aldolase/seizures

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[Activity of aldolase, lactate dehydrogenase and content of lactic and pyroracemic acids in different areas of the brain in rat, with repeated convulsions evoked by corazole].

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[A mitochondrial encephalomyopathy due to partial cytochrome c oxidase deficiency with giant evoked potentials--a case report].

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A case of mitochondrial encephalomyopathy with a partial cytochrome c oxidase deficiency was reported with special reference to electrophysiological studies. A 56-year-old man was readmitted to Himeji Central Hospital due to mental deterioration and character change. At the age of 44 when he was

Correlation between blood gases, glycolytic enzymes and EEG during electroconvulsive treatment in relaxation.

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Twenty-three psychiatric patients were investigated during electroconvulsive treatment in relaxation. The blood gases, pH and serum bicarbonate levels in blood samples from the internal jugular vein and the femoral artery were measured radiometrically. The LDH fractions were separated

Acute rhabdomyolysis following administration of high-dose cyclophosphamide: case report.

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Rhabdomyolysis is an unusual complication of hematopoietic stem cell transplantation (HSCT). Cyclophosphamide has been one of the key drugs in the most common preparative regimen for HSCT. We present here a rare case of acute rhabdomyolysis following administration of high-dose cyclophosphamide. A

Hereditary fructose intolerance presenting as Reye's-like syndrome: report of one case.

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Hereditary fructose intolerance (HFI) is an autosomal recessive disease caused by catalytic deficiency of aldolase B (fructose-1, 6-bisphosphate aldolase). Herein we report on a case of hereditary fructose intolerance with initial presentation of episodic unconsciousness, seizure, hypoglycemia,

Genetic disorder in carbohydrates metabolism: hereditary fructose intolerance associated with celiac disease.

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Celiac disease (CD) has been associated with several genetic and immune disorders, but association between CD and hereditary fructose intolerance (HFI) is extremely rare. HFI is an autosomal recessive disease caused by catalytic deficiency of aldolase B (fructose-1,6-bisphosphate aldolase). We

[Investigation of etiologies for acute renal failure due to rhabdomyolysis in 5 patients].

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We experienced 5 cases of acute renal failure due to rhabdomyolysis during the last two years and investigated those etiologies. Diagnosis of rhabdomyolysis was established by the detection of elevated serum creatine phosphokinase, myoglobin, aldolase, myoglobinuria as well as by the clinical

[Nontraumatic rhabdomyolysis: its etiology and the predictive factors of acute kidney failure].

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BACKGROUND The aim of this study was to determine the causes of non traumatic or medical rhabdomyolysis (RM) and analyze the clinical and biologic characteristics, as well as the predictive factors of acute renal failure (ARF). METHODS Fifty-nine patients with RM admitted to the Internal Medicine

[Rhabdomyolysis: clinical features, causes, complications and treatment].

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Rhabdomyolysis is a condition affecting body homeostasis that results from impaired supply of muscles with energy, nutritional factors and blood. Complex pathophysiological mechanism causes that extended myolysis may complicate different clinical conditions, such as: crush syndrome, excessive

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