thromboangiitis obliterans/groznica

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[Fever therapy in thromboangiitis obliterans].

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"Idiopathic eosinophilic vasculitis": another side of hypereosinophilic syndrome? A comprehensive analysis of 117 cases in asthma-free patients.

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The absence of asthma may rule out a diagnosis of eosinophilic granulomatosis with polyangiitis (EGPA) in patients with hypereosinophilic syndrome (HES) and features of vasculitis.To describe eosinophilic vasculitis (EoV) as a possible manifestation of HES

An uncommon systemic arteritis--a case report.

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A 26-year-old male shortly after an acute respiratory disease was affected by a thrombophlebitis of the left leg. After a few days he had two syncopal attacks. Later on, a myocardial ischemia was diagnosed. Subsequently the patient began to complain of a bilateral claudication of the calves; after

[Lower limb ischemia cured by stimulation electrode implantation assisted with microendoscopic discectomy system: A case report].

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A 58-year-old male patient diagnosed with thromboangiitis obliterans (Fontaine stage IV) was recently treated with microendoscope discectomy system-assisted spinal cord stimulation electrode implantation and cured by department of vascular surgery combined with department of spinal surgery at Peking

The arteritides.

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This article reviews the histopathological, clinical and immunological features of the arteritides. Based on these criteria, a classification scheme is proposed that includes infectious and non-infectious causes. Included in the non-infectious arteritides are: hypersensitivity vasculitis including

[Superficial migratory thrombophlebitis: a clinical and histologic review of 8 cases].

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BACKGROUND Superficial migratory thrombophlebitis (SMT) or thrombophlebitis migrans is characterized by recurrent episodes of localized thrombosis of the superficial veins in the limbs and trunk. It has been associated with various systemic diseases that should be taken into consideration when

Inflammation-induced thrombosis: mechanisms, disease associations and management.

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Although inflammation-induced thrombosis is a well-known entity, its pathogenesis remains complicated. There are complex interactions between inflammation and hemostasis, involving proinflammatory cytokines, chemokines, adhesion molecules, tissue factor expression, platelet and endothelial

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