altitude sickness/edema

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[Acute mountain sickness and high altitude cerebral and pulmonary edema].

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Altitude hypoxia is responsible for acute mountain sickness. It can worsen and generate a high altitude cerebral edema, which can be fatal. After reminding the reader clinical and epidemiological facts, this review aims to present new insights of the physiopathological continuity between these two

New epilepsy seizure at high altitude without signs of acute mountain sickness or high altitude cerebral edema.

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Neurological disturbances may be present at high altitude independently of high altitude cerebral edema. We report here the case of a patient who experienced for the first time generalized seizures after spending a night at an altitude of 5200 m, with no preceding symptoms of acute mountain

Disoriented and ataxic pilgrims: an epidemiological study of acute mountain sickness and high-altitude cerebral edema at a sacred lake at 4300 m in the Nepal Himalayas.

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OBJECTIVE To determine the incidence of high-altitude cerebral edema (HACE), acute mountain sickness (AMS), and high-altitude pulmonary edema (HAPE) in pilgrims. Although it is well known that western trekkers suffer from acute mountain sickness (AMS) in the Himalayas, not much is documented about

Quantification of optic disc edema during exposure to high altitude shows no correlation to acute mountain sickness.

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BACKGROUND The study aimed to quantify changes of the optic nerve head (ONH) during exposure to high altitude and to assess a correlation with acute mountain sickness (AMS). This work is related to the Tuebingen High Altitude Ophthalmology (THAO) study. RESULTS A confocal scanning laser

Blood rheology in acute mountain sickness and high-altitude pulmonary edema.

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The role of blood rheology in the pathogenesis of acute mountain sickness and high-altitude pulmonary edema was investigated. Twenty-three volunteers, 12 with a history of high-altitude pulmonary edema, were studied at low altitude (490 m) and at 2 h and 18 h after arrival at 4,559 m. Eight subjects

Acute mountain sickness and the edemas of high altitude: a common pathogenesis?

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Within days of ascent to high altitude when symptoms of acute mountain sickness (AMS) are common, pulmonary and cerebral edema may also develop. Although peripheral edema of the hands, face or feet may also appear, its association with AMS is unclear. In addition, persons with high altitude

Endothelial selectins in acute mountain sickness and high-altitude pulmonary edema.

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OBJECTIVE Mechanical or inflammatory injury to pulmonary endothelial cells may cause impaired pulmonary gas exchange in acute mountain sickness (AMS) and noncardiogenic pulmonary edema in high-altitude pulmonary edema (HAPE). This study was designed to determine whether markers of endothelial cell

A Tibetan with chronic mountain sickness followed by high altitude pulmonary edema on reentry.

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Chronic mountain sickness (CMS) and high altitude pulmonary edema (HAPE) each occur rarely in Tibetans, and they have previously not been reported in the same person. Here we describe a 37-year-old native Tibetan man with CMS at 4300 m, who developed HAPE after his return home from a 12-day visit to

[Acute mountain sickness and high-altitude pulmonary edema. How to protect the mountain climber from the effects of the "altitude haze"].

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Acute mountain sickness (AMS) usually occurs after 6-12 hours of acute exposure to altitudes above 2,500 m. If there is no further altitude gain, it normally resolves spontaneously within a day or two. However, it may, in rare cases, progress to life-threatening cerebral edema. High-altitude

Coagulation and fibrinolysis in acute mountain sickness and beginning pulmonary edema.

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To examine whether intravascular coagulation and/or decreased fibrinolysis precedes high-altitude pulmonary edema (HAPE) we examined 25 male mountaineers (median age 40 yr) at low altitude (550 m) and after 6, 18, and 42 h at an altitude of 4,559 m, which was climbed in 24 h. In 14 subjects, 2 of

Interstitial Pulmonary Edema Assessed by Lung Ultrasound on Ascent to High Altitude and Slight Association with Acute Mountain Sickness: A Prospective Observational Study.

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Background: Acute mountain sickness (AMS) is a common disease that may have a pulmonary component, as suggested by interstitial pulmonary edema quantified by the B-line score (BLS) on ultrasound (US). This subclinical pulmonary edema has been shown to increase with ascent to high altitude and

Cases from the aerospace medicine residents' teaching file. Case #44. An aviator with high attitude pulmonary and cerebral edema presenting acute mountain sickness.

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An aviator with high altitude pulmonary and cerebral edema presenting with Acute Mountain Sickness. The clinical presentation, evaluation and diagnosis of an aviator with acute mountain sickness are discussed. The aeromedical disposition of this patient is also presented.

The lung at high altitude: bronchoalveolar lavage in acute mountain sickness and pulmonary edema.

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High-altitude pulmonary edema (HAPE), a severe form of altitude illness that can occur in young healthy individuals, is a noncardiogenic form of edema that is associated with high concentrations of proteins and cells in bronchoalveolar lavage (BAL) fluid (Schoene et al., J. Am. Med. Assoc. 256:

High altitude cerebral edema and acute mountain sickness. A pathophysiology update.

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The diagnosis, treatment and prevention of high altitude cerebral edema (HACE) are fairly well established. The major unresolved issues are 1) the pathophysiology, 2) the individual susceptibility, and 3) the relationship of HACE to acute mountain sickness (AMS) and to high altitude pulmonary edema

Severe acute mountain sickness and suspect high altitude cerebral edema related to nitroglycerin use.

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An elite mountaineer reported severe acute mountain sickness and ataxia during an 8000-m expedition and concomitant use of transdermal nitroglycerin patches aimed to prevent frostbites. Use of nitroglycerin for this purpose is off-label, and its safety has not been assessed. Moreover, a relation

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