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dextrose/infarction
Veza se sprema u međuspremnik
Stranica 1 iz 107 rezultatima
Further observations on the effect of insulin, potassium and dextrose infusions in acute myocardial infarction with a study of changes in the electrocardiograms--rouleaux formation of red blood cells, blood viscosity, hematocit, bsr, fibrinogen and prothrombin time, etc.
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Group 3. Observations on 400 cases of acute myocardial infarction treated with Insulin, Potassium and Dextrose infusions.
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Evaluation of dextrose, insulin, and potassium on ventricular irritability in acute myocardial infarction.
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The clinical implications of no reflow demonstrated with intravenous perfluorocarbon containing microbubbles following restoration of Thrombolysis In Myocardial Infarction (TIMI) 3 flow in patients with acute myocardial infarction.
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Intravenous injections or infusions of perfluorocarbon-exposed sonicated dextrose albumin microbubbles were given 2.4 +/- 1.6 days following acute myocardial infarction to 45 consecutive patients. Patients were divided into 3 groups: patients with Thrombolysis In Myocardial Infarction (TIMI) grade 3
Blockade of brain 'ouabain' prevents the impairment of baroreflexes in rats after myocardial infarction.
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BACKGROUND
The purpose of this study was to test whether increased brain "ouabain" contributes to impairment of both arterial and cardiopulmonary baroreceptor reflexes in congestive heart failure (CHF).
RESULTS
Two to 5 days after coronary artery ligation (MI) or sham surgery in male Wistar rats,
Hyperglycemia reduces the extent of cerebral infarction in rats.
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Although hyperglycemia is known to exacerbate neuronal injury in the setting of reversible brain ischemia, its effect on irreversible thrombotic infarction is less well understood. In this study, unilateral thrombotic infarction was induced photochemically in the parietal cortex of Wistar rats.
The Hyperglycemia: Intensive Insulin Infusion in Infarction (HI-5) study: a randomized controlled trial of insulin infusion therapy for myocardial infarction.
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OBJECTIVE
There is conflicting evidence regarding the benefit of intravenous insulin therapy on mortality following acute myocardial infarction (AMI). The goal of the current study was to determine whether improved glycemic control, achieved through an insulin/dextrose infusion with a variable rate
Hyperglycemia increases infarct size in collaterally perfused but not end-arterial vascular territories.
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Hyperglycemia exacerbates neuronal injury in the setting of reversible brain ischemia, but its effect on focal thrombotic infarction has been less extensively characterized. We investigated this problem in two rat models of focal vascular occlusion. In Model I, the right middle cerebral artery (MCA)
Utilization of a Dextrose-Only Purge Solution for an LVAD in Patients With Suspected HIT: A Case Series
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The Impella devices are percutaneous intravascular ventricular assist devices indicated for use in patients with cardiogenic shock that occurs following acute myocardial infarction (MI) or open heart surgery. These devices must be used with a purge solution that contains heparin per manufacturer
[Treatment with 10% glycerin in acute ischemic cerebral infarct. Doubleblind study].
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56 patients with acute ischemic cerebral infarction were randomly allocated to intravenous treatment for 6 days with either 500 ml of a 10% glycerol solution or 500 ml of a 5% dextrose solution (placebo). The neurological status of the patients was evaluated by a blinded investigator before the
Low-dose glucose-insulin-potassium is ineffective in acute myocardial infarction: results of a randomized multicenter Pol-GIK trial.
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We aimed to assess the clinical efficacy of glucose-insulin-potassium (GIK) in acute myocardial infarction. Experimental data provided evidence of the beneficial effects of GIK on ischemic myocardium. The clinical trials, mostly uncontrolled and conducted mainly before the thrombolytic era, were
Metabolically controlled reperfusion in acute myocardial infarction: should the polarizing solution be given subselectively?
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BACKGROUND
In working rat hearts, metabolic support of injured tissue enhances recovery after acute myocardial infarction. Clinical experience with a systemic "polarizing solution" supports this claim.
OBJECTIVE
In a dog model of ischemia/reperfusion, we tested the feasibility of subselectively
Effect of modified glucose-insulin-potassium on free fatty acids, matrix metalloproteinase, and myoglobin in ST-elevation myocardial infarction.
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Insulin has a free fatty acid (FFA)-suppressive effect, vascular endothelial growth factor (VEGF)- and matrix metalloproteinase (MMP)-lowering effect, and a potential myocardial-protective effect. Whether low-dose insulin exerts these effects in patients with acute myocardial infarction (MI) was
Effects of glucose-insulin-potassium infusion on QT dispersion in patients with acute myocardial infarction.
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BACKGROUND
Increased QT dispersion during acute myocardial infarction (AMI) has been associated with the occurrence of ventricular arrhythmias. Also, serum potassium levels have been shown to be relevant to the arrhythmic risk in this group of patients. The aim of the present study was to assess
Persistent reduction in left ventricular asynergy in patients with acute myocardial infarction by intravenous infusion of nitroglycerin.
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Intravenous nitroglycerin (NG) infusion in patients with acute myocardial infarction (AMI) has been shown to improve left ventricular function and myocardial perfusion and to decrease ischemic injury and creatine kinase (CK) indexes of infarct size. To determine whether early NG infusions in
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