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hydrocarbon/infarction

Veza se sprema u međuspremnik
Stranica 1 iz 44 rezultatima

Expired hydrocarbons in patients with acute myocardial infarction.

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Prijava Registriraj se
Pentane and isoprene concentrations were analyzed in single end-expiratory breath samples using gas chromatography. Breath analysis was performed in 15 patients with acute myocardial infarction, 15 patients with stable angina, and 15 healthy control subjects. The two patient groups were well matched
Exposure to total hydrocarbons (THC) and volatile organic compounds from air pollution is associated with risk of coronary heart disease. THC exposure from oil spills might be similarly associated, but no research has examined this. We assessed the relationship between THC exposure during the

Myocardial infarction after toluene inhalation.

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Prijava Registriraj se
Hydrocarbon exposure occurs through both intentional and nonintentional substance abuse. We present the case of a young male who sustained an acute myocardial infarction temporally related to inhalation of toluene fumes. We propose possible mechanisms for this rare but serious complication of
OBJECTIVE Ischaemic post-conditioning (IPost-Co) exerts cardioprotection by diminishing ischaemia/reperfusion injury. Yet, the mechanisms involved in such protection remain largely unknown. We have investigated the effects of IPost-Co in cardiac cells and in heart performance using molecular,

Acute myocardial infarction and renal failure following naphtha ingestion.

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We present a case of a non-Q wave myocardial infarction and acute renal failure following an ingestion of naphtha, a petroleum distillate composed primarily of hydrocarbons. The patient's renal, metabolic, and cardiac status improved over several days with aggressive volume replacement and
As a non-invasive method, heart rate variability (HRV) has been widely used to study cardiovascular autonomous control. Environmental epidemiological studies indicated that the increase in an average concentration of particulate matter (PM) would result in a decrease in HRV, which was related to the

Two cases of chlorinated hydrocarbon-associated myocardial ischemia.

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Chlorinated hydrocarbons are well known to produce ventricular arrhythmias because of myocardial sensitization to endogenous catecholamines, but cases of myocardial ischemia have not been described frequently. We report 2 cases of myocardial ischemia after exposure to chlorinated hydrocarbons. The

Deepwater Horizon oil spill exposures and nonfatal myocardial infarction in the GuLF STUDY.

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Workers involved in the response and clean-up of the 2010 Deepwater Horizon oil spill faced possible exposures to crude oil, burning oil, dispersants and other pollutants in addition to physical and emotional stress. These exposures may have increased risk of myocardial infarction (MI) among oil

[Lactate metabolism in acute myocardial infarction].

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In 18 patients with acute myocardial infarction admitted to the Cardiological Care Department within 6 hours after the onset of chest pain, before administration of drugs and then in the 2nd, 3rd, 5th and 7th day, the levels of glucose, pyruvate, lactate in venous blood, the lactate/pyruvate ratio
Levels of polycyclic aromatic hydrocarbons (PAHs) and potentially toxic metals (PTMs) were determined in the commonly consumed beef sausage roll products (coded BS1 - BS6) in Nigeria. This was done in order to assess the safety of regular consumption of these products with respect to the substances
Reducing ischemia reperfusion (I/R) injury has been a major challenge in the treatment of cardiovascular disease. It is widely accepted that mitochondrial apoptosis is an important link in myocardial infarction induced by I/R damage. Studies suggested that aryl hydrocarbon receptor (AhR) pathway

Comparison of two indices of exposure to polycyclic aromatic hydrocarbons in a retrospective aluminium smelter cohort.

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BACKGROUND The association between coal tar-derived substances, a complex mixture of polycyclic aromatic hydrocarbons, and cancer is well established. However, the specific aetiological agents are unknown. OBJECTIVE To compare the dose-response relationships for two common measures of coal

The effect of myocardial infarction on the cardiac sensitization potential of certain halocarbons.

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We have previously shown that many halocarbons and hydrocarbons are capable of producing cardiac sensitization. Briefly, the test method involved exposure of healthy, unanesthetized, beagle dogs to various inspired levels of sensitizing agent, followed by an intravenous dose (8 mug/kg) of

Air Duster Inhalant Abuse Causing Non-ST Elevation Myocardial Infarction

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Inhalant abuse, also known as huffing, is common among teenagers and adolescents in the United States and worldwide. Inhaled aerosols are dangerous due to both the presence of volatile hydrocarbons causing direct organ damage and the risk of the compressed air causing physical trauma (e.g.
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