anticholinergic/infarction

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Leathanach 1 ó 82 torthaí

Delirium in acute stroke: a preliminary study of the role of anticholinergic medications.

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Logáil Isteach / Cláraigh

The pathogenesis of delirium in acute stroke is incompletely understood. The use of medications with anticholinergic (ACH) activity is associated with an increased frequency of delirium. We hypothesized that the intake of medications with ACH activity is associated with delirium in acute stroke

Antimuscarinics suppress adenosine triphosphate and prostaglandin E2 release from urothelium with potential improvement in detrusor overactivity in rats with cerebral infarction.

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Logáil Isteach / Cláraigh

OBJECTIVE Antimuscarinics improve detrusor overactivity. We evaluated the effects and action mechanisms of imidafenacin (Kyorin Pharmaceutical, Tokyo, Japan), a novel therapeutic agent for overactive bladder with antimuscarinic activity, on mediator release from urothelium and detrusor overactivity

Ionic channels, cholinergic mechanisms, and recovery of sensorimotor function after neocortical infarcts in rats.

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Logáil Isteach / Cláraigh

Unilateral photochemical infarcts were produced in the hind limb sensorimotor neocortex of 243 rats by intravenous injection of the fluorescein derivative Rose Bengal and focal illumination of the intact skull surface. Facial contact stimuli governed the degree and recovery rate of contralateral

[Effect of conorphone on the dimensions of the myocardial infarct and the ultrastructural changes in the adjacent tissue].

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Logáil Isteach / Cláraigh

Among contemporary problems of cardiovascular therapy, pain management in the cardiac patient is an important issue. Correct selection of a suitable analgesic drug is important in the acute phase of myocardial infarction because pain relief prevents adrenergic response which aggravates unbalance

Republished: pro-arrhythmic and pro-ischaemic effects of inhaled anticholinergic medications.

Ní féidir ach le húsáideoirí cláraithe ailt a aistriú

Logáil Isteach / Cláraigh

The majority of deaths in COPD are from cardiovascular causes. Several large randomized controlled trials demonstrate that inhaled anticholinergic agents ipratropium and tiotropium increase the risk of serious cardiovascular events, including cardiovascular mortality. Tiotropium Respimat is

Delayed oculogyric crises associated with striatocapsular infarction.

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Logáil Isteach / Cláraigh

Oculogyric crises are dystonic, usually upward, conjugate eye deviations. We describe an 11-year-old girl who developed oculogyric crises 3 1/2 years after infarction of the right caudate, putamen, and internal capsule. Her abnormal eye movements responded to anticholinergic agents. This is the

[Abnormal pulmonary function in patients with acute uncomplicated myocardial infarction (author's transl)].

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Logáil Isteach / Cláraigh

Pulmonary function has been evaluated in patients with acute uncomplicated myocardial infarction. Vital capacity,, flow-volume curves, oscillatory resistance and arterial oxygen tension have been measured within 24 hours of onset of symptoms and were repeated on day 4, 7 and 13 after infarction. In

[Vagus mediated elevation of respiratory resistance in patients with acute myocardial infarction].

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Logáil Isteach / Cláraigh

Changes in pulmonary function have been reported in patients after uncomplicated myocardial infarction, with reduction in lung volumes and flow rates. The significant increase in oscillatory resistance and impedance of the lung, was seen on the first day after myocardial infarction. The application

Paroxysmal kinesigenic dystonic choreoathetosis associated with a thalamic infarct.

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Logáil Isteach / Cláraigh

We describe a patient with the development of paroxysmal kinesigenic dystonic choreoathetosis (PKDC) after a thalamic infarct. PKDC consists of brief episodes of dystonia or choreoathetosis triggered by movement. PKDC improves with anticonvulsants, and in some cases, with L-Dopa or anticholinergics.

Conversion from intravenous to oral slow-release disopyramide in acute myocardial infarction.

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Logáil Isteach / Cláraigh

Twelve patients with acute myocardial infarction were given 1.5 mg/kg/5 min bolus +0.4 mg/kg/hr 6 hr IV infusion of disopyramide followed by 250 mg twice daily of a slow-release oral formulation of this drug. Plasma concentrations of total disopyramide rapidly reached steady-state within the

Rapid infusions of bidisomide or disopyramide in conscious dogs: effect of myocardial infarction on acute tolerability.

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Logáil Isteach / Cláraigh

The acute tolerability of rapid infusions of bidisomide or disopyramide was evaluated in normal conscious dogs and in conscious dogs 48 h after the creation of myocardial infarctions (MIs). Both drugs were given in total doses of 15 mg/kg (1.5 x the canine antiarrhythmic dose for each drug).

Haemodynamic and electrocardiographic effects of intravenous disopyramide (rythmodan) following acute myocardial infarction.

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Logáil Isteach / Cláraigh

1 The haemodynamic and electrocardiographic effects of intravenous disopyramide were studied in fifteen patients with acute myocardial infarction. 2 Five minutes after drug injection a rise in heart rate, aortic mean and diastolic pressures and systemic vascular resistence was noted which persisted

Cecal infarction secondary to a distal obstructing fecaloma: association with drug abuse.

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Logáil Isteach / Cláraigh

A case of cecal infarction proximal to an obstructing fecaloma is presented. The patient's medical records revealed a 19-year history of the abuse of prescribed narcotic, sedative, psychotropic, and anticholinergic drugs. Gangrene in this case was caused by compression of intramural vessels

Dementia and lower urinary dysfunction: with a reference to anticholinergic use in elderly population.

Ní féidir ach le húsáideoirí cláraithe ailt a aistriú

Logáil Isteach / Cláraigh

Urinary incontinence is common in patients with dementia, and is more prevalent in demented than in non-demented older individuals. Neurogenic incontinence is common in multiple cerebral infarction and dementia with Lewy bodies, and in both diseases walking difficulty and falls are common. In

[Are there cardiovascular adverse effects of inhaled anticholinergics?].

Ní féidir ach le húsáideoirí cláraithe ailt a aistriú

Logáil Isteach / Cláraigh

The purpose of this review is to discuss the cardiovascular risk associated with inhaled anticholinergics in chronic obstructive pulmonary disease. Several meta-analyses of data for tiotropium raised the possibility of an increased risk for arrhythmia, angina, myocardial infarction, etc. This review

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