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epinephrine/hypoxia
Veza se sprema u međuspremnik
Stranica 1 iz 595 rezultatima
Superoxide dismutase functional regulation in neonatal hypoxia: effect of glucose, oxygen and epinephrine.
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Hypoxia is one of the major causes of damage to the fetal and neonatal brain and cardiac functions. In earlier studies, we have reported the brain damage caused by hypoxia and resuscitation with oxygen and epinephrine and have found that glucose treatment to hypoxic rats and hypoxic rats treated
Effects of hypoxia on the contractions and lactate release induced by high-K+, ouabain and epinephrine in guinea-pig aorta.
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1. The 60 mM K+, 152 mM K+, Na-deficient medium and oubain-induced contractions of aorta were not so affected by severe hypoxia. 2. The 60 mM K+, 152 mM K+, Na(+)-deficient medium-induced responses were greatly reduced by deprivation of external Ca2+ in normoxia. 3. As the concentration of
Naloxone potentiates epinephrine release during hypoxia in fetal sheep: dose response and cardiovascular effects.
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The effect of opiate receptor blockade on the plasma catecholamine response to hypoxemia was studied in seven chronically catheterized fetal lambs in utero. All animals underwent treatment with hypoxia alone, naloxone infusion alone (2 mg/kg) and hypoxia with naloxone at four different dosages (0.1,
Magnesium sulfate and fetal plasma concentrations of epinephrine, norepinephrine, and vasopressin in response to acute hypoxemia in goats.
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OBJECTIVE
We measured fetal plasma concentrations of epinephrine, norepinephrine, and vasopressin during acute hypoxemia in goats and tested whether hypermagnesemia altered these endocrine responses.
METHODS
Five chronically catheterized goat fetuses at 124-129 days' gestation were used. After 4
Myocardial metabolism and performance in hypoxia: effect of epinephrine.
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Effects of epinephrine (10(-5) M) on mechanical performance, glycolysis, glycogenolysis, lipolysis, and metabolism of adenine nucleotides were studied in isolated hypoxic rabbit hearts. The exposure of hearts to hypoxia decreased their mechanical performance and heart rate, but increased their
Hypoxia increases endothelin release in bovine endothelial cells in culture, but epinephrine, norepinephrine, serotonin, histamine and angiotensin II do not.
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Calf coronary artery endothelial cells in culture were exposed to normoxic and hypoxic conditions (2% oxygen) for 4 hr and 24 hr. After 24 hr of incubation, there was a significant increase in the accumulation of immunoreactive endothelin (irET) in the media. Incubations with epinephrine,
Effects of epinephrine on the cardiorespiratory response to hypoxia in sedated newborn piglets with intact and denervated carotid bodies.
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In order to evaluate the effects of epinephrine on the cardiorespiratory response to hypoxia in the neonate, 35 sedated, spontaneously breathing newborn piglets (mean +/- SD, age 5 +/- 0.8 days; weight 1.6 +/- 0.3 kg) with intact (ICB) or denervated (DCB) carotid bodies were studied before and
Amelioration of hypoxia-induced striatal 5-HT(2A) receptor, 5-HT transporter and HIF1 alterations by glucose, oxygen and epinephrine in neonatal rats.
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Alterations in neurotransmitters and its receptors expression induce brain injury during neonatal hypoxic insult. Molecular processes regulating the serotonergic receptors play an important role in the control of respiration under hypoxic insult. The present study focused on the serotonergic
Cerebellar 5HT2A receptor function under hypoxia in neonatal rats: role of glucose, oxygen, and epinephrine resuscitation.
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Molecular processes regulating the cerebellar serotonergic receptors play an important role in the control of respiration and cognitive functions under hypoxia. The present study examined cerebellar 5HT receptor alterations and neuroprotective effect of glucose supplementation prior to current
Does sustained epinephrine release trigger a hypoxia-neovascularization cascade?
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BACKGROUND
Recent data regarding the long-term local administration of epinephrine to soft tissues for the purpose of inducing prolonged vasoconstriction have yielded promising results. These studies postulated that long-term release of epinephrine by a microsphere/drug delivery system caused
Epinephrine absorption following endotracheal administration: effects of hypoxia-induced low pulmonary blood flow.
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To investigate the effects of hypoxia-induced decreased pulmonary blood flow on the trans-pulmonary absorption of epinephrine, we measured pulmonary blood flow and arterial plasma tritium counts per minute following endotracheal [3H]epinephrine administration in six chronically instrumented newborn
[Features of blood coagulating and fibrinolytic properties under the action of epinephrine in hypoxia and hypercapnia].
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Acute experiments were carried out on 50 dogs to study the effect of epinephrine in hypoxic (N2 - 15 to 10% O2) or hypoxic-hypercapnic (N2 - 10%, O2 - 5% CO2) atmospheres. Epinephrine led to a maximum increase of blood coagulation and fibrinolysis in normoxic atmosphere. Hypoxia reduced the shift of
Metabolic acidemia with hypoxia attenuates the hemodynamic responses to epinephrine during resuscitation in lambs.
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OBJECTIVE
To examine the effects of metabolic acidemia and hypoxia on the hemodynamic responses to epinephrine in an intact neonatal animal model.
METHODS
Multi-experiment, randomized, controlled trial.
METHODS
Animal research laboratory of a university hospital.
METHODS
Sixteen lambs, ranging in
Utilization of endogenous lipids and glycogen in the perfused rat heart: effects of hypoxia and epinephrine.
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The interaction of hypoxia and catecholamines in the regulation of heart tissue lipid and glycogen utilization was studied in isolated perfused rat hearts with lipids prelabeled in vivo with [1-14C]palmitate. In well oxygenated hearts, epinephrine stimulated triglyceride mobilization by
Cortical 5HT 2A receptor function under hypoxia in neonatal rats: role of glucose, oxygen, and epinephrine resuscitation.
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Neonatal hypoxia induces brain injury through alterations in neurotransmitters and its receptors. Molecular processes regulating serotonergic receptors play an important role in the control of respiration under hypoxia. The present study evaluates the serotonergic regulation of neonatal hypoxia and
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