophagocytosis syndromes are uncommon disorders marked by generalized proliferation of benign histiocytes and multiple organ failure. A 24-year-old woman presented with a fulminant illness consisting of fever, hepatic insufficiency and anemia, followed by respiratory compromise, cardiomyopathy,
Hyperthermic injury versus crush injury in the rat sciatic nerve: a comparative functional, histopathological and morphometrical study.
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Functional and morphological changes of the rat sciatic nerve after local hyperthermia (30 min, 45 degrees C) and crush treatment were compared. After hyperthermic injury nerve function loss developed in a time period of about 7 h. Nerve crush led to an immediate loss of nerve function. Nerve
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