methyl jasmonate/hypoxia

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Methyl jasmonate delays the latency to anoxic convulsions by normalizing the brain levels of oxidative stress biomarkers and serum corticosterone contents in mice with repeated anoxic stress

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Objectives Repeated exposure to anoxic stress damages the brain through cortisol-mediated increases in oxidative stress and cellular-antioxidants depletion. Thus, compounds with antioxidant property might confer protection against anoxic stress-induced brain injuries. In this study, we further

MaRAP2-4, a waterlogging-responsive ERF from Mentha, regulates bidirectional sugar transporter AtSWEET10 to modulate stress response in Arabidopsis.

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As waterlogging and successive events severely influence growth and development of economically important plants, we attempted to characterize the role of a waterlogging-responsive group I (A-6) ethylene response factor (MaRAP2-4) from Mentha arvensis. Waterlogging, ethylene and methyl jasmonate

The dehydrin wzy2 promoter from wheat defines its contribution to stress tolerance.

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Dehydrins (DHNs), which are stress-related proteins, are important for plant survival under various abiotic and biotic stresses. To elucidate the regulatory mechanisms of wheat-derived DHNs under these stresses, we characterized the DHN wzy2 promoter of the wheat cultivar Zhengyin 1 and studied its

Abscisic acid-dependent and -independent expression of the carrot late-embryogenesis-abundant-class gene Dc3 in transgenic tobacco seedlings

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We studied the expression of three promoter 5' deletion constructs (-218, -599, and -1312) of the LEA (late embryogenesis abundant)-class gene Dc3 fused to beta-glucuronidase (GUS), where each construct value refers to the number of base pairs upstream of the transcription start site at which the

Jasmonate Regulates Plant Responses to Postsubmergence Reoxygenation through Transcriptional Activation of Antioxidant Synthesis.

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Submergence induces hypoxia in plants; exposure to oxygen following submergence, termed reoxygenation, produces a burst of reactive oxygen species. The mechanisms of hypoxia sensing and signaling in plants have been well studied, but how plants respond to reoxygenation remains unclear. Here, we show

Flooding of the apoplast is a key factor in the development of hyperhydricity.

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The physiological disorder hyperhydricity occurs frequently in tissue culture and causes several morphological abnormalities such as thick, brittle, curled, and translucent leaves. It is well known that hyperhydric shoots are characterized by a high water content, but how this is related to the

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