Leathanach 1 ó 25073 torthaí
High rates of overweight and obesity even in very young children argue the case for strategies to prevent overweight from very young ages. Historical studies, prospective birth cohorts, and more recently genetic studies all indicate that the rapid weight gain trajectory to later obesity starts in
The prevalence of childhood obesity has increased dramatically in the second half of the 20th century even in children before the age of 5 years. Many factors may interfere with early development, from the prenatal period to the first two years of life, and modulate susceptibility to subsequent
OBJECTIVE
To study the dynamic processes that drive development of childhood overweight by examining the effects of prenatal characteristics and early-life feeding (breastfeeding versus bottle feeding) on weight states through age 7 years. We test a model to determine whether prenatal
As the prevalence of obesity increases across the globe, vast efforts are being directed towards understanding the origins of obesity and mechanisms underlying this rapid increase. It is well known that the current environment of an individual can affect body weight, however, growing evidence
There is increasing evidence that obesity has its origins in early life. Predisposition is based on interactions between the genome and environmental influences acting through epigenetic modifications. Individuals most at risk are those whose ancestral line has made a rapid transition from a
Size at birth and patterns of postnatal weight gain have been associated with adult risk for the development of type 2 diabetes in many populations, but the putative pathophysiological link remains unknown. Studies of contemporary populations indicate that rapid infancy weight gain, which may follow
BACKGROUND
Constitutional advancement of growth (CAG), a.k.a. early growth acceleration, refers to a growth pattern that is characterized by growth acceleration soon after birth, reaching a zenith centile in the first 2 to 4 yr of life and followed by normalization of the growth rate until the onset