progesterone/edema

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Progesterone decreases cortical and sub-cortical edema in young and aged ovariectomized rats with brain injury.

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OBJECTIVE Traumatic brain injury (TBI) -induced brain edema can be reduced by acute progesterone (PROG) treatment in young adult males and females, and in aged males. To extend these findings we tested these hypotheses: 1. Acute PROG treatment post-TBI will reduce cortical edema in aged females as

Intralipid Vehicle Does Not Interfere with the Efficacy of Progesterone in Attenuating Edema following Traumatic Brain Injury.

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The recent disappointing results of phase III trials for progesterone (PROG) in traumatic brain injury (TBI) have triggered speculation about reasons for the negative outcomes. One confounding factor may have been the vehicle used to administer PROG. Virtually all of the many pre-clinical

[Aldosterone and progesterone content and blood renin activity in women with idiopathic edema syndrome in various phases of menstrual cycle].

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Aldosterone and progesterone content and renin activity were studied by the radioimmunological method in women with the idiopathic edema syndrome and in healthy women in various phases of the menstrual cycle. The most significant differences were revealed in the luteinic phase of the cycle. A

Second-trimester maternal serum progesterone levels in Turner syndrome with and without hydrops and in trisomy 18.

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Placental proteins, such as inhibin A and hCG and its subunits, as well as the placental steroid progesterone, are elevated in second-trimester maternal serum from cases of fetal Down syndrome. Since different cellular mechanisms are required for protein versus steroid synthesis and secretion, these

[Effect of progesterone on cerebral cortex edema in rats exposed to focal ischemia/reperfusion].

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OBJECTIVE To explore the effects of progesterone (PROG) on brain edema in rats. METHODS Forty eight rats were randomly divided into 6 groups that were ischemia/reperfusion (I/R), dimethylsulfoxide (DMSO), and pretreatment, pre + posttreatment, posttreatment with PROG, and dexamethasone (DEXA)

Serum progesterone levels correlate with decreased cerebral edema after traumatic brain injury in male rats.

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Previous animal research suggests that progesterone may have powerful neuroprotective effects in traumatic brain injury (TBI). This experiment tested the hypothesis that progesterone levels correlate with decreased cerebral edema in male rats with bilateral medial frontal cortex injuries. Three

Progesterone treatment attenuates brain edema following contusion injury in male and female rats.

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To assess the effectiveness of progesterone as a treatment for edema following traumatic brain injury, male and female rats were injected with progesterone or the oil vehicle over a 3-day period following a cortical contusion injury. Oil-treated rats showed significant localized edema as measured by

Progesterone rapidly decreases brain edema: treatment delayed up to 24 hours is still effective.

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Cerebral edema is a serious side effect of traumatic brain injury. We have previously established that progesterone injections, initiated within 1 h after cortical contusion injury, reduced edema when assessed 3 days later. To determine how rapidly progesterone can reduce edema, male and female rats

Progesterone is neuroprotective by inhibiting cerebral edema after ischemia.

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Ischemic edema can alter the structure and permeability of the blood-brain barrier. Recent studies have reported that progesterone reduces cerebral edema after cerebral ischemia. However, the underlying mechanism of this effect has not yet been elucidated. In the present study, progesterone

Effects of the duration of progesterone treatment on the resolution of cerebral edema induced by cortical contusions in rats.

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PURPOSE. The aim of the present study was to assess the effect of different durations of administration of progesterone (4 mg/kg) on the resolution of edema 6 days after medial frontal cortex contusions (MFC) in male adult rats. METHODS Animals sustaining injury were injected with progesterone or

Both estrogen and progesterone attenuate edema formation following diffuse traumatic brain injury in rats.

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Females have reduced brain edema compared to males after experimental brain trauma, although contradictory reports exist as to whether this is due to either estrogen or progesterone. In the present study, we demonstrate in both male and ovariectomized female rats that a single physiological dose of

Progesterone attenuates cerebral edema in neonatal rats with hypoxic-ischemic brain damage by inhibiting the expression of matrix metalloproteinase-9 and aquaporin-4.

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The aim of this study was to investigate the effects of progesterone (PROG) on blood-brain barrier (BBB) permeability, cerebral edema and the expression of matrix metalloproteinase-9 (MMP-9) and aquaporin-4 (AQP-4) in neonatal rats with hypoxic-ischemic brain damage (HIBD) and to explore the

Effect of estrogen and/or progesterone administration on traumatic brain injury-caused brain edema: the changes of aquaporin-4 and interleukin-6.

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The role of aquaporin-4 (AQP4) and interleukin-6 (IL-6) in the development of brain edema post-traumatic brain injury (TBI) has been indicated. The present study was designed to investigate the effect(s) of administration of progesterone (P) and/or estrogen (E) on brain water content, AQP4

Progesterone administration modulates AQP4 expression and edema after traumatic brain injury in male rats.

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This study investigates whether progesterone administration regulates AQP4 and GFAP expression in rats with bilateral contusion injuries of the medial frontal cortex. Male rats were given 0 or 16 mg/kg injections of progesterone at 1, 6, 24, and 48 h post-injury. Brains were extracted at 24 h or 72

[Ovarian function in orthostatic idiopathic edema. Oral administration of progesterone and changes in capillary permeability].

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The ovarian function was investigated in 30 women with postural idiopathic oedema by measuring plasma oestradiol and progesterone levels between the 21st and 23rd days of the menstrual cycle. Plasma progesterone concentrations were found to be lower than 5 ng/ml in 53% of the cases and than 10 ng/ml

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