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Stranica 1 iz 501 rezultatima
Prosthetic material degeneration over time as a possible factor in delayed recurrence of hemifacial spasm after successful microvascular decompression.
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UNASSIGNED
The effectiveness of microvascular decompression in treating hemifacial spasm is widely accepted. However, some experience recurrence of hemifacial spasm after successful decompression surgery. Especially, delayed recurrence more than 5 years after surgery is rare and the cause of this
[A case of unilateral masticatory spasm without hemifacial atrophy].
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Masticatory spasm is a rare clinical feature and mostly associated with disorders of general movement. Usually it occurs bilaterally. Unilateral masticatory spasm is mostly associated with facial hemiatrophy. So, unilateral masticatory spasm without facial hemiatrophy is extremely rare. As far as we
Muscle spasms associated with Sudeck's atrophy after injury.
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Four patients developed abnormal involuntary movements of a limb after injury. All subsequently developed sympathetic algodystrophy with Sudeck's atrophy and then abnormal muscle spasms or jerks of the affected limb, lasting years. Sympathetic block in three patients did not relieve the abnormal
Treatment of hemifacial spasm in patient with hemifacial atrophy using combination therapy (ultrasound therapy and TENS): a case report.
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A 19-year-old male patient presented with facial hemi-atrophy with unilateral spasms of the masseter and temporalis muscles. Ultrasound therapy and Transcutaneous Electric Nerve Stimulation therapy, known as combination therapy, were given on alternate days for 2 weeks. At the end of 2 weeks of
Hemimasticatory spasm in hemifacial atrophy: diagnostic and therapeutic aspects in two patients.
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We report two cases of hemimasticatory spasm in association with progressive hemifacial atrophy. On the basis of neurophysiological and magnetic resonance imaging assessments, a peripheral irritation of the trigeminal nerve--probably due to entrapment of the motor branches in the infratemporal
Alexander disease with mild dorsal brainstem atrophy and infantile spasms.
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We present the case of a Japanese male infant with Alexander disease who developed infantile spasms at 8 months of age. The patient had a cluster of partial seizures at 4 months of age. He presented with mild general hypotonia and developmental delay. Macrocephaly was not observed. Brain magnetic
Smooth muscle cell-extrinsic vascular spasm arises from cardiomyocyte degeneration in sarcoglycan-deficient cardiomyopathy.
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Vascular spasm is a poorly understood but critical biomedical process because it can acutely reduce blood supply and tissue oxygenation. Cardiomyopathy in mice lacking gamma-sarcoglycan or delta-sarcoglycan is characterized by focal damage. In the heart, sarcoglycan gene mutations produce regional
Increased plasma xanthine oxidoreductase activity deteriorates coronary artery spasm.
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Increased reactive oxygen species (ROS) contributes to the development of endothelial dysfunction, which is involved in coronary artery spasm (CAS). Xanthine oxidoreductase (XOR) plays a pivotal role in producing both uric acid and ROS. However, the association between plasma XOR activity and CAS
Muscle spasms associated with Sudeck's atrophy after injury.
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Reversible cerebral atrophy in infantile spasms caused by corticotrophin.
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By the way, doctor. My father had a massive stroke that left him paralyzed on one side. He's aware of all that's going on and can talk some, but he has severe spasticity in his left elbow, shoulder, and hand, along with muscle atrophy. Would Botox injections help the painful spasms in his arm and hand?
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[Studies on chronic poliomyelitis. III. Progressive muscular atrophy developing after ten years, clinical picture of spasms and trophic disorders but without sensory disorders].
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Progressive generalized brain atrophy and infantile spasms associated with cytochrome c oxidase deficiency.
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Parry-Romberg syndrome with hemimasticatory spasm in pregnancy; A dystonia mimic.
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Parry-Romberg syndrome (PRS) with hemimasticatory spasm (HMS) is quite an uncommon overlapping phenomenon which very often mimics jaw closing dystonia. A previously healthy 35-year-old female, during her 5(th) month of pregnancy started developing intermittent unilateral painful spasms of jaw while
[MR imaging in infantile spasms].
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CT and MRI in 13 patients with infantile spasms were analyzed. Cortical atrophy was found in 8 cases and ventricular dilatation in 9 cases. The patients with severe cortical atrophy or ventricular dilatation had poorer prognosis than the patients with normal CT findings. In addition to cortical
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