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amide/infarction
Veza se sprema u međuspremnik
Stranica 1 iz 94 rezultatima
Metabolically-inactive glucagon-like peptide-1(9-36)amide confers selective protective actions against post-myocardial infarction remodelling.
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BACKGROUND
Glucagon-like peptide-1 (GLP-1) therapies are routinely used for glycaemic control in diabetes and their emerging cardiovascular actions have been a major recent research focus. In addition to GLP-1 receptor activation, the metabolically-inactive breakdown product, GLP-1(9-36)amide, also
Correlations of amide proton transfer-weighted MRI of cerebral infarction with clinico-radiological findings
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Objective: To clarify the relationship between amide proton transfer-weighted (APTW) signal, which reflects intracellular pH, and clinico-radiological findings in patients with hyperacute to subacute cerebral infarction.
Novel gradient echo sequence‑based amide proton transfer magnetic resonance imaging in hyperacute cerebral infarction.
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In the progression of ischemia, pH is important and is essential in elucidating the association between metabolic disruption, lactate formation, acidosis and tissue damage. Chemical exchange‑dependent saturation transfer (CEST) imaging can be used to detect tissue pH and, in particular, a specific
N-Propargyl caffeate amide (PACA) prevents cardiac fibrosis in experimental myocardial infarction by promoting pro-resolving macrophage polarization.
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Macrophages control the initiation and resolution of cardiac fibrosis in post-infarction cardiac remodeling. The aim of the present study was to investigate whether N-propargyl caffeate amide (PACA) could suppress myocardial fibrosis via regulating macrophage polarization. By using rat model of
[Treatment of ventricular arrhythmias after acute myocardial infarct with procaine amide and mexiletine].
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[Prognostic and diagnostic significance of hiccup during myocardial infarction; therapeutic effect of procaine amide].
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[Prevention of sudden death in heart infarct by means of quinidine or procaine amide].
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[On the treatment of the coronary infarct with quinidine-procaine amide].
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Procaine amide overdosage in myocardial infarction.
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Prophylactic procaine amide in myocardial infarction.
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[Ventricular flutter in myocardial infarct treated with procaine amide pronestyl].
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Long-term use of procaine amide following acute myocardial infarction.
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[Post-infarction ventricular paroxysmal tachycardia; recurrent attacks at first controlled with procaine amide and thereafter definitively blocked with sparteine iodomethylate].
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The effects of intravenous procaine and procaine amide (pronestyl) upon ectopic ventricular tachycardia accompanying acute myocardial infarction.
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Glucagon-like peptide-1 (7-36) amide prevents the accumulation of pyruvate and lactate in the ischemic and non-ischemic porcine myocardium.
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Glucagon-like peptide-1 (7-36) amide (GLP-1) has been studied as a treatment option in diabetic patients. We investigated the effect of recombinant GLP-1 infusion on hemodynamic parameters, myocardial metabolism, and infarct size during normoxic conditions as well as during ischemia and reperfusion
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